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Module 4 > Parkinsons Disease > Flashcards

Parkinsons Disease Flashcards

1
Q

Parkinsons disease

A

Chronic progressive neurodegenerativve disease of the CNS NO DISEASE

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2
Q

Causes of PD

A

Unknown could be enviromental or a persons-genetic

Increase risk well water, pesticides, herbicides, industrial chemicals, wood, pulp mills rural residence

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3
Q

Dopamine

A

Degeneration of dopamine

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4
Q

Clinical manifestions of PD

A

Onset is gradual and insidious with ongoing progression
Tremor(resting tremor)
Rigidity
Akinesia(absent of movement) / OR bradykinesia(slow/movement).
Postural instability

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5
Q

Beginnings stages

A

Mild tremor, slight limp, decrease arm swing

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6
Q

Later stages

A

shuffling propulsive gait with arms flexed, loss of postural reflexes
Diaphragm , tongue lips jaw may be involved

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7
Q

Tremor

A

Often first sign
Pill rolling hand tremor
Initially minimal
More prominent at rest
Aggravated by emostional stress
And increase concentration

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8
Q

PD drool so they are at risk for

A

Aspiration

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9
Q

What do 90% of PD pt experience

A

Hypokinetic dysarthria

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10
Q

Rigidity

A

Cogwheel rigidity (jerky quality,
Sustained muscle contraction
Complaints of soreness
Feeling tired and achy
Pain in the head upper body spine or legs
Slowness of movement

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11
Q

Characteristic is of a person with PD

A

Stooped posture
Masked face
Drooling
Destination (shuffling gait)

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12
Q

Nonmotor symptoms

A

Depression and anxiety
Apathy
Fatigue
Pain
Urinary retention andconstipation
Erectile dysfunction
Memory changes

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13
Q

Sleep inPD

A

Sleep problems common
Difficulty staying asleep
Restless sleep
Nightmares
Drowsiness during the day
REM behavir disorder
(Violent dreams potentially dangerous motor activity during sleep )

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14
Q

Complications as disease progresses

A

Motor symptoms
Weakness
Akinesia
Neuro problems
Neuropsychiatric problems
Dementia often results (increase mortality)

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15
Q

Dysphagia results from PD what can that cause

A

Malnutrition and aspiration

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16
Q

What PD complications of general debilitation (weakness) may lead

A

Pneumonia UTIs skin break down

17
Q

Orthostatic BP results from PD what risk does this increase

A

Fall an injuries

18
Q

Diagnostic test PD

A

No definite diagnostics ***
Pt history and clinical features
Presence of two or more cardinal manifestations TRAP
Medicalhistory presenting symptoms neuro exam

POSITIVE RESPONSE TO ANTIPARKINSONIAN DRUGS

19
Q

What are we things that PD suffer from

A

Focusing on two things at once ( writing small- focusing on writing big)

Overactive bladder (if they have an urgency but walk slow)

20
Q

What teaching can we do for rigidity ?

A

Passive range of motion so they dont develop atrophy

21
Q

PD drug therapy

A

Aimed at correcting imbalances of neurotransmitters within the CNS
Antiparksinson drugs either enhance or release supply of dopamine
Antagonize or block the effects of overactive cholinergic neurons in the striatum

22
Q

Deep brain stimulation

A

Most common surgical treatment
Reversible and programmable
Improves motor function
Reduces dyskinesia and medications
Pace maker for the brain
In clavicle but if they have enough fat but if not they put it in abdomen

23
Q

Ablation therapy

A

Destroy tissue that produces abnormal chemical or electrical impulses leading to tremors or other symptoms

24
Q

Transplantation

A

Research and clinical trials ongoing

25
Q

Nutritional therapy

A

At risk for mal nurtirion or constiipation
Pt with Dysphagia and bradykinesia need food that is easily chewed and swallowed
Adequate fiber
Eating more numerous small meals
Provide ample time

26
Q

Nursing diagnosis

A

Self care deficit
Chronic confusion
Impaired physical mobility
Impaired verbal communication
Impaired swallowing
Risk for imbalanced nutrition less than body requirements

27
Q

Nursing management

A

Maximize neuro function
Maintain independence-ADLS
Optimize psychosocial well being
Administer medication as prescribed
Facilitate nutritional intake
Interdisciplinary collaboration -PT , OT speech

Module 4 (47 decks)

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