Exotics: Anaesthesia of Exotics Flashcards

1
Q

What are the defenitions of ASA 1-5?

A

ASA 1- normal healthy patient
ASA 2- mild systemic disease
ASA 3- Obvious systemic disease
ASA 4- severe systemic disease, constant treat to life
ASA 5- not expected to survive without the operation

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2
Q

What avian patients are of specific concerns?

A

No commonly kept
* Ratites
* Cranes
* Flamingos
* Charariforms (waders)
* Penguins
* Marine/diving birds

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3
Q

What are general considerations for avian anasethesia?

A
  • High incidence of chronic and subclinical disease
  • Susceptiple to heat loss and hypothermia
  • Most species no domesticated- peri-anaesthetic stress
  • Small species- difficult IV access, limited monitoring
  • > morbidity/mortality
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4
Q
  1. What species do not have nostrils?
  2. What is the route of air through nostrils?
A
  1. Cormorants, pelicans, gannets
  2. Nasal cavities to infraorbital sinuzes to cervico-cephalic air sacs- reduces in diameter

Diameter > trachea

Glottis not visible in flamingos

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4
Q
  1. What species do not have nostrils?
  2. What is the route of air through nostrils?
A
  1. Cormorants, pelicans, gannets
  2. Nasal cavities to infraorbital sinuzes to cervico-cephalic air sacs- reduces in diameter

Diameter > trachea

Glottis not visible in flamingos

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5
Q

How are birds ETT related to anatomy?

A
  • Complete/partiall overlapping rings- uncuffed ETT
  • Proximal narrowing of lumen- ETT advancment
  • Long complez shapes (cranes, swans) > dead space
  • Bifurcated in some species
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6
Q

What is the syrinx?
What can cause a complication with ETT in pigeons and ducks?

A

Syrinx- bifurfaction of major bronchi responsible for vocalisation during insp/expiration

Tracheal mucus can cause partial/complete ETT obstruction increased in pigeons and ducks

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7
Q
  1. Which position reduces ventilation of birds and why?
  2. What can compress air sacs?
A
  1. reduced ventilation on dorsal recumbancy (pecoral muscles weight)
  2. Compressed by effusions, enlarged organs or fat deposits

Birds have higher O2 demands- counter-current

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8
Q

What are the different air sacs in birds?

A

Paired
* Cervical
* Cranial thoracic
* Caudal thoracic
* Abdominal

Unpaired: clavicular

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9
Q

Why do birds have a higher tolerance to haemorrhage and anaemia?
Why does renal excretion of drugs need to be considered before use?

A

RBCs have a shorter half life

Renal portal system

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10
Q

How should bird be prepared before anaesthesia?

A
  • Weight taken
  • Complete clinical exam
  • Fasting- not advised <500g, 2-4 hours for larger, comfirm crop empty
  • If possible correct any abnormality prior to anaesthesia: glucose
  • Eye lubricant
  • Mentation- hypoglycaemia, bradycardia, dyspnoea, neuro
  • MMs- pigmented
  • African greys- hypocalcaemia
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11
Q
  1. What are indications for sedation of birds?
  2. What are the important effects?
  3. What is often used?
A
  1. Clinical exam, short procedures, pre-med for anaesthesia
  2. Reduces stress of handling, anaesthetic sparing, amnesic effect
  3. Midazolam 0.5-2mg/kg + butorpahnol 0.5mg/kg IM
    reverse with flumazenil = volume midazolam
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12
Q

Other then midazolam what can also be used for sedation?
What is the dose in budgies, rattites, cranes?

A

Alfaxalone- period of exitability, apnea on induction, variable recovery

Budgies- 10-15mg/kg IM
Birds of prey 10mg/kg IM
Cranes- 7mg/kg IV

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13
Q

When are the following types of drugs used in birds?
Parasympatholytics
Alpha-agonists
Propofol

A

Parasympatholytics- Bradycardia/CPR situations

Alpha-agonists- birds resistant to effecs, combine with ketamine

Propofol- induction for larger species?
causes resp depression, apnoea, hypotension

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14
Q

What can be used for field conditions for injectable anaesthesia?

A

Midazolam 1mg/kg + butorphanol 1mg/kg

Ketamine 3-7mg/kg + metomidine 0.07-0.1mg/kg IM reverse with atipamezole

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15
Q

What NSAIDs and Opioids are used in birds?

What are toxic?

A

NSAIDs:
Meloxicam- varying doses
Carprofen

Opioids:
Butorphanol
Buprenorphine
Fentanyl- bradycardia, hypotension, sedation
Tramadol- post-op

TOXIC:
Flunixin meglumine
Ketoprofen
Diclofenac

16
Q

What can be used for local anaesthesia in birds?

