READING: The weak neonatal foal Flashcards

1
Q

What diseases can result in a weak foal? 6

A
Sepsis
Neonatal encephalopathy/NE
Prematurity/dysmaturity
Neontal isoeryhtrolysis/NI
Uroperitoneum
MSK abnormalities (e.g. tendon contracture)
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2
Q

What may precocious lactation suggest? 2

A

placentitis or twinning

puts foal at risk of FPT

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3
Q

How heavy is an abnormal placenta?

A

> 11% foal body weight –> suggests placentitis –> may lead to NE or foal sepsis

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4
Q

When might crackles be normally heard in foals?

A

if foal has been laterally recumbent for several hours, crackles may be audible over the dependent lung, associated with inflation of the atelectatic lung

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5
Q

When does foal first urinate?

A

usually within first 12 hours of birth (very dilute, due to diet, should have a USG >1.0.20.

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6
Q

When should meconium be passed?

A

within first few hours, enemas not routine (some foals sometimes strain)

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7
Q

Another name for Neontal encephalopathy = ?

A

hypoxic-ischaemic encephalopathy.
initially hyperreactivity
can progress to depression and possibly seizures (focal or generalised)

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8
Q

When might bradycardia occur?

A

severe septic shock
hypothermia
hypoglycaemia
hyperkalaemia (e.g. ruptured bladder)

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9
Q

What heart murmurs are abnormal?

A

murmurs that are grade 5-6/6, that are bilateral or are accompanied by clinical signs such as cyanosis may suggest a congenital heart anomaly

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10
Q

What might poor pulse quality suggest? 2

A

hypotension as a result of septic or hypovolaemic shock

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11
Q

T/F: neonatal foals rarely develop a cough or nasal discharge in conjunction with lower respiratory tract disease

A

True

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12
Q

When should you treat suspected hypovolaemia?

A

If cool extremities, poor peripheral pulses, prolonged CRT, tachycardia, depression

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13
Q

‘On the farm’ treatment - suspected hypovolaemia

A

polyionic, crystalloid fluids (Hartmann’s, Lactated Ringers or Normosol-R)

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14
Q

‘On the farm’ treatment - suspected sepsis

A

BS parenteral ABs (e.g. cephalosporins, penicillin/ aminoglycoside combination)

Plasma - use in septic foals even if normal IgG at 24 hours. Septic foal will ‘use up’ immunoglobulins (generally each L of plasma with increase the IgG concentration by 2g/L

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15
Q

True/false: drug uptake from muscle may be compromised in hypovolaemic/septic foals

A

True

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16
Q

What should be assessed in foals treated with aminoglycosides?

A

renal function

17
Q

How should you treat hypoglycaemic foals (blood glucose < 3mmol/l)?

A

5% or 50% glucose solutions
Using 50% glucose, add 10-20ml to each litre of resuscitation fluid. In hospitals, give as continuous infusions (to avoid hyperglycacemia, glucosuria and possible electrolyte derangements associated with rapid infusion)

18
Q

When should you refer a weak neonatal foal? 10

A
IF:
seizuring
no response to initial fluid boluses
GI dysfunction --> inability to nurse
Ruptured bladder
Suspected septic arthritis
Respiratory distress
Severe diarrhoea
Recumbent foal
Suspect NI
Prematurity
19
Q

How to treat seizuring foal?

A

Diazepam. (repeated doses can lead to respiratory depression)

20
Q

What is indicated if a foal still shows signs of hypotension/hypovolaemia despite fluid resuscitation?

A

that the foal is suffering from severe hypovolaemic/ septic shock and will likely need further support (e.g. dobutamine)

21
Q

What is seen on abdominal ultrasound when a foal has a ruptured bladder?

A

large volumes of anechoic peritoneal fluid

bladder may still be visible, especially if tear is dorsal

22
Q

Initial treatment - ruptured bladder

A

0.9% NaCL (classically hyponatraemic and hypochloraemic, may be hyperkalaemic if HR is lower than expected)

23
Q

Treatment -severe diarrhoea

A

IV fluids, plasma and AMs

not difficult to administer but is time intensive so often requires hospital management

24
Q

For a blood transfusion to a foal, when can you use the mare’s serum?

A

after the mare’s RBCs have been washed