Pulm Flashcards
spirometry
Measures flow rate and volume in inhalation and exhalation
Measures respiratory capacity
FEV1 baries based on
gender, age, height, ethnicity
sx asthma
vary, seasonal/allergies, wheezing, cough, breathlessness, chest tightness/discomfort
allergies and nasal polyps, accessory muscle use, hyperexpansion of chest and hunched shoulders, tachycardic, pulsus paradoxus, wheezing, long expiration
asthma
Spirometry will show obstructive pattern with bronchodilator (BD) response (witches hat)
Allergy testing, chest xray and bronchial provocation
intermittent asthma meds
No daily meds just SABA PRN
mild persistent asthma meds
Low dose inhaled steroid. If that does not work, then use a leukotriene modifier such as theophylline
moderate persistent asthma meds
Low dose inhaled steroid and LABA or medium dose inhaled steroid. If that does not work, give low dose inhaled steroid and leukotriene modifier such as theophylline
severe persistent asthma meds
High dose inhaled steroid and LABA
acute management asthma meds
immediate SABA (albuterol). If no complete response within 20 minutes, five 3 more SABA trmts in within 1 hour. If still no response, give oral or IV corticosteroids. If it is still not working, admit pt
emerency asthma meds
give Epi
what vassines should asthma pts get
influenza, pertussis, and pneumonia
copd?
Chronic bronchitis + emphysema that leads to a fixed airway obstruction
copd complications
Resp. Infections (bronchitis and pneumonia)
Heart failure (hard to get air in lungs? Hard to get blood too)
Cardiac ischemia
ss copd
Daily productive cough 3 months or more for at least 2 yrs, high HgB, rhonchi, wheezing, peripheral edema, CXR hyperinflation and flat diaphragm, ABG and SpO2 are lower, Inc CO2 leads to respiratory acidosis and they compensate by metabolic alkalosis
dx copd
Spirometry- concave witches hat pattern
Peak expiratory flow is NOT a criteria like asthma
DLCO
CXR- hyperinflated and flat diaphragm on emphysema
CBC
Oximetry
ABG
Serum alpha 1 antitrypsin in pts under 45
trmt copd
Inhaled ipratropium (SAMA) with or without albuterol (SABA)
LABA or LAMA
Oral theophylline if not responsive to other meds
Lung transplant, lung reduction volume, bullectomy
copd exacerbation
IV ATB such as macrolides (azithromycin) in patient
emergency copd
breathing trmt with SAMA/SABA like albuterol and ipratropium, CPAP, BiPAP, intubation, vent support
copd vaccines?
Get flu and pneumonia vaccines
CF triad sx
COPD
Pancreatic exocrine insufficiency
High sweat electrolyte concentrations
Bacterial pneumonia caused by staph aureus then pseudomonas aeruginosa (not in many other conditions). Pseudomonas aeruginosa is impossible to get rid of
cf
dx cf
sweat chloride test
if ng get dna test- 7q
cf trmt
Minimize airway reactivity and infections
MC bacteria in bronchiectasis sputum are
H influenzae, pseudomonas aeruginosa, and strep pneumoniae
Sx bronchiectasis
dyspnea, wheezing, chest pain, crackles, productive cough
dx bronchiectasis
XCR- dilated airways and mucus plugging
CT- airway changes
MC of the interstitial lung diseases
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia is a dx of____
exclusion
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia sx
Hypoxemia, inspiratory crackles, tachypnea
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia dx
1st a Physical exam
2nd High resolution thin cut (HRTC) CT chest shows interstitial reticular linear changes and possible honeycombing
3rd Bloodwork to r/o rheum markers
4th Special studies
Bronchoalveolar lavage- r/o infections and malignancies
Transbronchial biopsy- r/o sarcoidosis, malignancy, TB
Surgical open lung biopsy- gold standard, but it’s riskier
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia trmt
high dose steroids
UIP- usual interstitial pneumonia (a type of IPF)
progressive scarring of the lungs
UIP- usual interstitial pneumonia (a type of IPF) trmt
steroids, Antifibrotic and immunosuppressive modulators Not responsive
Only definitive trmt is a long transplant
Idiopathic pulmonary fibrosis/ Idiopathic interstitial pneumonia can be caused by this medicine
amiodarone
pts taking amiodarone should get this frequently
pft
Pneumoconiosis sx
Sx: weak, pale, cyanotic, rhonchi, wheezing, crackles/rales (heard on inspiration), pleural friction rub
Pneumoconiosis dx
PFT- restrictive
DLCO- reduced diffusion
CXR- linear/reticular interstitial marlins in the lower and mid lung fields and calcified thickening along the chest wall/diaphragm, ground glass appearance
Note: lung biopsy not needed
Pneumoconiosis trmt
supplemental O2
Lung cancer and mesothelioma are huge concerns!
pneumonicosis
sarcoidosis?
Inflammatory disease with noncaseating granulomas
sarcoidosis cause
Unknown cause possibly propionibacter acnes
Mc in nordic population
sarcoidosis
sarcoidosis sx
Multisystem- need 2+ organs to dx
sarcoidosis dx
XCR- shows micronodular interstitial densities and nodular granulomatous changes
Chest CT- nodules and mediastinal hilar adenopathy
sarcoidosis trmt
Single organ: topical therapy
Multi-organ: oral steroids
sarcoidosis pts must have this test done
ophthalmic
pe ss
Ss: Sudden onset dyspnea, hypoxemia, tachycardia, light headed
pe dx
CXR normal
Chest CT with contrast (angio/spiral ct) shows small arterial emboli
VQ scan- old standard before CT, used only if pt has dye allergy
Serum D dimer should be positive
Ekg should be normal, but if you see S1Q3T3, it’s most likely PE
pe trmt with rhf and hypotension
thrombolysis or thrombectomy
pe trmt with normal r ventricle function
anticoagulation
Heparin drip
Anticoagulation for 6 months (or 3 if only DVT)