Pig reproduction and infertility 1+2 Flashcards

1
Q

What is the pigs/sow/year target?

A

22-26, perhaps 28 is target (Depends on type of unit); consists of number of litters/year (aim for 2-2-2.4, some farms will do 2.6), litter size (12-14) and pre-weaning mortality (<10%).

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2
Q

When do gilts reach puberty?

A

6-8 months old, need to achieve a minimum weight of 120 kg

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3
Q

When do you first mate a gilt?

A

2nd or 3rd oestrous (if you wait for a later litter, you tend to have a slightly larger litter size, 3rd is optimal)

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4
Q

How long is the lactation?

A

28 days (legal requirement, if you can fulfil certain criteria, you can wean earlier, perhaps 3 weeks).

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5
Q

What is the WOI?

A

3-5 days later. WOI = weaning to oestrous interval

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6
Q

When are sows culled?

A

Culled = after 6 litters (some companies beginning to say their sows can last 8 litters)

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7
Q

What is the MRP sign in pigs?

A

Oestradiol from the piglet

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8
Q

When does a gilt/sow ovulate?

A

Ovulates 2/3 of the way through oestrous (48-72 hours).

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9
Q

How do you induced farrowing?

A

Prostaglandin and oxytocin, work out farm gestation length (2-3 days before expected farrowing, not before otherwise you induce abortion and have dead piglets)

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10
Q

Define stillbirth

A

some farmers say anything that dies in first 24 hours is stillbirths when it is actually a neonatal death – bear this in mind when investigating farm records. Vet definition tends to be an animal that is not born alive/never lived outside the sow. Ask for definitions of farm’s measurements.

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11
Q

What is a mummified foetus?

A

don’t macerate because it is a sterile environment, leathery – water absorbed around them, there can be an infectious cause for this, ok to have the odd one of these but no more

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12
Q

What do regular and irregular returns to oestrous suggest?

A

regular – an indicator of fertilisation problem such as boar problem

irregular - management problem such as incorrect AI timing OR early embryonic loss

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13
Q

What is an average cull rate?

A

40% sows/year

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14
Q

What proportion of abortions have an infectious origin?

A

30-40%

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15
Q

What is suggested by mummified foetuses/stillbirths/ weak pigs of all one size?

A

there has been a single insult to the uterine environment (stress, fever)

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16
Q

What is suggested by mummified foetuses/stillbirths/ weak pigs of differing sizes?

A

consider this prolonged damage to foetuses. Think infectious causes – commonly viral – parvo, PRRS and CSF etc. (e.g. virus that infects the foetuses but doesn’t give the sow a fever)

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17
Q

List common (4) and uncommon (3) infectious causes of abortion.

A

o Commonly: parvovirus, PRRS, leptospirosis, erysipelas

o Uncommon but severe: Aujeszky’s, ASF, CSF

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18
Q

List the marker stages of gestation

A

o < 35 days of gestation – most absorbed
o 35-40 days – ossification begins
o Foetus dying > 35 days – mummified foetuses through to stillborn piglets (aborted at any stage)
o 70 days foetus immunocompetent, look for evidence of an infectious cause
o Full term at 112-116 days

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19
Q

List non-infectious causes of abortion

A

o Husbandry and management – stockman, hygiene, policies (age structure of the herd)
o Environment – season, heat stress.
o Genetics – parental/ progeny
o Nutrition - micronutrient deficiencies (Vit E and A)
o Toxic agents - misuse of hormonal drugs, chemicals (teratogens in hemlock), mycotoxins (i.e. Zearalenone)

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20
Q

Define SMEDI. Cause?

A

Stillbirth mummification, embryonic death infertility

Majority of these are caused by porcine parvovirus, (porcine enteroviruses are less frequently implicated)

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21
Q

Classical signs of SMEDI

A

Full term litter consisting of small mummified foetuses, full grown stillborn, live weakly piglets, ***RARELY CAUSES ABORTION

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22
Q

Transmission - SMEDI - 2

A

oronasal and venereal

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23
Q

Treatment - SMEDI

A

none

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24
Q

Control - SMEDI

A

vaccination is available and widely practised. Vaccinate 8 weeks before service on first occasion and 2 weeks before for subsequent boosters. It is actually a good thing to have a few cases (but not too many) of mummified foetuses on a farm because it shows the virus is still circulating and that the vaccine is working. Manufacturers say you need to vaccinate before serving, but you don’t need to do this.

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25
Q

Causes of sequential embryonic death

A

usually viral.
PRRS,
Swine influenza virus (SIV),
erysipelas (skin diamonds, placental vasculitis), leptospirosis (debate over importance),
PCV-2 (unknown role in sow reproductive performance).
NOTIFIABLES – AD, CSF, ASF.

26
Q

What was the original name for PRRS?

A

Blue ear disease

27
Q

Clinical signs - PRRS

A

reproductive losses, increased pre-weaning mortality, severe respiratory effort in neonates (thumps), blue extremities such as ears (<1% cases, not pathognomic), flu-like signs in older pigs. Immunosuppressed animals are very susceptible to many secondary pathogens.

28
Q

Control - PRRS

A

originally notifiable, now reported throughout the UK, vaccine available since 2001. Vaccination against PRRS (when present on farm) has decreased mortality seen with PMWS.

