Ch 84 Abdo wall recon and hernia Flashcards
hernias
- ring may be confined within a normal aperture in the abdominal wall true hernia) or (false hernias) as a result of trauma/incision
- congenital hernias, the sac is a mesothelial membrane (peritoneum) covering the contents,
- acute traumatic or incisional hernias, no sac is present
- Hernial contents without a mesothelial covering are at risk for developing adhesions
- classified by their location or by whether they are spontaneous or acquired.
What is an auto-penetrating hernia?
A traumatic abdominal wall hernia caused by a fractured rib penetrating through abdominal musculature
In the cranial, middle and caudal thirds of the abdomen, which aponeuroses are sitting superficial and deep to the rectus abdominis?
the external abdominal oblique is always superficial to the rectus
Cranial:
- External Oblique - Superficial
- Internal oblique - Both
- Transverse Abdominis - Deep
Middle
- External Oblique - Superficial
- Internal Oblique - Superficial
- Transverse Abdominus - Deep
Caudal
- All superficial
- external rectus sheath contains tendinous aponeuroses of the abdominal muscles that run superficial to the rectus abdominis muscle
- external rectus sheath has been shown to be the primary strength-holding layer throughout ventral midline closures
Where do the internal and external abdominal obliques and the transverse abdominis originate from?
External abdominal oblique
- Originates from 4/5th to 12th rib and from last rib and thoracodorsal fascia
- Runs in caudoventral direction
Internal Obdominal oblique
- Originates from thoracolumbar fascia caudal to last rib and from tuber coxae
- Runs cranioventral
Transverse Abdominis
- Lumbar portion arising from the transverse processes of the lumbar vertebrae and thoracolumbar fascia
- Costal portion - Arising from medial sides of 12th and 13th rib as well as 8th-11th costal cartilages
- Runs in dorsoventral direction
Location of Abdominal Hernias
trauma
* Paracostal hernia;
* dorsal lateral hernia;
* pre-pubic ligament rupture
* femoral hernia;
congenital
* umbilical hernia;
* ventral hernia (subxiphoid)
* scrotal hernia
* inguinal hernia;
What are substernal midline defects often associated with?
Congenital peritoneal pericardial diaphragmatic hernia
PPDH often assoc with incompletely fused caudal sternebrae
Pathophysiology of Abdominal Hernias
- Hernias can result from genetically impaired collagen, wound healing deficiencies; traumatic injury; failed closures
- overall success of repair rarely depends on the repair itself but sequelae to organ herniation or trauma that impaired normal body function > loss of domain, incarceration or obstruction, or strangulation, traumatized organs
acquired hernias
- mechanical strain on load-bearing tissues induces secondary changes in fibroblast > results in abnormal collagen and fascial weakness.
Space-Occupying Effects
“Loss of domain”
- abdominal wall has become accustomed to a relatively small intraabdominal volume because of organ displacement
Forcing the herniated contents can result in:
1. excessive tension on the repair (risk for recurrence)
2. acute pulmonary complications, (restriction of diaphragm function)
3. poor organ perfusion (abdominal compartment syndrome)
List methods of explansion used in human surgery for loss of domain
Progressive pneumoperitoneum
Silastic expanders
Staged reduction
Prosthetic material
Incarceration
- incarcerated organs are irreducible and can become lethal, strangulated obstructions within hours
- incarcerated obstruction depend on contents of the hernia and size of the defect > inelastic hernial rings, such as scrotal or femoral hernias, are at high risk
- intestine, uterus, or bladder
- SI: risk is greatest when the hernial ring approximates the size of the bowel.
- urinary bladder: perineal, ventral, inguinal, and traumatic pubic hernias
- decompression and diversion of urine flow before sx (cysto, catheter, cystostomy tube)
Strangulation
- implies contents are incarcerated and undergoing devitalization from arrested circulation.
- Impaired circulation> venous or arterial occlusion or a combination
- Early venous obstruction > reversible organ engorgement
- subsequent arterial stagnation occurs due to interstitial pressure at capillary beds > causes rapid organ necrosis (wall compromise or rupture)
mechanisms:
- constriction of the blood supply at the hernial ring
- torsion of the vascular pedicle
- Traumatic > adhesions trap organs within the hernia, and contraction during wound healing eventually constricts the hernia ring
- complications and death is 50% higher in humans with incarcerated or strangulated hernias
How can intestinal strangulation lead to rapid systemic illness?
