Heart Week 1b: Rhythmns, etc. Flashcards

1
Q

PSVT

A

Paroxysmal Supraventricular Tachy:

a. sudden onset and termination
b. atrial rates between 140 and 250 bpm
c. normal QRS
d. often causes by reentry

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2
Q

Tachycardia

A

Heart rate > 100bpm

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3
Q

Sinus Bradycardia

A

P and QRS is normal, but rate is < 60bpm

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4
Q

Escape Rhythm

A

a. normal QRS but no P

b. wide QRS, maybe no P (Ventricular)

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5
Q

Tell me about the three AV blocks.

A

a. 1st Degree: PR interval > 5 small boxes (0.2 secs)
b1. 2nd Degree, Mobitz: P is constant, but QRS gets closer and closer until a P happens with no QRS
b2. Mobitz II: all looks normal, then QRS drops out, sometimes the QRS is wide
c. 3rd degree: aka- complete block, QRS and P are completely independent

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6
Q

What is normal sinus rhythm?

A

60-100 bpm

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7
Q

What is sinus tacky?

A

100-180 bpm

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8
Q

What is Reentrant SVT?

A

a. 140-250 bpm
b. P wave is hidden or retrograde
c. may abruptly terminate

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9
Q

Focal Atrial Tachy

A

a. 130-250 bpm
b. P wave is not normal, but presentation may vary
c. AV block may increases, but rarely reverts

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10
Q

Atrial Flutter

A

a. 180-350 bpm
b. “saw-toothed” (different from atrial fibrillation)
c. AV block may increase

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11
Q

Atrial Fibrillation

A

no distinct P waves (different than atrial flutter)

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12
Q

Give me the M/E (mechanism/Example) for decreased automaticity

A

M: decrease in phase 4 depolarization, eg- parasympathetic stimulation

E: sinus bradycardia

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13
Q

give me the M/E for conduction block.

A

M: ischemia, anatomics or drug-induced impaired conduction

E: 1st, 2nd, and 3rd degree AV block

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14
Q

Give me the M/E for sinus node enhanced automaticity.

A

M: increased phase 4 depolarization, e.g.- sympathetic stimulation.

E: sinus tachycardia

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15
Q

Give me the M/E for enhanced automaticity of the AV node, ectopic focus

A

M: acquires phase 4 depolarization

E1: AV junctional tachy
E2: ectopic atrial tachy and some forms of VT

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16
Q

Gimme the M/E of DAD (delayed afterdepolarization).

A

M: high intracellular Ca++ (e.g.- digitalis toxicity)

E: premature atrial/ventricular beats, idiopathic VT and digitalis

17
Q

Gimme the M/E of EAD.

A

M: prolonged AP duration

E: Torsades de Pointe

18
Q

Compare DAD and EAD.

A

DAD: has p wave very soon after QRS

EAD: has p wave while QRS is still happening, ends up with a P wave that can look like an M.

19
Q

What channels can cause the EAD?

A

a. during phase 2, it is most likely Ca++

b. during phase 3, it is most likely a recovery of some inactivated Na+ channels

20
Q

What causes a DAD?

A

high intracellular Ca++ concentration, can be caused by digitalis toxicity.

21
Q

What is the difference between ionotropic and metabotropic receptors?

A

Ionotropic: becomes a channel, ion flows right in, acts more quickly

Metabotropic: uses a second messenger like G-protein, acts slower, ion does not enter the cell.

22
Q

During what part of the EKG are the K+ channels maximally open?

A

Q-T Interval.

Most K+ channels are opened when the cell depolarizes and stay open until repolarization.

23
Q

Why is the done by the left ventricle greater than the work done by the right ventricle?

A

Work is proportional to output. Since the R/L ventricles have equal output, then you consider the resistance against which it moves, AFTERLOAD. Aortic pressure is greater than the pulmonary pressure, so the L ventricle does more work.

24
Q

What area of the heart does the LAD supply?

A

apex, ant wall of LV and ant 2/3 of septum

25
Q

What area of the hear does the LCX supply?

A

lateral wall of LV.

26
Q

What area of the heart does the RCA supply?

A

RV free wall, post 1/3 of sept, posterior wall of LV.

27
Q

How big are arterioles? What surrounds them?

A

10-60 microns with 1-5 layers of smooth muscle.

28
Q

How big are capillaries? What surrounds them?

A

5-10 microns in diameter (aout 0.5 to 1.0 mm in length). No smooth muscle.

29
Q

How big are venules? What surrounds them?

A

~10-60 microns in diameter. No smooth muscle.