Quiz 1-rapidly progressive cresentic glomerular nephritis Flashcards

1
Q

Type I anti-GBM

A

devotement of aggression against basement membrane (type II hypersensitivity)E.G. Good pasture’s syndrome

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2
Q

Good pasture’s syndrome

A

attacks alveolar basement membrane=hemoptysis coughing up blood causing exudates to prevent air exchangeattacks glomerular basement membrane= destruction of tissue around the membranes involves both respirator system and kidneys

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3
Q

CURABLE VIA PLASMAPHERESIS

A

good pasture’s syndrome

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4
Q

Type II- immune complex deposition

A

type III hypersensitivity1. systemic lupus erythematosus2. Henoch-Schonlein Purpura

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5
Q

systemic lupus erthyematosus: TMT and survival rate

A

90% survival rate, corticosteroids and immunosuppressants

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6
Q

systemic lupus erthyematosus

A

attacks antigens in cell cytoplasmwomen, 20s african descentinvolves htn which may lead to stroke, butterfly rash,

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7
Q

lupus nephritis

A

involvement of kidneys, vasculitis of cerebral vessels

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8
Q

Henoch-Schonlein purpura

A

hemorrhagic vasculitis usually in boys around 20s1. abdominal syndrome2. cutaneous syndrome3. articular syndrome4. crescentic glomular nephritis (kidney syndrome)

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9
Q
  1. abdominal syndrome2. cutaneous syndrome3. articular syndrome
A

90% survival rate with these syndromes70% with kidney syndrome

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10
Q

Type III - Pauci-immune

A

most dangerous and characterized by vasculitis and anti-neutrophil pasmocytic auto-antibodieswegener’s granulematosuspolyarteritis nodosaIGA Nephropathy (Berger’s disease)

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11
Q

most common glomerular disease in worldwide

A

Berger’s disease

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12
Q

patient will die due to renal failure or other secondary complications

A

wegeners granulematosus

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13
Q

necrotizing vasculitis of upper and lower respiratory tracts and melting of bond on face

A

wegener’s granulematosus (Type III Pauci immune)

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14
Q

characterized by vasculitis in vessels throughout the body. This can eventually lead to pouching and obstruction of lumen. DOES NOT OCCUR IN LUNGS AND ARCH OF AORTA

A

polyarteritis nodosa

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15
Q

hematuria following acute respiratory, intestinal or urinary infection. IGA deposits everywhere and if not controlled can manifest to chronic glomerulonephritis

A

IGA Nephropathy

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16
Q

associated with raynaud’s syndrome and smokingvascular phlebitis in veins. results in amputation

A

thromboangitis obliterans “burger disease”