Locomotion & Posture Flashcards

1
Q

Problems as reported by the patient are known as…

A

Symptoms

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2
Q

What is a sign?

A

observable measurements of the patient’s symptoms (can include absence)

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3
Q

What is a syndrome?

A

Cluster of signs and symptoms

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4
Q

what are the two phases of locomotion?

A

Stance and swing

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5
Q

Where is the central pattern generator located?

A

Lumbo-sacral plexus

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6
Q

where does the Central Pattern Generator receives its input?

A

Muscle spindles and Golgi apparatus

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7
Q

locomotion pattern still remains if patient is decerebrate T/F?

A

T, but there is no tone

patient can speed up and slow down but there is no avoiding of obstacles

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8
Q

Babies have 2 limb locomotion

A

yes, but there is no postural tone

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9
Q

in % how much force does the activation of the first 1/4 of motor units produce?

A

5%

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10
Q

Muscle strength is incremented in small increments T/F

A

T

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11
Q

First motor neurons recruited are for (1) big/small muscle fibres that can generate force (2) aerobically/anaerobically for (3) long/short periods of time

A

1 Smal
2 Anaerobically
3 Long

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12
Q

Define: Fibrilation

A

tiny contractions of the muscle without activation of the synapse (might be due to ACh receptor activation without stimuli)

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13
Q

Fasciculations are…

A

Group of muscle fibres contracting involuntarily (synaptic origin) maybe due to a degenerating axon

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14
Q

what is the result of long term denervation?

A

Atrophy and degeneration (irreversible)

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15
Q

when are muscles completely inactive?

A

REM

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16
Q

What descending pathway is in charge of axial muscles?

A

Ventromedial Pathway

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17
Q

What pathways and what do they sense feed into the ventromedial pathway?

A
Coliculospinal tract (from visual fields)
Vestibulospinal Tract (balance)
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18
Q

What does the lateral corticospinal tract controls?

A

Motor activation of muscles

19
Q

where does the lateral corticospinal tract decussate?

A

decussates at the pyramidal decussation in the lower medulla

20
Q

Last type of fibres activated when contracting a muscle?

A

Thick, fast fibres, work aerobically but tire quickly

21
Q

Hemiparetic Gait is described as…

A

Weakness (increased muscle tone) on CONTRALATERAL side [whole body] due to a brain lesion of the motor cortex

22
Q

Circumducting Gate is…

A

Weakness of the lower extremity without involvement of the upper limb (limb is in full extension)

23
Q

What is worst, decerebrate or decorticate?

A

Decerebrate indicates involvement of the brainstem. Lesion has affected the midbrain at least

24
Q

how are the limbs in the decerebrate position?

A

Both extended

25
how are the limbs in the decorticate position?
Flexion of the upper limb | Extension of the lower limb
26
Cortext sends mainly inhibitory signals T/F
F, but in the case of motor control it is true
27
The reticular formation is part of the --------- pathway. And this pathway decussates at the level of ------
Ventromedial Pathway | Does not decussate also called 'Direct Pyramidal Pathway'
28
Where is the reticular formation found?
Very diffuse in the pons and medulla
29
the reticular formation's output is mainly... and to the ------
inhibitory | lower limb flexors
30
An important part of the lateral pathway is...
The Red Nucleus
31
the Red Nucleus is found in the...(1) and controls mainly the (2)
(1) Midbrain | (2) Upper Limb
32
The red nucleus' fibres reaches to and beyond the lumbar area T/F
F, only reaches the cervical part of the spinal cord
33
Via which tract does the Red Nucleus Projects its fibres?
Rubrospinal Tract
34
Output of the Red Nucleus is mainly (1) and to the (2) of the upper limb
1 Excitatory | 2 Flexor Muscles
35
Because there is involvement of the midbrain in the ----(1)----- posture, the patient will have the the lower limbs extended and the upper limbs ----(2)---
1 Decerebrate | 2 Extended
36
LMN disease shows
relaxation of muscles, weakness, decreased reflexes,
37
UMN Disease shows
Increased tone, weakness due to increased tone, increased reflexes
38
The forehead is supplied by (1) nerve and its got ipsilateral/contralateral supply (2)
1 Trigeminal 2 Both this is way it can tell us whether it is an UMN disease or a LMN disease
39
the forehead will NOT move in which type of MN disease
Lower
40
What two parts constitute each movement?
Anticipation and Control of Muscle Position
41
what is ANTICIPATION? what is its feedback mechanism?
activity before movement of the limb has been initiated based on what is believed is going to happen FeedForward thus not perfect
42
Anticipation is controlled by which tract?
Ventral Corticospinal Tract
43
Control of Muscle Position is
the accomodation of the limb to achieve the desired outcome by feedbackwards loop