L2: AGN

Question 1

Def of Nephritic Syndrome

Question 2

Etiology of Nephritic Syndrome

Question 3

Etiology of Nephritic Syndrome

• Causes of hypo-complomentic Glumeronephritis

Question 4

Etiology of Nephritic Syndrome

• Causes of normo-complementic glumeronephritis

Question 5

Alport Syndrome

Question 6

Goodpasture Syndrome

Question 7

The decrease in complement is D2 consumption in formation of Ag-Ab complex.

Answer 7

…

Question 8

Incidence of Acute Post-Infectious Glumeronephritis

Question 9

Age in Acute Post-Infectious Glumeronephritis

Question 9

Sex in Acute Post-Infectious Glumeronephritis

Question 10

Etiology of Acute Post-Infectious Glumeronephritis

Question 11

Etiology of Acute Post-Infectious Glumeronephritis

• Strept pharyngitis

Question 12

Etiology of Acute Post-Infectious Glumeronephritis

• Strept pyoderma

Question 13

Other Organisms Causing Acute Post-Infectious Glumeronephritis

Question 14

Chronic Post-Infectious Glumeronephritis

Question 15

• The reasons may include the easier access to the treatment of streptococcal infections and the widespread presence of fluoride in water, which decreases virulence factors of Streptococcus pyogenes.

Question 16

• Acute poststreptococcal glomerulonephritis is used synonymously with AGN because AGN follows upper respiratory or skin infection with group A -hemolytic streptococcus in over 90% of cases

Question 17

Pathogenesis of APSGN

Question 18

Pathology of APSGN

Question 19

Pathology of APSGN

• Light Microscopy

Question 20

Pathology of APSGN

• Immuno-Fluoresence Microscopy

Question 21

Pathology of APSGN

• EM

Question 22

CP of APSGN

Question 23

CP of APSGN

• Subclinical Course

Question 24

CP of APSGN

• Nephritic Syndrome

Question 25

CP of APSGN

• Hematuria

Question 26

CP of APSGN

• Proteinurea

Question 27

CP of APSGN

• Edema

Question 28

CP of APSGN

• Oliguria

Question 29

CP of APSGN

• HTN

Question 30

Complications of APSGN

Question 31

Complications of APSGN

• ARF

Question 32

Complications of APSGN

• Rapidly Progressive Azotemia

Question 32

Complications of APSGN

• Hyperkalemia

Question 33

Complications of APSGN

• Circulatory Congestion

Question 34

Complications of APSGN

• Hypertensive Encephalopathy

Question 34

Incidence of Hypertensive Encephalopathy in ASPGN

Question 35

CP of Hypertensive Encephalopathy in ASPGN

Question 36

Cause of Hypertensive Encephalopathy in ASPGN

Question 37

MRI of Hypertensive Encephalopathy in ASPGN

Question 38

Neurological complication in APSGN cannot be attributed exclusively to hypertensive encephalopathy or abnormal serum biochemistry.

Question 39

With advances in neuroimaging techniques, there is clear evidence that some children may develop cerebral vasculitis.

Question 40

INVx in ASPGN

Question 41

INVx in ASPGN

• Urine Analysis

Question 42

INVx in ASPGN

• Blood Tests

Question 43

INVx in ASPGN

• Evidence of recent streptococcal infection

Question 44

INVx in ASPGN

• Renal Imaging

Question 45

INVx in ASPGN

• Renal Bx

Question 46

Atypical Presentation of APSGN

Question 47

Atypical resolution in APSGN

Question 48

Typical Presentation of APSGN

Question 49

DDx of APSGN

Question 50

TTT of APSGN

Question 51

TTT of APSGN

• Diet

Question 52

TTT of APSGN

• Bed Rest

Question 53

TTT of APSGN

• Edema

Question 54

TTT of APSGN

• HTN

Question 55

TTT of APSGN

• Hyperkalemia

Question 56

TTT of APSGN

• Antibiotic

Question 57

TTT of APSGN

• Immediate Dialysis

Question 58

Prognosis of APSGN