EIM 9 Cardio Flashcards
Tricuspid Valve Regurg
usually physiologic, silent on auscultation in clinically normal horses
detectable by Doppler studies in many clinically normal horses.
What murmur grade is less likely to be clinically relevant?
2 or less
If a diastolic mitral valve fluttering is identified, what other pathology needs to be ruled out?
Aortic valve regurgitation
What is the most commonly recognized congenital heart defect in foals?
Perimembranous ventricular septal defect
When pulmonary-to-systemic flow ratio (Qp:Qs) of a shunt exceeds _____ the shunt is considered clinically relevant, resulting in obvious volume overload of the left atrium and left ventricle
1.8:1 (>1:1 implies a left to right shunt)
What is the most consistent physical examination finding associated with a ventricular septal defect?
Harsh holosystolic/pansystolic murmur loudest just below the tricuspid valve region–most defects communicate near the tricuspid valve
pentalogy of Fallot
Large, unrestrictive ventricular septal defect
Overriding and malalignment of the aortic root
Right ventricular outflow tract obstruction
Right ventricular hypertrophy
Patent ductus arteriosus
Which echographic finding is most indicative of hemodynamically important mitral regurgitation?
Circular, turgid appearance to the left atrium–often enlarged as well; indicative of volume overload and decreased function
Define the relationship between cardiac output in the left and right ventricles
COleft = COright
Because they are arranged in series, cardiac output on each side must be equivalent. Therefore compensatory changes can be seen in the opposite side of the heart in unilateral ventricular failure/dysfunction.
Incompetent valves
result in eccentric ventricular hypertrophy
Double apex sign
is an indicator of marked right ventricular enlargement
Which type of adrenergic receptors predominate in the equine heart?
Beta 1
Atrial contraction is responsible for ___% of ventricular filling at rest
15-20
direct determinants of ventricular stroke volume
Preload (ventricular end-diastolic volume)
Contractility
Afterload (wall tension required to eject blood)
Cardiac lesions increasing workload
Frank-Starling mechanism
The ability of the heart to change its force of contraction in response to changes in preload
This changes stroke volume, allowing the heart to respond to systemic changes