Orthopaedics: Foot and Miscellaneous Flashcards

1
Q

What are the main conditions that can affect the foot?

A
  • Fractures
  • Luxations of joints
  • Pad injuries- corn
  • Foreign bodies
  • Nail injuries

Sesamoid disease

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2
Q

What is a corn?

A

Focal area of hyperkeratosis
* excess keratin, thickening of pad

Keratin is a structural fibrous protein

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3
Q

How is a corn treated?

A

Historically:
* Hulling/excision (primary cause not addressed- recur)

Regression:
* Silicone gel implants
* Revision surgery on distal amputations

If the pad is unloaded then the corn will grow out

Superficial digital flexor tendonectomy
* removal of 1cm + of tendon

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4
Q

How are fractures of metatarsals/metacarpals treated?
What are the potential complications?

A

Single
* External coaptation

Multiple
* III IV need internal fixation
* Dowel pinning
* Spider

Complications
* Extensive soft tissue injuries- check viability
* Synostosis between bones

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5
Q

What are indications for toe amputation?

What is the technique?

A

Severe luxations/fracture/neoplasia

Y-shaped incision to preserve the pad- can cut through joint

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6
Q

What is the scientific name for marie’s disease?

What kind of disease is it?

A

Hypertrophic Oesteopathy

Paraneoplastic bone disease

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7
Q

What nutritional bone diseases can develop in small animals?

A
  • Nutritional secondary hyperparathyroidism
  • Renal secondary hyperparathyroidisim
  • Hypovitaminosis D
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8
Q
  1. What is panostitis?
  2. What are the clinical signs?
  3. What bones are more commonly affected?
A
  1. Painful inflammation of the periosteum
  2. Classically shifting lameness, acute onset, morelikely forelimb
  3. Ulna > radius > Humerus
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9
Q

How is panostitis diagnosed?
How is it treated?

A

Diagnosis- signalment, history, clinical signs and radiographs (young large breed dogs)

Treatment
* Self limiting
* Excercise control/restriction
* Analgesics

Lack of definition between cortex and medulla (thumbprint opacities)
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10
Q
  1. What is the usualt signalment of metaphyseal osteopathy?
  2. What is the aetiology?
  3. What are the clinical signs?
  4. How is it diagnosed?
  5. How is it treated?
A
  1. Young, rapid growing breeds- 2-6mo
  2. Unknown
  3. Mild lameness to severe collapse- swelling
  4. Signalment, history, CS, radiography
  5. Self-limiting- supportive care, analgesics
Radiolucent line and increased radiodensity in metaphysis parallel to physis
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10
Q
  1. What is the usualt signalment of metaphyseal osteopathy?
  2. What is the aetiology?
  3. What are the clinical signs?
  4. How is it diagnosed?
  5. How is it treated?
A
  1. Young, rapid growing breeds- 2-6mo
  2. Unknown
  3. Mild lameness to severe collapse
  4. Signalment, history, CS, radiography
  5. Self-limiting- supportive care, analgesics

Possible anglular limb deformities

Radiolucent line and increased radiodensity in metaphysis parallel to physis
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11
Q
  1. What is craniomandibular osteopathy?
  2. What is the signalment?
  3. What are the CS?
  4. How is it diagnosed?
A
  1. Non-inflammatory, non-neoplastic proliferative bone disease
  2. 4-10mo, WHWT, scottish terrier, cairn terrier
  3. Mandibular swelling/thickening, inability to open mouth, salivation, anorexia, pain on eating
  4. Signlament, history, CS, radiography
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12
Q

Whar radiographic changes can be seen with craniomandibular osteopathy?

How is it treated?

A
  • Changes usually bilateral
  • Palisading proliferation on the mandible and tympanic bullae
  • Temporal, frontal and maxillary bones
  • Occasionally affects long bones

Treatment
* Supportive care
* Analgesics

Prognosis
* Self limiting at 11-13 months
* Euthanasia may be requested

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13
Q

What is the aetiology of marie’s disease?
What are the clinical signs?