- Bacterial transmigration (through the compromised tissue, rupture may cause toxaemia and septicaemia)
- Vasoactive substances release (arachidonic acid metabolites, cytokines, leucotrienes, kinins) from tissue and blood cell autolysis
- Redistribution of fluids and severe cardiopulmonary effects
- surgical reduction of strangulated hernias may causerapid release of vasoactive substances into the circulation
Principles of Abdominal Hernia Repair
indications:
(1) the hernia is symptomatic (abdominal discomfort, organ obstruction),
(2) the hernia results in significant protrusion that affects the animal’s quality of life or the owner
(3) the hernia poses a significant risk for hollow organ obstruction
Goals of sx
(1) ensure the viability of entrapped hernia contents;
(2) release and return contents into their normal location within the abdominal cavity;
(3) obliterate redundant hernia sac tissue;
(4) provide a tension-free and, if possible, secure primary closure of the defect using strong, healthy surrounding tissue.
Ventral Abdominal Hernias
Anatomy, Etiology, and Pathogenesis
- embryo the abdominal wall is formed by migration of cephalic, caudal, and lateral folds.
- The umbilical aperture, passageway for cord (umbilical blood vessels, small vitelline duct, and stalk of the allantois), remains after normal migration and fusion of these folds
- falciform ligament and middle umbilical ligament of the bladderare attached to the internal aspect of the umbilicus.
- During embryologic development the umbilical ring allows herniation of the midgut into the coelom of the umbilical cord, where it has more room to grow.
- Failure of fusion or delayed fusion of the lateral folds result in a congenital umbilical hernia
- Most umbilical hernias are inherited and are probably the result of a polygenic
- concomitant incomplete caudal sternal fusion or diaphragmatic hernias
- falciform ligament (the remnant of the umbilical vein)
- middle umbilical ligament of the bladder (the remnant of the urachus)
What is the embryological cause of ventral abdominal wall hernias?
Failure of fusion or delayed fusion of the lateral folds (primarily the rectus abdominis)
What disorders have been associated with congenital ventral abdominal hernias?
search for other congenital defects
Fucosidosis
Ectodermal dysplasia
Cryptorchidism
What initially protects and omphalocoele?
Omphaloceles are large midline umbilical and skin defects that permit abdominal organs to protrude from the body.
- A thin transparent membrane of amniotic tissue
What is gastroschisis?
A congenital ventral abdominal hernia, very similar in appearance to an omphalocoele but it is paramedian
What breeds are predisposed to umbilical hernias?
Airedale terriers
Basenjis
Pekingese
Pointers
Weimeraners
treatment
- large umbilical or supraumbilical abdominal wall defects, especially when incomplete caudal sternal fusion is present, are specifically examined for other congenital diaphragmatic or cardiac defects before correction.
- suspicious of entrapped viscera and obstruction when animals present with acute gastrointestinal signs (vomiting, anorexia) and a firm, irreducible, painful umbilical mass
- further examined by radiography, ultrasonography
sx
- small (<2 to 3 mm) hernias are treated conservatively because spontaneous closure has been reported as late as 6 months of age
- neutered because of genetic predisposition
- approximate the size of intestine (one-finger size in a small- to medium-sized dog) sx is indicated
- sac is dissected free
- If fat alone > neck is ligated, sac and contents are excised.
- Small sacs can be inverted into the abdomen.
- Marginal debridement of wound edges or rectus sheath is generally recommended
- Umbilical hernias containing abdominal organs may require more extensive surgery.
- incarcerated hernias without strangulation, the hernial sac is dissected free, The hernial ring is enlarged to release the contents into the abdomen, sac is excised, and the hernial ring is debrided,
- Most umbilical hernias can be primarily closed
- Fascial releasing incisions can be made to reduce tension on the primary suture line (at least 2 cm away from the defect and through the external rectus fascia only) fascia is elevated and shifted toward the midline
-
Caudal Abdominal Hernias
- inguinal
- scrotal
- femoral
- inguinal region are categorized as direct or indirect
- Direct hernias are usually large, and most do not cause incarceration or strangulation
- scrotal hernia—an indirect inguinal hernia
- inguinal hernias are more common in female dogs
- more unilateral inguinal hernias occur on the left side than on the right
- Large hernias may contain a gravid uterus (hysterocele), bladder, or jejunum
- vomiting for 2 to 6 days before admission predicted a strangulated small intestine.
- risk for strangulated intestine in dogs with long-standing inguinal hernia is less than 5%.
What are the 2 broad classifications of inguinal hernias?
- Indirect - hernia contents enters the cavity of the vaginal process
- Direct - Hernia contents pass through the inguinal ring adjacent to the vaginal process (less common)