Abomasal Diseases Flashcards

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1
Q

What is Left Abomasal Displacement (LDA)? Causes? who is affected?

A
  • Displacement of the abomasum to the left between the rumen and body wall
  • Cause: Increased production of gas and reduced motility
  • Occurrence:
    • Dairy primarily
    • Multiparous cows
    • Calves up to several months of age not uncommon
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2
Q

What are the risk factors for LDA?

A
  • Breed (dairy)
  • Parity (metabolic differences)
  • Peri-parturient
    • Calving (abdominal space)
    • Diseases (metritis/mastitis/metabolic
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3
Q

What is the clinical presentation of an LDA?

A
  • Drop in Milk Production
    • sudden or Intermittent
  • Anorexia or “sorting” for fiber in feed
  • Ketosis
  • Often in conjunction with or occurring after treatment of other peri-parturient diseases
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4
Q

How is LDA diagnosed?

A
  • Left abdomen:
    • Auscultate over 9-12th IC spaces
    • Simultaneous percussion and auscultation
    • “Map” ping
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5
Q

What is a RDA? RAV?

A
  • Right Displaced Abomasum
    • Similar presentation
  • Right Abomasal Volvulus
    • more severe clinical depression and instability
  • Same risk factors
  • Right sided pings are a priority call
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6
Q

How does RDA occur?

A
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7
Q

How does RAV occur

A
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8
Q

What clinical presentation of RDA/RAV

A
  • Less frequent than LDA
  • Ping on right side
  • HyPOchloremia
  • HyPOkalemia
  • Metabolic alkalosis
  • Colic
  • Dehydration/Shock
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9
Q

What is the Treatment for LDA?

A
  • Replace and fix
  • Treat concurrent problems
  • Usually outpatient or on farm Tx
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10
Q

What is the Treatment for RDA/RAV

A
  • Replace and Fix
  • Treat concurrent problems and status
  • Often Critical patient with RAV
    • vagal indigestion
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11
Q

What is the medical support for LDA/RDA/RAV?

A
  • Correct Electrolyte Deficiencies
    • Potassium, Chloride, Phosphorus, Calcium
  • Alkalemia
  • Hypovolemia
  • Anti-inflammatory Support - NSAIDs
  • Antimicrobials
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12
Q

What causes abomasal ulcers?

A
  • Stress associated
    • Show
    • Weaning
    • Weather/Feed change
  • High Starch diets
  • Systemic disease
    • post partum
    • Pneumonia
    • Lymphosarcoma
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13
Q

What are the signs of abomasal ulcers?

A
  • Can be non-perforating or perforating
    • Type 1 = mucosal
    • Type 4 = perforation, hemorrhage
  • Anorexia
  • +/- Pain - Bruxism
  • Decreased ruminal contractions
  • Mild to severe colic
    • focal pain over abomasum
  • Normal hematocrit to severe anemia
  • Decreased PCV, with normal to decreased protein
  • Normal feces/diarrhea/melena
  • Afebrile to febrile (<104)
    • +/- peritonitis
  • Pallor, decreased milk production, shock, sepsis
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14
Q

What signs are associated with perforated abomasal ulcers?

A
  • Leukocytosis/leukopenia
  • hyperfibrinogenemia
  • fever
  • scleral injection
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15
Q

How are abomasal ulcers diagnosed?

A
  • Clinical signs and signalment
  • Abdominal ultrasound
  • Abdominocentesis
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16
Q

What is the treatment for abomasal ulcers?

A
  • Removal of grain from diet
  • Reduce stresses
  • Correct concurrent diseases
  • High quality hay, stall rest
  • Oral alkalinizing agents
    • pepto? kaopectate?
  • Blood transfusions
  • Antibiotics for sepsis with perforation
    *
17
Q

Who suffers from clostridial abomasitis?

A
  • Calves
  • Feeding risk:
    • heavy milking cow
    • tube feeding
      *
18
Q

What are the signs of Clostridial Abomasitis?

A
  • Acute - May be found dead
  • Abdominal distention
  • +/- diarrhea
  • Succussion and Ping in right abdomen
  • Emphysematous and necrotizing abomastitis with marked edema
  • Gm+ bacilli found in mucosa and submucosa
19
Q

How is Clostridial Abomasitis treated/prevented?

A
  • Treat:
    • Antibiotics (Penicillin, Oxytet)
    • Anti-inflammatories
    • Fluids
  • Prevent:
    • Milking and milk feeding strategies
    • ID at risk cows
20
Q

What is Abomasal impaction?

A
  • Sequelae to poor nutrition
    • Sand eaters
    • Foreign objects
    • Straw/bedding chips (low fiber diets)
    • Trichobezors
21
Q

What are the clinical signs of abomasal impaction?

A
  • Decreased feed intake
  • Dehydration
  • Dry, decreasing feces
  • Pear/Papple shaped abdomen
  • Weak rumen contractions
  • Fluid rumen on succussion
  • Distended rumen on palpation
22
Q

How is Abomasal impaction diagnosed?

A
  • Clinical signs
  • Ultrasound
  • Variable blood work:
    • chloride/Acid-base status
23
Q

How is abomasal impaction treated

A
  • Erythromycin 8,8mg/kg
    • abomasal contractions
  • Cathartic and laxatives
    • Oil
    • Magnalax
    • Dioctyl Sodium Succinate (DSS)
  • Surgery
24
Q

What is the prognosis for Abomasal impaction?

A
  • Poor prognosis
  • Sx poor due to limited exposure and rupture of abomasum
  • Slaughter salvage often chosen