29
Q

Clinical signs - leptospirosis

A

most commonly abortion and reproductive failure

30
Q

Diagnosis - leptospirosis

A

serology, dark ground microscopy, FAT

31
Q

Treatment - leptospirosis

A

AB medication (Streptomycin, TCs). In theory the whole herd should be treated at one time, this rarely happens. Recent moves towards the use of cattle lepto vaccines in pigs.

32
Q

What are pigs vaccinated against?

A

parvovirus for reproductive herds, erysipelas – definitely for outdoor units, PCV-2

33
Q

How do you estimate the gestation length with a foetus?

A

= 21 + (3 x crown/rump length (cm)

34
Q

How does infection in utero affect piglets?

A

causes foetuses to die at different gestational ages

35
Q

When is the foetus immunocompentent?

A

By day 70 gestation

36
Q

What should foetal fluids be tested for?

A

parvovirus Ab, Leptospira bratislava Ab and swine influenza (new serotypes?). Also can do antigen detection tests (parvo and lepto).

37
Q

What does a VLA Pig Abortio Kit allow you to test for?

A

swine influenza, erysipelas, parvovirus, PRRS, leptospira

38
Q

Treatment - vaginal prolapse

A

replace and hold in place with purse string suture or Buhner suture. Use ABs and NSAIDs to reduce infection and swelling

39
Q

Causes of swollen/damaged vulva

A

May follow vulva biting, trauma related to dystocia, farrowing crate injuries (if too small), zearelenone toxicity (oestrogenic, tends to be a herd problem

40
Q

What does brown staining of the trachea indicate?

A

meconium lost during parturition and the pig has inhaled it. Suggests piglet died during parturition

41
Q

When does piglet fostering need to be done?

A

within first 24 hours

42
Q

What is the most common cause of death in piglets?

A

crushign

43
Q

How can piglet birth weights affect mortality?

A

birth weights <0.75kg are unlikely to live

44
Q

What is ‘splayleg’ in piglets?

A

congenital abnormality: affected piglets do the splits with their back legs. Considered a muscle weakness problem in adductor muscles in heavy male piglets.

45
Q

What is epitheliogenesis imperfecta?

A

raw patches of flesh with skin curled up at the edges on new-born piglets. Piglets may die or recover. Must be differentiated from other pig wounds. Possibly inherited so use different boar on sow at next mating.

46
Q

How do you diagnose primary uterine inertia?

DDx?

A

absence of straining, cervix is dilated, no obstruction present, lack of uterine tone.

DDx: secondary uterine inertia

47
Q

Treatment - primary uterine inertia

A

assist at farrowing, oxytocin, small amounts regularly, not a big bolus, 2-5 international units (0.2-0.5ml), IM, give at 30 minutes interval. If toxaemia/infection present give ABs and/or NSAID. Check for blockage at the bifurcation.

48
Q

Cause of secondary uterine inertia

A

obstruction caused by: malpresentation, 2 foetuses together, distended bladder, vaginal prolapse, vulval haematoma.

49
Q

Treatment - secondary uterine inertia

A

correct malpresentation, manually deliver foetus, once obstruction is relieved give 1-5 IU of oxytocin IM. CS are performed? Economically justified.

50
Q

Treatment - uterine prolapse

A

euthanize or immediate on-farm slaughter. Cross-foster piglets.

51
Q

Causes - downer sow

A

Many causes, include: lactation osteoporosis  femur/hip fracture, muscle weakness, apophysiolysis (separation of epiphysis from the bone shaft?), rupture of lesser trochanter

52
Q

Treatment - downer sow

A

cull if fractures present, move onto deep straw or put straw rubber matting under sow to prevent sores, encourage movement a few times a day

53
Q

Clinical signs - hyocalcaemia

A

recumbency, coma, death, eclampsic form with convulsions, rapid response to calcium borogluconate IV/SC

54
Q

What are the main causes of agalactia? 4

A

Failure to let milk down
Hot painful immature glands with normal milk
Ergot poisoning
Water deprivation

55
Q

Treatment agalactia - failure to let milk down

A

inject oxytocin (10IU) once

56
Q

Treatment agalactia - Hot painful immature glands with normal milk

A

NSAIDs. Repeated injections (every 3 hours). Can take > 3 days to resolve so will need to supplement affected litters.

57
Q

How much water do sows need a day?

A

25-40L

58
Q

Causal agents - coliform mastitis

A

: coliform bacteria, Pseudomonas, Enterobacter, Citrobacter and Morganella. Most commonly E.coli or Klebsiella. Also environmental pathogens possible

59
Q

Treatment - coliform mastitis

A

AB (potentiated sulphonamides) required for at least 305 days, NSAIDs, oxytocin encourages milk flow. Supply supplementary feeding for the surviving litter.

60
Q

Cause - pyogenic mastitis

A

Stapylococci or Streptococci (also Actinomyces)

61
Q

Clinical signs - urinary tract disease - pyelonephritis / cystitis

A

haematuria, pyrexia, bloody vulval discharge, fibrin, pus and blood when urinating, inappetence, depression, death.

62
Q

Cause - urinary tract disease - pyelonephritis / cystitis

A

the bacterium Actinobaculum suis found in the prepuce of most boars > 10 weeks old (previously named Eubacterium suis, Actinomyces suis, Corynebacterium suis).