pain sensation Flashcards
بسم الله عليه توكلت وعليه فليتوكل المتوكلون
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ماهو الألم
هو شعور غير سار يعمل رد فعل انعكاسي يخليك تزيل محفز او مثير الألم
عدد مستقبلات الألم
specific polymodal nociceptors FNE
mct
Mechnosensitive paine receptors
Chemosensitive pain receptors
Thermosensitive pain receptors 10 45
ايه هي وسيلة تحفيز المستقبلات الخاصة بالألم ياعم عمر ؟
هو انه بيطلع من منطقة ضرر النسيح مواد كيميائية
2k h 2p
Kinnis k+ histamine substance p proteolytics
+ chemosensitive pain receptors
and lowering the threshold of
Thermo and Mechanosensitive pain receptors
area without pain receptors is
painless structure
pain receptors are more in ?
less in and lesser in ?
Skin Arterires and cranial venous sinuses
Joint capsule and Parietal layer of periosteum
less in muscles
lesser in viscera
painless structures?
Brain Bone
Serous membrane visceral layer
alveloi of lung
Liver
Retina
Pain afferents?
-A delta and C fibers
Pathway of Pain
Lateral divison of Anterolateral system
adaptation of pain receptors
Very slow or non adapting
Tonic
types of pain
epicritic sfc sharp fast cortical
protopathic dst dull aching slow thalamic
mentio pain classes according to site
Cutanuous
Deep muscular
Visceral
كل واحد عاوزين التعريف والاسباب والخصائص
cutanuous pain causes mention 3
فاكر قانون فرق الجهد ^_^
v=3IR
Injury or inflammation of skin
Irritation of dorsal root nerves
Referred pain by the deep and visceral diseasd tissues
Compare between fast and slow cutanuous pain at characters
fast cutanuos pain : Acute sharp immediate pricking
Slow : Chronic burning aching throbbing
fast cutanuous pain at characters
fast cutanuos pain : Acute sharp immediate pricking
slow cutanuous pain at characters
Slow : Chronic burning aching throbbing
Compare between fast and slow cutanuous pain at receptors
Fast mainly mechanoreceptors and thermoreceptors
Slow Mainly chemoreceptors and other typer mechano and thermal
Compare between fast and slow cutanuous pain at fibers
fast =A delta
slow= c fibers
Compare between fast and slow cutanuous pain at nerotrasmitters
fast glutamte
slow substance p
Compare between fast and slow cutanuous pain at pathway
both by lateral spinpthalamic tract but
Fast : Neo-spinothalamic tract
slow : paleo spinothalamic tract
Compare between fast and slow cutanuous pain at perception
fast 90 in cortec and 10 in brainstem
slow 10 in cortex 90 in brain stem as:rtpt
1-Reticular formation
2-Tectum of mid brain
3-Peraqduecta gray area
4-non specific thalamic nuclei
slow cutanuous pain at perception
slow 10 in cortex 90 in brain stem as:rtpt
1-Reticular formation
2-Tectum of mid brain
3-Peraqduecta gray area
4-non specific thalamic nuclei
Compare between fast and slow cutanuous pain at site
fast skin
slow skin ( deep structures -viscera من أول لحظة )
Compare between fast and slow cutanuous pain at summation
fast non summation
slow summated as more receptors are stimulated
Compare between fast and slow cutanuous pain at localization
fast sharply localized
slow poorly localized
Compare between fast and slow cutanuous pain at onset
fast after 0.1 sec
slow after 1 second
Compare between fast and slow cutanuous pain at duration
fast 1-2 sec
Slow long may be untolerable
Compare between fast and slow cutanuous pain at COD ReAN P2S2L
كود ريان بصل
كود ريان بصل
كود ريان بصل
كود ريان بصل
Characteristics
Onset
Duration
Receptors
Afferents
Neurotrasmitters
Pathway
Perception
Summation
Site
Localiztion
Deep pain and it causes illustrate
Deep pain from deep structers as muscles
Causes are:
1-Injury to deep structures
2-inflammation to deep structures
3-Ischemia to deep structures
4-spasm to deep structures muscles
characters of deep pain show ?
As Slow cutanuous pain charcaters
throbbing burning chronic
and may be localized to one limb but not one point
and Cause depressor parasympathatic effect decreasing HR And ABP
اشرح الألم الاحتشائي
نوع من الألم العميق نتيجة نقص الامداد الدموي للتراكيب الداخلية زي العضلات
زي ما حصل في
Angina - Intermittent caludications
وده يحصل لي؟
-Decreased blood supply casues accumulation of metabolite as lactic acid and adenosine in tissue casuing pain
-tissue damage from ischemia release chemicals as kinins and histamins stimulating pain receptors
describe visceral pain
pain from visceral srtuctures
casues:
Injury
inflammation as peritoneal
irritation as peptic ulcer by HCL
Ischemia at visceral organ
Overdistension bladder and spasm ureter
اشرح لي SPASM &Overdistension
وهما بيعملوا الم حشوي ازاي
مثلا ضيق الحالب او تمدد قوي للمثانة
Mechnosnesitive pain receptors stimulation
Ischemia as occluding blood vessels
accumulating metabolites
Mention three charcters of Visscera lpain
It is as Slow cutanuous pain ya man
-Depressor parasympathatic effect
-Guardening rigidity
-Referred pain
dgr ضجر وملل
Fast fibers relay on —- but slow on —-
Lamina Marginalis
Substania gelatinosa
fast pain
slow deep visceral
flexor withdrawal reflex
Rigidity
pressor sympathatic effect
depressor parasympathatic effet
emotional saddnes
hyperalgesis زيادة الاحساس بالألم
الحمدلله رب العالمين
