Myocardial Infarction Flashcards

1
Q

When does acute coronary syndrome occur

A

when the heart tissue endures prolonged ischemia without recovery

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2
Q

How is acute coronary syndrome classified

A

STEMI or nonSTEMI

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3
Q

STEMI heart attack

A

elevated ST, damage goes through the entire cardiac wall

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4
Q

nonSTEMI heart attack

A

depressed ST, damage does not go through the entire cardiac wall (subendocardial)

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5
Q

When does myocardial infarction begin

A

with coronary artery atherosclerosis with causes obstruction then ischemia

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6
Q

What are the ways an obstruction can occur

A

ruptured plaque attracts platelets, which then forms a thrombus

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7
Q

What can result if coronary artery blood supply remains constant but myocardial demands increase

A

severe hypertension, left ventricular hypertrophy

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8
Q

Risk factors

A

atherosclerosis, hyperlipidemia, diabetes, hypertension, smoking, family history

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9
Q

What is the extent of damage influenced by

A

location/level of occlusion in coronary artery, length of time the coronary artery is occluded, and heart’s availability of collateral circulation

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10
Q

Womens s/s

A

SOB, sweating, nausea, vomiting, ingestion, panic/anxiety

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11
Q

Diagnostic tests

A

ECG, blood tests, echocardiogram, coronary angiography

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12
Q

Blood tests

A

CPK-MB fraction, troponin 1

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13
Q

Why are blood tests used

A

when heart cells die, intracellular contents spill into the bloodstream

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14
Q

Troponin 1

A

a cardiac protein highly specific for cardiac muscle necrosis released only when myocardial cell death has occured

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15
Q

When does troponin increase

A

cell death

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16
Q

Echocardiogram

A

identifies what portion of the heart has been affected by the MI, identify abnormal wall motion/contractile dysfunction

17
Q

Primary goal of treatment

A

re-establish blood flow to the heart muscle and minimize damage

18
Q

What can be taken at the start of chest pain

A

antiplatelet agent (aspirin)

19
Q

What does morphine do

A

pain relief, anxiety relief, improve gas exchange

20
Q

Treatment options

A

antiplatelet agent, nitrates, morphine, thrombolytic medicine, beta-blockers, heparin, PTCA/CABG

21
Q

Drug eluting stent

A

a polymer coating over the mesh, that emits a drug over time to help keep the blockage from coming back, stay slippery to prevent cells from getting stuck

22
Q

Complications

A

dysrhythmias, papillary muscle rupture, thromboembolism, ventricular rupture, pericarditis, heart failure, cardiogenic shock

23
Q

What happens if the cardiac muscle loses blood supply and electrical pathways are disrupted

A

the precise electrical pathways become interrupted because infarcted tissues no longer conduct

24
Q

What complications can arise from disruption in electrical pathways

A

fatal dysrhythmias, ventricular tachycardia, ventricular fibrillation

25
Q

What does slowing/blocking of impulses cause

A

some impulses to change direction, disrupt conduction, and the cardiac tissue to fire inappropriately

26
Q

Follow-up

A

cardiac stress testing, cardiac rehabilitation, nutrition consultation, routine cardiologist visits