Neurogenic Communication Terms Flashcards

1
Q

Anomia

A

word finding difficulties

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2
Q

Phonemic paraphasia

A

word retrieval error with mispronunciation or substitution of phonemes in words. example, someone might say “spot” instead of “pot”

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3
Q

Semantic Paraphasia

A

word is substituted for another word with a similar meaning. example, fork instead of spoon

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4
Q

Neologistic Paraphasia

A

word is substituted for a made up, nonsense word

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5
Q

Perseveration

A

inappropriate repetition of a word or idea previously mentioned

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6
Q

Agrammatism

A
  • grammar deficits, inadequate sentence production
  • use of content words without function words
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7
Q

Alexia

A

acquired reading impairment following brain damage

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8
Q

Agraphia

A

acquired writing impairment following brain damage
- motor dysfunction or spelling impairments

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9
Q

Neologism

A

made up word is created, the word has no meaning to the speaker and is entirely different from intended word

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10
Q

Circumlocution

A

talking around the intended word or idea, not getting to the point

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11
Q

Jargon

A

continuous, fluent utterances that make little sense but appear to make sense to the speaker

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12
Q

Left Hemisphere Damage

A
  • expressive & receptive deficits
  • global deficits
  • cognitive impairment
  • right visual field impairment
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13
Q

Right Hemisphere Damage

A
  • spatial & perceptual deficits
  • discourse & pragmatic deficits
  • impulse behavior & attention difficulty
  • judgement and reasoning problems
  • poor awareness of deficits
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14
Q

Ischemic Stroke

A
  1. occurs due to blockage of blood vessel
  2. Thrombotic: blood clot develops in blood vessels inside brain
  3. Embolic: blood clot develops elsewhere in body and travels to brain through brainstem (ie. heart)
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15
Q

Hemorrhagic Stroke

A
  1. Occurs due to bleeding, blood vessel rupture
  2. Intracerebral: most common, artery bursts; flooding tissues with blood
  3. Subarachnoid : bleeding in areaw between arachnoid matter & pia matter
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16
Q

Ataxia

A
  1. degenerative disease of the nervous system
  2. symptoms mimic being drunk (eg. lack of coordination, slurred speech, falling, eye movement abnormalities)
17
Q

Encephalitis

A

inflammation of the brain

18
Q

Aneurysm

A

abnormal ballooning that forms in the blood vessel

19
Q

Spaced Retrieval Training

A

recall attempts are spaced over increasing lengths of time.

20
Q

Errorless Learning

A

technique for constraining response sets to reduce the number of errors made during learning by persons with dementia

21
Q

Simulated Presence Therapy

A

therapy technique in which a familiar caregiver’s speech or voice is recorded and played back as a cue to reduce agitation and negative behaviors in patients with moderate and greater severity of dementia

22
Q

ALZHEIMER’S DISEASE

A
  1. Abnormal deposits of proteins
    form amyloid plaques and tau
    tangles throughout the brain.
    Characteristics:
    - Wandering and getting lost
    - Repeating questions
    - Problems recognizing friends
    and family
    - Impulsive behavior
    - Cannot communicate
23
Q

FRONTOTEMPOTAL DEMENTIA

A

Abnormal amounts or forms of
tau and TDP-43 proteins
accumulate inside neurons in the
frontal and temporal lobes.
Characteristics:
- Difficulty planning and
organizing
- Impulsive behaviors
- Emotional flatness or excessive
emotions
- Shaky hands
- Problems with balance and
walking
- Difficulty making or
understanding speech

24
Q

LEWY BODY DEMENTIA

A

Abnormal deposits of the
alpha-synuclein protein, called
“Lewy bodies,” affect the
brain’s chemical messengers.
Characteristics:
- Inability to concentrate, pay
attention, or stay alert
- Disorganized or illogical ideas
- Muscle rigidity
- Loss of coordination
- Reduced facial expression
- Excessive daytime sleepiness

25
Q

Vascular Dementia

A

Conditions, such as blood clots,
disrupt blood flow in the brain.
Characteristics:
- Forgetting current or past
events
- Misplacing items
- Trouble following
instructions or learning new
information
- Hallucinations or delusions
- Poor judgment

26
Q

Huntington’s Disease

A
  1. Mutant huntingtin protein, loss of neurons in basal ganglia,
    caudate nucleus, putamen, substantia nigra; possible atrophy in prefrontal, temporal, and parietal lobes; reduced levels of
    inhibitory neurotransmitters, especially GABA
  2. Characteristics:
    – Movement disorders (rigidity, bradykinesia), then personality
    changes
    – Rigidity, bradykinesia, chorea.
    Tic-like movements, gait disturbances, reduced voluntary
    movements; increased fall risk
    – Hyperkinetic dysarthria
27
Q

Parkinson’s Disease

A
  1. Basal ganglia and brain-stem degeneration, presence of Lewy
    bodies in the substantia nigra, frontal lobe atrophy, reduced
    inhibitory dopamine
  2. Bradykinesia, tremors in resting muscles, rigidity, reduced
    eye blinking, festination, disturbed posture, frequent falls,
    impaired initiation
28
Q
A