EIM 9 Cardio Flashcards

1
Q

Tricuspid Valve Regurg

A

usually physiologic, silent on auscultation in clinically normal horses​

detectable by Doppler studies in many clinically normal horses.

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2
Q

What murmur grade is less likely to be clinically relevant?

A

2 or less

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3
Q

If a diastolic mitral valve fluttering is identified, what other pathology needs to be ruled out? ​

A

Aortic valve regurgitation

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4
Q

What is the most commonly recognized congenital heart defect in foals?

A

Perimembranous ventricular septal defect

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5
Q

When pulmonary-to-systemic flow ratio (Qp:Qs) of a shunt exceeds _____ the shunt is considered clinically relevant, resulting in obvious volume overload of the left atrium and left ventricle

A

1.8:1 (>1:1 implies a left to right shunt)​

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6
Q

What is the most consistent physical examination finding associated with a ventricular septal defect?​

A

Harsh holosystolic/pansystolic murmur loudest just below the tricuspid valve region–most defects communicate near the tricuspid valve​

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7
Q

pentalogy of Fallot

A

Large, unrestrictive ventricular septal defect​
Overriding and malalignment of the aortic root​
Right ventricular outflow tract obstruction​
Right ventricular hypertrophy​
Patent ductus arteriosus

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8
Q

Which echographic finding is most indicative of hemodynamically important mitral regurgitation?​

A

Circular, turgid appearance to the left atrium–often enlarged as well; indicative of volume overload and decreased function

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9
Q

Define the relationship between cardiac output in the left and right ventricles

A

COleft = COright

Because they are arranged in series, cardiac output on each side must be equivalent. Therefore compensatory changes can be seen in the opposite side of the heart in unilateral ventricular failure/dysfunction.

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10
Q

Incompetent valves

A

result in eccentric ventricular hypertrophy​

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11
Q

Double apex sign

A

is an indicator of marked right ventricular enlargement

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12
Q

Which type of adrenergic receptors predominate in the equine heart?​

A

Beta 1

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13
Q

Atrial contraction is responsible for ___% of ventricular filling at rest​

A

15-20

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14
Q

direct determinants of ventricular stroke volume

A

Preload (ventricular end-diastolic volume)​
Contractility​
Afterload (wall tension required to eject blood)​
Cardiac lesions increasing workload

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15
Q

Frank-Starling mechanism

A

The ability of the heart to change its force of contraction in response to changes in preload

This changes stroke volume, allowing the heart to respond to systemic changes

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16
Q

Systolic clicks

A

Over great vessels - benign

Over the mitral valve -thought to be a marker for MV disease or prolapse of the valve into the left atrium​

17
Q

Identify the two most common arrhythmias in the horse

A

2nd-Degree AV Block​

Sinus arrhythmia

18
Q

systolic ejection murmur

A

It is the most common functional murmur​

It must occur between S1 and S2​

It can be up to grade 4-5/6, with significant daily variation​

It is best heard over the aortic and pulmonic valves, and may project into their arteries at the left cardiac base.​

19
Q

How soon after exercise must a stress echocardiogram be performed to identify global or regional myocardial dysfunction?​

A

Up to 2-3 minutes, HR > 100 BPM

20
Q

Treatment of AF is recommended for all horses that are still in use for any type of athletic activity when the average maximal HR during sustained maximal exercise exceeds how many beats/min

A

220

21
Q

Electrical cardioversion (TVEC) involves a timed shock delivery on which ECG wave? ​

A

R

22
Q

Ventricular origin impulses​

A

Are conducted abnormally and more slowly, resulting in a widened QRS, an abnormal QRS orientation, and abnormal T waves. ​

23
Q

Junctional impulses​

A

Are more likely to result in a narrow, relatively normal-appearing QRS complex with normal initial activation and electrical axis, because they originate above the ventricular myocardium. ​

24
Q

features of complex or potentially “malignant” ventricular arrhythmias: ​

A

-severe hemodynamic compromise (systemic hypotension, poor pulse quality, -prolonged capillary refill time, weakness, collapse)​
-sustained VT​
-rapid ventricular rate (greater than 120 beats/min)​
-short coupling intervals (particularly with R-on-T complexes)​
-multiform or polymorphic QRS morphology (including torsades de pointes)​

25
Q

hyperkalemia

A

Broadening and flattening of the P wave are the most consistently observed change. Prolongation of the PQ interval and bradycardia develop, excitability decreases, and atrial standstill (sinoventricular rhythm) characterized by complete absence of P waves may be observed. ​
Either inversion or enlargement (tenting) of the T waves is also likely. ​
Marked widening of the QRS complex may be noted as near-lethal concentrations of potassium are approached ​