Cadiovascular System Flashcards

1
Q

Impact of physical activity on heart

A

Prevents clots
Prevent blockages
Healthy 02 supply
Prevents build of plaque (athersma)

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2
Q

Impact of physical activity on high blood pressure

A

Aerobic exercise can :

Lower systolic pressure
Lower dystolic pressure

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3
Q

Impact of physical activity (HDL)

A

Good cholesterol- positive anti- oxidant

Protect blood cells

Protects artery walls

Remove LDL

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4
Q

Impact of PA on (LDL)

A

Decreases cholesterol (LDL)

Reduces inflammation of artery walls (lower calcification)

Reduced chance of heart attacks

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5
Q

Impact of PA (stroke)

A

Strokes caused by low 02 delivery to brain

Lower blood pressure - lower chance of vessel being burst - leading to strokes

Healthy weight

Maintains strong vessels - lower tendency to burst and 02 delivery to be sustained

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6
Q

Cardiac output

A

Volume of blood leaving left ventricle per minute

SRxHR

Trained = steeper cardiac gradient

Untrained = shallower cardiac gradient

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7
Q

Why cardiac output is different in trained vs untrained

A

Trained - hypertrophy

, higher diastolic volume (more blood in the heart at resting)

lower resting heart rate =more flexibility

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8
Q

Heart rate responses

A

Resting HR

Anticipatory rise - release of adrenaline - stimulates SA node - CCS

Rapid increase- heart trying to increase 02 to working muscles

Steady state exercise - D=S

Steep decline - EPOC

Levelling off occurs - EPOC

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9
Q

Medulla oblongata

A

Cardiac control centre and is linked to the SA node to control HR

Through two systems

Sympathetic - increase in HR

Parasympathetic- lowers HR

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10
Q

Heart rate control

A

Neural)Chemoreceptor - more c02 increasing acidity meaning medula will increase HR

Baroreceptors - BP increases - sympathetic and VICE VERSA

Proprioceptors - more movement - increase HR

Adrenaline (hormonal) - release of adrenaline - onto SA node increasing HR

Intrinsic - heart can sense changes itself - temperature and contractivility - more venous return increasing HR

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11
Q

Heart values

A

Stroke volume - amount of blood leaving the heart

Venous return - amount of blood returning to the heart

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12
Q

Venous return mechanisms

A

Skeletal muscle pump- blood squeezed up via muscle contractions

Pocket valve- prevent back flow of blood in muscles

Smooth muscle - constrict and pump blood back to heart

Respiratory pump - thoracic cavity pumps outwards when breathing in , pressure changes squeezes blood back to the heart

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13
Q

Blood pressure

A

Force exerted on the walls of arteries

Factors affecting this

LDL- artheroma- higher blood pressure

Initial exercise

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14
Q

Gase exchange

A

External - lungs - standard diffusion

Internal - muscles - high PP of 02 in capillary diffuses into muscle

and c02 in muscle HIGH PP
Diffuses into capillary to be carried back into the lungs for the external process

VICE VERSA DURING RECOVERY/rest

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15
Q

Oxygen disassociation (Bohr shift)

A

The dissociation of 02 from HB into tissue

Stimulated through exercise

Causing

Lower PH
Presence of co2
La
Temprature

This then shifts the curve to the right

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16
Q

CV drift

A

An increase in HR despite same intensity as stroke volume and HR drift apart

Fluid loss through sweating will increase viscosity lowering SR and HR will increase to compensate

Refuelling
Rehydrate
Temperature

17
Q

Av02 difference

A

Arterial - venial

Difference between arterial and venial blood

Greater av02 diff
- greater performance due to more arterial blood
- greater 02 extraction
-02 used for energy
-

How to improve av02
- training - increase in aerobic capacity
- increase temperature- Bohr shift
- effective warm up

18
Q

Starlings law

A

Increase in venous return will increase stroke volume as heart stretches and pumps more per beat

19
Q

Heart issues

A

Heart attack - caused by blockages

Heart failure - doesn’t pump blood as well

20
Q

Fats to athletes

A

Used at low intensities

Too much can cause weight gain

21
Q

Carbs to athletes

A

For moderate to high intensity exercises