oedema and congestion Flashcards

1
Q

what is congestion ?

A

this is relative excessive blood in vessels in the tissue or organs

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2
Q

examples of congestion

A

deep vein thrombosis
hepatic cirrhosis
generalised acute congestion
congestive cardiac failure

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3
Q

what causes hepatic cirrhosis

A

liver damage from alcohol and hepatitis virus

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4
Q

congestive heart failure

A

stenosis or ischemia may cause reduced pumping capacity of the heart there is decreased cardiac output and raas system is activated to increase retention leading to fluid overload

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5
Q

effects of cardiac congestion

A

raised JVP
pulmonary oedema
peripheral oedema
liver congestion tachycardia
hepatomegaly

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6
Q

presentation of hepatic central venous congestion

A

there is stasis of blood which is poorly oxygenated that is the pericentral hepatocytes

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7
Q

periportal hepatocytes

A

relatively better oxygenated due to proximity of hepatic arterioles

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8
Q

what is oedema

A

Accumulation of abnormal amounts of fluid in the extravascular compartment ​

intercellular tissue compartment (extracellular fluid) ​ body cavities ​

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9
Q

peripheral oedema

A

increased interstitial fluid in tissues

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10
Q

what is an exudate

A

Part of inflammatory process* due to  vascular permeability​

Tumour, inflammation, allergy​

Higher protein/albumin content (cells)​

H2O & electrolytes​

High specific gravity​

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11
Q

pathophysiology of oedema

A

there is increased hydrostatic pressure which leads to transudate

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12
Q

pathophysiology of peripheral oedema

A

right side failure , hypertrophy of the right ventricle means high pressure difficult for the return of blood, blood retention in systemic veins .

there is increased blood pressure in capillaries and increased filtration rate

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13
Q

pathophysiology of lymphatic blockage

A

lymphatic Obstruction – hydrostatic pressure upset​

Lymphatic drainage is required for normal flow​

If lymphatic system blocked  lymphoedema​

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14
Q

pathophysiology of oedema in abnormal renal function

A

Abnormal renal function results in Salt (NaCl) and H2O retention​

Secondary in heart failure - reduced renal blood flow​

Primary: acute tubular damage eg hypotension​

 renal function is the result of both​

  salt and H2O ​

 intravascular fluid volume ​

 secondary  PC ​

 oedema​

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15
Q

pathophysiology of low protein oedema

A

due to the insufficient proteins in the body there is decreased oncotic pressure and therefore increased filtration levels

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16
Q

causes of low protein levels

A

e.g. nephrotic syndrome  leaky renal glomerular basement membrane; lose protein; generalised oedema​

e.g. hepatic cirrhosis  diffuse nodules and fibrosis in liver; liver unable to synthesise enough protein ​

e.g. malnutrition  insufficient intake of protein​

LP.S[(PC-Pi)-(C-i)] =JV​

17
Q

pathophysiology of permeability oedema

A

there is increased endothelial permeability causes exudate.

18
Q

examples of pathophysiology of permeability oedema

A

burns
acute inflammation such as pneumonia