A

Aetiology
* Paraneoplastic- secondary to intrathoracic or abdominal neoplasia
* Neural- ?

Clinical signs
* Lameness can develop over several months
* Single or multiple limbs
* FIrm swelling along bone of distal extremities
* Pain in early stages

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14
Q

How is maries disease diagnosed or treated?

A

Diagnosis
* History and clinical signs
* Thoracic radiographs and abdominal ultrasound
* Radiographic changes- periosteal new bone formation, increased bone density

Treatment
* Symtomatic
* Remove primary cause- resolution of new bone formation

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15
Q

What causes nutritional secondary hyperparathyroidism?

A
  • Diets high in phosphorous or low in calcium
  • Usually meat based diets
  • Ideally 1.2: Ca:P 1:1 in cats
  • Hypocalcaemia- increased PTH
16
Q

What are the clinical signs of Secondary hyperPTH?

How is it diagnosed?
How is it treated?

A

CS:
* Lameness
* Skeletal pain
* Swollen metaphysis
* Pathological fracture

Diagnosis- history and radiographs
* Decreased bone density/thinned cortices
* Mushroom shaped metaphysis

Treatment
Rest, diet correction, oral calcium supplementation, NSAID

Thin cortices with fractures
17
Q

Why does CKD cause increased PTH?

Why does impaired vit D cause increased PTH?

A

Impaired phosphate excretion
* Hyperphosphataemia- causes hypocalcaemia

Impaired vit D production causes depressed enteric calcium absorption- rickets

18
Q

What are the clinical signs of CKD osteopenia?

How is it treated?

A

CS
* Pliable mandible
* Loose teeth
* Skeletal pain
* Pathological fractures
* Bowing of bones

Treatment
* Reduce phosphate intake/phosphate binder
* Calcium or calitrol supplementation

19
Q

What is 1st, 2nd and 3rd degree ligament injuries?

A

1st- minimal tearing, rapid healing
2nd- partial rupture associated with haemorrhage and oedema
3rd- complete rupture- avulsion

Sprain is ligament, Strain is tendon

20
Q

How are different degree ligament injuries treated?

A

1st degree
* Healing is rapid- transient lameness
* 1 week rest- restricted excercise and NSAIDs

2nd degree
* Weight bearing- support for 3-4 weeks
* Excercise controlled for upto 3 months

3rd degree
* Not weight bearing, joint alterations
* Surgical repair, reattachment or replacement

21
Q

How are stains of tendons treated?

A
  • In acute phase need to reduce inflammation- alternate cold warm compresses
  • Surgical treatment for ruptures or displacement- monofilament nylon sutures
  • Immobilisation for 4-6 weeks post op
  • Gradual increase in excercise over the following 2 months
22
Q

How are tendons surgically treated?

What post-operative care is needed?

A
  • Early repair better
  • Monofilament nylon sutures used to attach ends

Imombilisation necessary for 4-6 weeks
* Splint for DFT/extensor tendons
* Transarticular external skeletal fixator
* Hock/ankle held in extension with bone screw for common calcanean tendon

23
Q

What extra support is needed after surgical repair of which tendon injuries?

A
  • Splint for digital flexor/extensor tendons
  • Transarticular external skeletal fixator
  • Hock/ankle held in extension with bone screw for calcanean tendon
24
Q

What does this image show?

A

Reversible contracture of the flexor carpi ulnaris muscle

25
Q

What is the signalment, clinical signs and treatment of reversible contraction of the flexor carpi ulnaris?

A

Signalment
* young dogs 6-8 weeks

Clinical signs
* flexed carpus that cannot be extended
* Tendon of FCU is taut

Treatment
* Resolution usually after 2-3 weeks
* Carpal supports
* FCU tendinecromy in rare cases