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KSUCPM REHAB > TENDINOPATHIES TERMS > Flashcards

TENDINOPATHIES TERMS Flashcards

1
Q

inflammation of the tendon and results from micro tears that happen when the musculotendinous unit is acutely overloaded with a tensile force too heavy or too sudden

A

tendinitis

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2
Q

a degeneration of the tendons collagen in response to chronic overuse

A

tendinosis

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3
Q

name this classification of tendon

inflammatory in nature with acute and chronic stages

A

tendonitis

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4
Q

name this classification of tendon

tendon degeneration
absence of inflammatory cells
hypercellularity
neovascularization
increased proteoglycans
increased type III collagen
collagen bundle separation
A

tendonosis

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5
Q

name this classification of tendon

aka tenosynovitis
inflammation of paratenon
inflammatory cells present
hypervascularity 
fibrinous exudate in sheath
myofibrolblast in wound
crepitation
A

paratenonitis

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6
Q

this is inflammatory in nature
involves cardinal signs of Inf
Inf. cells present
pronounced tears/damage

Itis or Osis

A

Itis

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7
Q

non-inflammatory in nature
lacks cardinal signs of inflammation
no Inf cells present under microscope
lacks evidence of large scale damage

itis or osis

A

Osis

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8
Q

inflammation of only the tendinous sheath

A

paratenonitis

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9
Q

paratenon inflammation with intratendinous degeneration

same as paratenonitis BUT with decreased tendon collagen, fiber disorientation, scattered vascular ingrowth

clinical exam same as paratenonitis but with nodule formation

A

paratenonitis with tendonosis

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10
Q

intratendonous degeneration with atrophy

non inflammatory collagen degeneration, fiber disorientation, hypocellularity, necrosis and calcifications

nodular appearance, little if any edema

A

tendinosis

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11
Q

caused by improper ability of tissues to manage load/volume

tendon injuries account for up to 50% of all sport related pathology

A

tendonopathy and overuse injuries

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12
Q

name this tendon function

regular collagen
minimal vascularity
spindle shaped tenocytes

A

normal tendon

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13
Q

name this tendon function

disorganized type III collagen
neovascularization/neuronal ingrowth
round tenocytes
abundant ground substance

A

tendonopathy

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14
Q

name this loading type

bodyweight, jumping, running, cycling

A

cyclical

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15
Q

name this loading type

weight training

A

mechanical

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16
Q

name this Theory of Tendon Injury

• Oldest and Most Open To Challenge

• Normal Collagen Fibers Can’t Tear In Vivo Without Substantive
Alterations In Non-Collagenous Matrix

• Collagen Tearing and Remodeling Does Not Occur As A Result of
Loading

A

Collagen Disruption Model

17
Q

name this Theory of Tendon Injury

• Classic Inflammation Following Tendon Laceration

• Inflammatory Cells Observed In Pathologic Tendons But Not In
Traditional Inflammatory Response

A

Inflammatory Model

18
Q

name this Theory of Tendon Injury

• Tenocytes Responsible For maintaining Extracellular Matrix In
Resposne to Environment

• Tendon Load Changes Are Sensed and Trigger Cascade Response

• Understimulation of Tendon Due to Lack of Loading Plays A Role In
Degeneration

A

Tendon Cell Response Model

19
Q

what is this classification of tendon injury

  • Non-Inflammatory Proliferative Response In The Cell and Cell Matrix
  • Occurs Due tO Acute Tensile or Compressive Loads
  • Results IN Short-Term Adaptive Thickening of Focal Areas
A

Reactive Tendon

20
Q

what is this classification of tendon injury

• Describes the Attempt of Tendon Healing Similar To Reactive
Tendonopathy But With More Matrix Breakdown

  • Increased Number of Cells Causes increased Pr Production
  • More Focal and Matrix Related Changes Than Reactive
A

Tendon Disrepair

21
Q

what is this classification of tendon injury

• Cell Death Due To Apoptosis, Trauma, Tenocyte Exhaustion

  • Acellular Areas, Large Areas of Disordered Matrix, Little Collagen and Matrix Breakdown
  • Little Capacity For Reversal!
A

Degenerative Tendon

22
Q

name this clinical presentation

Younger (18-25)
Rapid Onset
Extreme Load
Easily Aggravated
PAINFUL
UNCOMMON
A

Reactive

23
Q

name this clinical presentation

Older(40-60)
Past Hx
After Overload
Variable Swelling
PAINFUL
VERY COMMON
A

Reactive on DEG

24
Q

name this clinical presentation

Older(30-60)
Long Hx
Swelling/Lumps
Unloading/Atrophy
NOT PAINFUL
COMMON
A

DEG

25
Q

name this treatment strategy that utilizes load principles and contiuum

  • 5x45Seconds With One Minute Rest
  • -Decreased Pain 45 Minutes Post Exercise
  • Implementable During Other Training
  • Good For Reactive Tendons
  • Mid-Range Contractions
A

Isometrics

26
Q

name this treatment strategy that utilizes load principles and contiuum

• 3x15 Reps, unilateral, slow 3 second contractions, 2 minutes rest
between sets
• 2 Exercises
• -Heel Drops Knees Straight
• -Heel Drop Knees Bent
• Twice Daily
• 7 Days/Week
• 12 Weeks
A

Eccentrics

27
Q

name this treatment strategy that utilizes load principles and contiuum

  • -3x Per Week
  • -3 second concentric and 3 second eccentric contraction (6 total seconds)
  • -RPE 8 on Last 2 Reps
  • Week One 3x15 RM
  • Week Two-Three 3x12 RM
  • Week Four-Five 4x10 RM
  • Week Six-Eight 4x8 RM
  • Week Nine-Twelve 4x6 RM
  • 3 Two Legged Exercises
  • -Seated Heel Raises-Bent Knee
  • -Leg Press Heel Raises-Knee Straight
  • -Forefoot Elevated Toes Raises With Bar
A

HSR

KSUCPM REHAB (51 decks)

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