Prep 8 Flashcards
falls when shaving face
carotid sinus syndrome
ECG changes in COPD
Right axis deviation, RVH (hyperexpanded lungs, pulmonary hypertension stress on right side of the heart)
porminent p waves and low voltage QRS
aortic dissection ix
CT
ECG changes in autonomic neuropathy
loss of sinus arrythmia (no decrease in hr after taking a deep breath)
ECG changes in hypothermia
J waves
ECG changes in hypocalcemia
U waves
Side effect of CCbs
ankle odema
SEs of thiazide diuretics
gout (DCT block Na+-Cl−)
doxazosin
alpha 1 blocker
ACEI se and CI
dry cough, renal artery stenosis
Statin Se
muscle weakness and raise CK
BB SE
cold hands and feet
amphetamines SE
arrythmiass
On 3 antihypertensives and keeps fainting
iatrogenic
CO
SV x HR
chest lead at apex
V4
Defibrillation
VF or pulseless VT (immediately if witnessed cardiac arrest or after 2 mins of CPR is unwitnessed out of hospital)
Parasympa outflow
3, 7, 9, 10
rib notching on CXR
coarcation of aorta
collapsing pulse
aortic regurge
machine like murmer
patent ductus arteriosus
slow rising pulse
aortic stenosis
cresendo decresondo murmur
aortic stenosis
haemosideran staining
brown oatcthes in lower legs associated with venous stasis
marjolins ulcer
agressive ulcerating SCC
ABPI >1.2
calcified stiff arteries
calculate toe brachial index
ABPI <0.8
arterial disease (PAD) if <0/4 refer immediately for vascular opinion
cyclizine mechanism
antihistamine (antiemetic)
odansatron
5ht3 antagonist (antiemetic)
prochlorprazine, metoclopramide
antidopaminergic (antiemetic)
Hyoscine
anticholinergic (antiemetic)
ABGs in chronic antacid use
metablic alkalosis
ABGs in DKA
metabolic acidosis
BE value in metabolic
NEGATIVE
ABGs in hyperventilation
respiratory alkalosis
ABGs in meningococcal septicemia
Mixed resp and metabolic acidosis
ABGs in COPD exacerbation
respiratory acidosis with metabolic compensation
ABGs in vomitting
metabolic alkalosis
gum hyperpigmentation
synacthen test (addisons)
easy bruiding and puffy face
overnight dexamethasone test (cushings)
sweating, palpiations
urinary chatecholamines (phaochromocytoma)
woman collapses high potassium low sodium glucose 3.1 daughter says she has not taken medication
addisonian crisis (lack of aldosterone decreases Na reabsorption)
Diabetic drug causing hypos
SUs (glicazide)
Diabetic drug causing weight gain
SUs (glicazide)
Diabetic drug causing fluid retention
glitazones
swimming in lake malawi now haematuria
schistosomasis
back from kenya with fever
malaria
white raised patches on back of throat back from africa
HIV
legionella test
urine antigen
gardiella test
stool microscopy
viral hepatitis test
blood serology
rhythm control in AF
HF, first onset AF, reversible cause
amioderone if structural heart disease
flecainide without structural heart diseae (if >48 hour symtpoms anticoagulate for 3 weeks)
electrical cardioversion if unstable (anticoagulate for 4 weeks after)
lab findings in osteomalacia/rickets
low vit D low Ca low phostphate high PTH (released when low Ca, trying to restore Ca- secondary hyperparathyroidism) high ALP (osteoblast activity increased)
lab findings in hyperparathyroidism
high PTH (bengign adenoma, hyperplasia or malignancy of parathyroid galnad increasing pth production) high Ca (PTH increases Ca levels in the blood) Phosphate normal or low
what happens in tertiary hyperparathyroidism
in chronic secondary hyperparathyroidism tissue hyperplases.When the biocemical causing original problem is corrected the parathyrpid gland still produces lots ofPTH
lab findings in osteoporosis
normal (quantative issue not qulatative)
lab findings in pagets
ca, phosphate and PTH normal
ALP high
(FH, old, bone pain)
lab findings in bone mets
high Ca
low PTH
high ALP