Unit 3 - Environmental and Disaster Flashcards

1
Q

What is a disaster?

A

A serious disruption of the funcitoning of a community or society causing widespread human, material, economic, or environmental losses which exceed the ability of the affected community or society to cope using its own resources

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the types of disasters?

A

Natural - weather related, earthquake, fires, volcanic activit y

Man facilitated - algal blooms, botulism

Man-made - spills, nuclear incidents, fires, explosions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What harmful events are caused by tornadoes?

A

Blunt force trauma, few common toxicological issues, molds

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What harmful events are caused by hurricanes?

A

Flooding/drowning

Contamination of drinking water

Localized chemical spills

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What harmful events are caused by floods?

A

Similar to hurricanes

Contamination of drinking water - chemicals, heavy metals, bacteria, mycotoxins

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What harmful events are associated with droughts?

A

Increased risk of toxic plant ingestion

Stress on grain resulting in an increased mycotoxin risk

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What harmful events are caused by blizzards?

A

The normal feed isn’t available so there is an increased risk of toxic plant ingestion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is eutrophication?

A

The excessive concentration of nitrates and phosphates due to runoff from land

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Why is eutrophication bad?

A

It increases algae growth/blooms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How has the zebra mussel introduction resulted in an animal welfare issue?

A

The mussel increased Cladophora growth resulting in the release of toxin and its enterance into the food chain. Birds get toxin and get botulism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

T/F: Chemical spills are only harmful to animals.

A

False - there are human and animal concerns

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What species are commonly affected by crude oil spills?

A

Invertebrates, fish, reptiles

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What do crude oil spills do to reptiles?

A

Epidermal injury and respiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What do crude oil spills do to birds and mammals?

A

Loss of thermoregulation and buoyancy

Alimentary tract irritation

Hypoglycemia

Aspiration

Hemolysis (birds only)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

How do crude oil spills affect bird/reptile eggs?

A

Reduced hatching success

Teratogenic effects

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How do you stabilize animals affected by crude oil spills?

A

correct hypothermia, correct nutritional & hydration imbalances, acclimate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

How do you decontaminate animals after crude oil spills?

A

liquid dish detergent, minimize stress, avoid hypothermia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What is the leading cause of morbidity and mortality in burn patients?

A

Smoke inhalation

19
Q

What asphyxiant gases can be present in fires? What do they do?

A

CO2 and methane

Displace O2

20
Q

What corrosive gases can be present in fires? What do they do?

A

NH3 and HCl

Proximal airway damage and oxidant injury in lung

21
Q

What systemically toxic gases can be present in fires?

A

CO and cyanide

22
Q

What size particulates are cleared by the nasopharynx?

A

> 5 micrometers

23
Q

What does smoke inhalaiton do to the upper airway (MOA)?

A

Primarily thermal injury
Heat destroys epithelium - denatures proteins & activates complement
Production of ROS - inflammation, edema, further injury
Loss of cilia - infections

24
Q

What clinical signs are associated with upper airway smoke inhalation? When?

A

Stridor, dyspnea

Can be delayed up to 18 hours

25
Q

What lesions are associated with smoke inhalation of the upper airway?

A

Thermal injury of airways

Chemical irritation of RT

Systemic toxicosis

26
Q

What does smoke inhalation do to the tracheobronchial tree?

A

Mostly chemical injury

Bronchoconstriction

27
Q

What clinical signs are associated with smoke inhalation at the tracheobronchial tree?

A

Cough, wheezing, sooty secretions, dyspnea

28
Q

What lesions are associated with smoke inhalation at the tracheobronchial tree?

A

High protein transudate

Airway casts

29
Q

What clinical signs are associated with smoke inhalation at the lung level?

A

Delayed onset of dyspnea

30
Q

What lesions does smoke inhalation cause in the lungs?

A

Alveolar collapse, edema, surfactant loss, fibrin deposition

Neutrophil activation results in further injury and neutrophil depletion (more infection)

31
Q

What is the systemic response to smoke inhalation?

A

Onset - 24-48 hours
Hypermetabolic state from burns
Vascular leakage
Decreased O2 to GIT
Myocardial depression
Pro-inflammatory mediators
Antioxidant depression

32
Q

What causes delayed posthypoxic leukoencephalopathy? When does it occur? What lesions are associated?

Can you tell I am getting tired of writing flashcards?

A

Smoke inhalation (CO)
Onset - 1-3 weeks post ‘recovery’
Die off of neurons - laminar cortical necrosis, reactive gliosis

33
Q

What are the risk factors for working dogs?

A

Exposure to hazardous substances - illicit drugs, accelerants, contaminants

Environmental hazards - zootoxins, plant toxins

34
Q

What are the risk factors for dogs at urban diseaster sites?

A

Low to ground

Lack of protective gear

Confined spaces

Highly contaminated environment

35
Q

What are the sources of oral exposures in the environment?

A

Puddles, deposits, and inhaled particulates

36
Q

Oral exposures can result in what?

A

Local injury and systemic absorption

37
Q

What are the possible respiratory exposure methods that are of significant concern at urban disaster sites?

A

Dust, ash, gases, toxic fumes

38
Q

What are the solid and liquid tox hazards at disaster sites?

A

Toxic metals

Soaps and detergents

Hydrocarbons

Glycols

Phenols

Alcohols

Drugs and medications

39
Q

What are the particulate hazards at urban disaster sites?

A

Asbestos

Heavy metals

Mold spores

Hydrocarbons

Glycols and solvents

Nontoxic dust

40
Q

What is key to reducing the exposure to toxic gases?

A

Ventilation

41
Q

What are the sources of polychlorinated biphenyls?

A

Coolants for transformers

Extenders in paints and pesticides

Lubricants

42
Q

What type of compounds are polychlorinated biphenyls?

A

Synthetic chlorinated organic compounds

43
Q

What on site management needs to occur in disaster situations?

A

Know the expected toxicants

Frequent decontamination

Allow for downtime

44
Q

I feel like everything else in this ppt is common sense….

A

I stopped on the ‘on-site’ management slide