Acute Kidney Injury Flashcards

1
Q

A wet bed

A

Acid base balance
Water removal
Erythropoesis
Toxin removal
Blood pressure control
Electrolyte balance
Vitamin d activation

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2
Q

Renal failure

A

AKI
CKD

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3
Q

AKI

A

Acute, rapid loss of renal function
Oliguria may be present
Uremia may be present

May see metabolic acidosis

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4
Q

Oliguria

A

Abnormal small amounts of urine

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5
Q

Uremia

A

Urine in the blood

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6
Q

AKI s/s

A

Rapid decrease in renal function
Increase in BUN creatine and K+(heart)
Decreased of urine output
Azotemia

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7
Q

Azotemia

A

Biochemically abnormally nitrogenous compounds

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8
Q

AKI 3 major categories for causes

A

Pre renal intrarenal postrenal

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9
Q

Prerenal

A

Due to decreased blood flow into the kidneys

Hemorrhage
Renal artery issue
Loss of fluid

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10
Q

Post renal

A

Obstruction from the outflow of the kidneys

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11
Q

Unilateral obstruction

A

Kidney stone

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12
Q

Bilateral obstruction

A

From a tumor (recognized quicker)

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13
Q

Intrarenal

A

Damage to tubeless, the glomerulus or the interstitium (space between the tubules)

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14
Q

What causes intrarenal

A

Acute tubular necrosis
big cause can be From prerenal
(Depending how long and how much blood flow was compromised can cause tissue to die and result in CKD

Glomerulonephritis

Acute interstitial nephritis (days to weeks)

Nephrotoxic drugs

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15
Q

What causes POSR renal

A

Intraabdominal tumors benign prostatic
Hyperplasia
Kidney stones

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16
Q

Acute kidney injury

A
17
Q

AKI s/s Oliguric phase

A

Oliguria <400 ml/day occurs within 1-7 days of KI
Urinalysis casts RBC wbc sp gr fixated at 1.010
Metabolic acidosis
Hyperkalemia &Hyponatremia
Elevated BUN &creatine
Fatigue and malaise

18
Q

Diuretic phase s/s

A

Gradual increase in urine output 1-3 l/d may reach 3-5 l a day
Hypovolemia , dehydration
Hypotension
BUN and creatine levels began to normalize

19
Q

Recover stage s/s

A

Begins when GFR increase
BUN and creatine levels plateau then decrease

20
Q

Initiating phase

A

First indication

21
Q

In the oliguric stage what are we going to watch?

A

The heart =hyperkalemia in this stage

22
Q

What are we going to monitor closely in diuretic phase?

A

Hypovolemia , dehydration hypotension

23
Q

AKI s/s

A

Decrease urine output
Swelling legs , ankle, feet
SOB(Dyspnea)
Fatigue
Loss of apetite
N/v
Arrhythmia
Chest pain or pressure
Easy or unusual bleeding (low platelets )
Confusion (toxins cause neuro effect)

24
Q

What stage is your pt a candidate for dialysis ?

A

ESRD only

25
Q

Nursing management

A

Eliminate cause, prevention complications, assist with recovery
Vitals
I&O daily weight
Oxygenation
Manage fluid balance
Positioning
Skin care
Dialysis may need to be considered

26
Q

Acute on chronic

A

When kidneys shut down & unknown cause of lack of blood flow
They don’t recover how the kidneys use to work

27
Q

What is sodium polystyrene sultanate-kayexalate used for?

A

Hyperkalemia

28
Q

What meds do we administer in AKI

A

Diuretics(furosemide)
Kayexalate (sodium polyrene)
Calcium chancel blockers

29
Q

Nephrotoxic agent

A

Contrast dye
Chemo
Some antibiotics

30
Q

Nutrition we can teach

A

High calorie
Low sodium
Low potassium
Lean meats
Avoid can food , table salts, if they do can foods rinse them

31
Q

Other therapy involved

A

Renal replacement therapy

32
Q

In older pt

A

GFR decline
Increase risk of diabetes and CVD

Increase risk for dehydratopm

Older kidney does not compensate as well for fluid volume , solute load and cardiac output