A

Lidocaine and Bupivicaine combination

Line, splash, brachial plexus block

17
Q

What veins are used for venous access in birds?

A

Medial metatarsal vein: poultry, waterfowl

Ulnar (brachial vein): IV needs to be butterflied and sutured in

Intra-osseous catheters: smaller species, proximal tibiotarsus, distal ulna

18
Q

When should a face mask be used?

A
  • Short, non-invasive procedures
  • Induction for ETT placement
19
Q

When would an abdominal air sac tube be placed?

A
  • Oral/tracheal surgery
  • Upper airway obstruction
  • Induction with facemask
  • Cuffed ETS in emergency
20
Q

What are the choices of gas anaesthesia for birds and considerations of both?

A

Isoflurane
* Induces arrythmias, tachycardia and hypertension in some birds of prey
* Decreases ionized calcium
* Induction 3-4%, maintenance 1-2%

Sevoflurane
* Lower blood solubility
* Faster induction/recovery

Non-rebreathing systems- T-piece

21
Q

What is monitored directly during GA in birds?

A

Direct observation
* Palpebral/corneal reflex
* Wing tone
* Feather plucking reflex
* Deep pain/toe pinch reflex
* Sudden feather erection > cardiac arrest
* Clocacal reflex

HR values > or < baseline are abnormal, shifts/trends, oesophageal stethoscope in larger patients
RR chest movements- deep, increased etc

22
Q

How is the patient monitored with equipment during GA?

A
  • Doppler probe on ulnar artery
    Conscious psittacines 90-180mmHg, GA 90-150mmHg
  • Pulse oximeter- larger species
  • Electrocardiogram- attaching leds and keeping a good sign is difficult
  • Temperature- cloacal probes
  • Capnography- accuracy variable
23
Q

What problems can arrise from GA of birds?

A
  • Hypothermia
  • Blood loss/hypovolaemia
  • Brady cardia
  • Hypotension
  • CR arrest- window for resuscitation <30s
  • Post-ETT tracheal stenosis- 1-3 weeks post anaesthesia, too wide ETT
24
Q

How should GA complications in birds be approached?

A
  1. Confirm ETT is patent
  2. Stop anaesthetic gas
  3. Start IPPV
  4. Hypertonic saline 3ml/kg/IV/IO until BP >90mmHg
  5. Crystalloids and colloids
  6. Consider dextrose
  7. Atropine
  8. Start chest compressions
  9. Adrenaline
25
Q

Describe recovery from GA for birds?

A
  1. Stop anaesthetic gas delivery while providing O2
  2. Remove IV/IO catheters
  3. Check glottis for mucus build up after removing ETT
  4. Incubator- careful with handling and orthostatic hypertension, inc risk of hypotension and hypoglycaemia, careful with hyperthermia
26
Q

What is important to note about for anaesthesia of reptiles?

A
  • Poikilothermic- cannot regulate body temp
  • POTZ for each species
  • Typically long recoveries
27
Q

What is notable about reptiles CV system?

A
  • Most species have 3 chambered heart: 2 atria 1 ventricle (muscle ridges prevent mixing)
  • Renal portal system: blood from 1/2 caudal body can reach kidneys
  • Hepatic portal system- blood from 1/2 caudal body passes the liver before general circulation, drugs metabolised by liver admin on forelimbs
28
Q

What happens to reptiles with increased/decreased environmental temp?

A

Increase
* cutaneous vasodilation
* BP reduces and HR increases
* L to R heart shunt
* Warmed blood returns and increases body temp

Reduction
* Cutaneous vasoconstriction
* Blood pooled in muscles keeps body heat
* HR reduces

29
Q

How is apnea and hypoxia different in reptiles?

A

Very tolerant to myocardial hypoxia
Diving reflex in acquatic reptiles
* Bradycardia and direct blood to vital organs
* R to L shunt and bypassing pulmonary circuit
* Reserved with first post-dive breath

30
Q

What respiratory physiology is important for anaesthesia of reptiles?

A

Significant variation
* Anaerobic metabolism
* Some resp exchanges in skin and mucosa of species
* Hypoxia increases RR
* Hypercapnia increases tidal volume
* High O2 environment depresses RR- recovery and CPR
* Temperature- increases demand of O2, increases tidal volume

31
Q

Briefly describe the unique chelonians respiratory anatomy?

A
  • Short trachea with complete rings- short uncuffed ETTs
  • Dorsal sac-like lungs: insp/exp active process
  • No intercostal muscles
  • Ribs fused with shell
  • 2 paired antagonistic abdominal muscles
  • Horizontal septum
  • Gular movements- not RR