cardio day 2 PP1 Flashcards

1
Q

metabolic system need 3+ of the following

A

high wait circ. , high trigly, high bp, high fasting glucose, red HDL

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2
Q

angina cause

A

Anything that makes the heart muscle need more O2 such as cold weather, exercise, large meals, stress, abnormal heart rhythms, anemia, valvular disease, heart failure, hyperthyroidism

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3
Q

angina sx

A

Central tight pressure to squeezing, radiates to the arm or jaw, relieved with rest
Dyspnea on exertion, SOB, NV, weakness/fatigue, epigastric pain, indigestion, change in mental status

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4
Q

Leading cause of death in industrialized countries

A

ischemic heart disease

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5
Q

ischemic heart disease?

A

Ischemia (less blood supply) to the heart muscle caused by CAD and narrowing/hardening of the arteries

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6
Q

ischemic heart disease dx

A

Coronary angiogram
Lab tests
ECG
Stress test, nuc med test, exercise stress
Echo
Coronary CT angiogram, electoral beam CT (EBCT), cardiac MRI

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7
Q

Ranexa

A

for microvascular and chronic angina

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8
Q

goals stable angina

A

Prevent MI and death

Improve QOL

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9
Q

stable angina is resolves with

A

rest or nitroglucerin

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10
Q

Prinzmetal’s Angina?

A

Vasospasm induced angina that occurs in cycles, mainly at night

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11
Q

Prinzmetal’s Angina cause

A

Vasospasm- contractions of smooth muscle

NO CAD

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12
Q

Prinzmetal’s Angina dx

A

Inject coronaries with Ergonovine- vasospasm should occur and is diagnostic
ECG- ST elevation

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13
Q

Prinzmetal’s Angina trmt

A

Vasodilation with nitrate or CCB

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14
Q

cocaine use trmt

A

Benzodiazepines, nitrates, CCB

NO beta blockers, they cause alpha mediated vasoconstriction

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15
Q

cocaine use dx

A

stress test or cardiac cath

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16
Q

Syndrome X aka microvascular angina

A

Angina with dec blood flow to the heart
Vasospasm and/or small vessel disease with more pain perception
Coronary arteries look normal

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17
Q

dx Syndrome X aka microvascular angina

A

MRI may help confirm

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18
Q

trmt Syndrome X aka microvascular angina

A

beta blockers or nitrates

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19
Q

pathogenesis acs

A

Inflammation
Endothelial activation and fibrous cap is formed
Plaque and fibrous cap rupture causing prothrombin and thrombin to activate which obstructs blood flow
Platelets aggregate to form a thrombus

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20
Q

unstable angina dx

A

EKG changes

Normal cardiac markers

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21
Q

NSTEMI dx

A

High cardiac biomarkers- CK, CK-MB, myoglobin, troponin I

ECG- ST depression and T wave inversion

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22
Q

this acs has the highest mortality

A

stemi

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23
Q

TIMI risk score

A

stemi

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24
Q

stemi sx

A

May have LHF with hypoxia- dyspnea, hypoxemia, pulmonary edema, respiratory compromise
Ventricular arrhythmias- treat immediately

25
dx stemi
EKG- ST and J point elevation and hyperacute T waves
26
stemi statin trmt
atorvastatin 40 to 80 mg; Rosuvastatin 20-40 mg (know)
27
FFR-
fractional flow reserve
28
Bare metal stent (BMS)-
ASA lifelong and (DAPT) dual antiplatelet therapy minimum 3 months
29
Drug eluting stent (DES)-
ASA lifelong and (DAPT) dual antiplatelet therapy 12 months +
30
Antiplatelet therapy-
plavix, brilinta, effient
31
when should you do fibrinolysis in stemi
after pci
32
IABP- intra aortic counter pulsation
Percutaneous impella implant | Increases coronary filling and decreases afterload
33
Spontaneous coronary dissection
Tear in layers of coronary artery causing hematoma or dissection flap to obstruct blood flow
34
Spontaneous coronary dissection mechanism
unknown
35
dx Spontaneous coronary dissection
Coronary angiogram | IV US
36
trmt Spontaneous coronary dissection
Risk factor modification PCI- stent, CABG Not musch bc usually dx after death
37
cardiac troponins
Troponin C, I and T
38
high levels of cardiac troponins indicate
cardiac muscle cell death
39
tropinin 1
Raises 3-12 hours after onset of ischemia Peaks 12-24 hrs Lasts 7-14 days Must raise to a certain amount for it to be considered myocardial injury Minimally elevated levels suggest type 2 MI/“demand ischemia”
40
Heart score for chest pain pts in the ED
0-3 discharge 4-6 admit for observation 7-10 early invasive strategies
41
CARDIOGENIC SHOCK
Ventricles fail and blood does not perfuse organs | Less oxygen, vasoconstriction, hypotension
42
sx cardiogenic shock
Cold extremities, oliguria, change in LOC, weak pulse, mottled skin, hyperventilation, pulmonary edema, JVD
43
90% MI pts get this
Post infarction arrhythmias
44
Post infarction arrhythmias ?
Electrical conduction is affected after the MI Enhanced sympathetic activity, inc catecholamines, autonomic imbalance Arrhythmia depends on the area affected
45
Types: supraventricular arrhythmias, accelerated junctional rhythms, av blocks, IV blocks, ventricular arrhythmias, reperfusion arrhythmias, junctional bradycardia
Post infarction arrhythmias
46
Accelerated junctional arrhythmias
usually inferior MI
47
Sinus bradycardia
Inferior or posterior MI | 1.2 hrs after the MI
48
Sinus bradycardia trmt
IV atropine, dopamine, epi | Transcutaneous or transvenous pacer
49
AV blocks
1st and 2nd degree | Mobitz type 2 has the highest risk for a complete heart block
50
av block trmt
pacer
51
intraventricular blocks
HIS bundle | Usually anterior MI
52
Not treated with antiarrhythmic therapy
pvc
53
Risk of hemodynamic collapse
nonsustained vt
54
sustained vt
Amiodarone- antiarrhythmic drug | Electrolytes- K and Mg
55
post MI with revasc. when to give ICD?
wait 90 days
56
post MI without revasc. when to give ICD?
wait 40 days
57
highest risk in 1st 90 days after MI
scd
58
VF
Usually 1st hr after MI | After 48 hrs its usually associated with pump failure and cardiogenic shock