TENDINOPATHIES HIGH YIELD Flashcards

1
Q

what is the acute stage of tendonitis?

A

inflammatory cells

hypervascularity

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2
Q

what is the chronic stage of tendonitis?

A

collagen disorganization

fibroblast proliferation

edema

fat accumulation

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3
Q

this is also known as tenosynovitis?

A

paratenonitis

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4
Q

tendonitis, tendinosis, paratenonitis equals?

A

specific pathological conditions

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5
Q

what is tendonopathy?

A

generic descriptor of clinical conditions in and around tendons

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6
Q

name this condition?

histologically
inflammatory cells in the paratenon/peritendinous areolar tissue

clinical exam
warm, edema present, point tender, paratenon thickened and adhered to normal tissues

A

paratenonitis

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7
Q

name this condition?

histologically
same as paratenonitis BUT with decreased tendon collagen, fiber disorientation, scattered vascular ingrowth

clinical exam
same as paratenonitis BUT with nodule formation

A

paratenonitis with tendonosis

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8
Q

name this condition?

histologically
non inflammatory collagen degeneration, fiber disorientation, hypocellularity, possible necrosis and calcifications

clinical exam
nodular appearance, little if any edema

A

tendinosis

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9
Q

reactive tendinopathyt can lead to this type of tendinopathy when the load is excessive?

A

degenerative tendinopathy leading to a rupture

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10
Q

strains are what type of overload and base?

A

acute overload

intensity based

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11
Q

tendonopathy are what type of overload and base?

A

chronic overload

volume based

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12
Q

tendon injuries account for what percent of all sports related pathology?

A

50%

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13
Q

what are the tendon responses to loading types, 2 types?

A

cyclical

mechanical

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14
Q

how does mechanical loading and collagen synthesis work?

A

intern signaled when under strain

release of collagen mRNA and TGF-8-1 and types I/III new collagen synthesized

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15
Q

exercise concepts to consider when treating tendonopathies?

A

tendons do not like compression

high response to mechanical loading

increased stiffness after resistance

chronic habitual loading needed

an effective training program with high loads

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16
Q

what are the 3 theories applying science to tendon injuries?

A

collagen disruption model

inflammatory model

tendon cell response model

17
Q

how are tendon injuries classified?

A

reactive

tendon disrepair

degenerative

reactive on
degenerative

18
Q

what are the two clinically relevant stages?

which ones are reversible/nonreversible

A

reactive/early disrepair
-reversible

late disrepair/degenerative
-nonreversible

19
Q

what are the treatment strategies utilizing load principles and the continuum?

A

isometrics

eccentrics-GOLD STD Alfredson Protocal

HSR-Heavy Slow Resistance
Beyer Protocal

20
Q

Eccentrics is what protocol?

A

alfredson protocal

21
Q

HSR is what protocol?

A

Beyer protocal

22
Q

where do palliative treatments come into play?

A

laser
massage
IASTM
dry needling

23
Q

what is important for endurance athletes?

A

strengthening

24
Q

this type of tendon does not like compression

stretch-shortening acommodates for this until tendon is no longer reactive

A

reactive tendons

25
Q

injured are a quick fix, T/F?

A

F

26
Q

how long should you be exposed to fluoroquinolone?

A

6 months

27
Q

what type of treatment modality is preferable in non-athletes?

for the reactive stage/in-season?

A

eccentrics

isometrics