Abomasal Diseases Flashcards
1
Q
What is Left Abomasal Displacement (LDA)? Causes? who is affected?
A
- Displacement of the abomasum to the left between the rumen and body wall
- Cause: Increased production of gas and reduced motility
- Occurrence:
- Dairy primarily
- Multiparous cows
- Calves up to several months of age not uncommon
2
Q
What are the risk factors for LDA?
A
- Breed (dairy)
- Parity (metabolic differences)
- Peri-parturient
- Calving (abdominal space)
- Diseases (metritis/mastitis/metabolic
3
Q
What is the clinical presentation of an LDA?
A
- Drop in Milk Production
- sudden or Intermittent
- Anorexia or “sorting” for fiber in feed
- Ketosis
- Often in conjunction with or occurring after treatment of other peri-parturient diseases
4
Q
How is LDA diagnosed?
A
- Left abdomen:
- Auscultate over 9-12th IC spaces
- Simultaneous percussion and auscultation
- “Map” ping
5
Q
What is a RDA? RAV?
A
- Right Displaced Abomasum
- Similar presentation
- Right Abomasal Volvulus
- more severe clinical depression and instability
- Same risk factors
- Right sided pings are a priority call
6
Q
How does RDA occur?
A
7
Q
How does RAV occur
A
8
Q
What clinical presentation of RDA/RAV
A
- Less frequent than LDA
- Ping on right side
- HyPOchloremia
- HyPOkalemia
- Metabolic alkalosis
- Colic
- Dehydration/Shock
9
Q
What is the Treatment for LDA?
A
- Replace and fix
- Treat concurrent problems
- Usually outpatient or on farm Tx
10
Q
What is the Treatment for RDA/RAV
A
- Replace and Fix
- Treat concurrent problems and status
- Often Critical patient with RAV
- vagal indigestion
11
Q
What is the medical support for LDA/RDA/RAV?
A
- Correct Electrolyte Deficiencies
- Potassium, Chloride, Phosphorus, Calcium
- Alkalemia
- Hypovolemia
- Anti-inflammatory Support - NSAIDs
- Antimicrobials
12
Q
What causes abomasal ulcers?
A
- Stress associated
- Show
- Weaning
- Weather/Feed change
- High Starch diets
- Systemic disease
- post partum
- Pneumonia
- Lymphosarcoma
13
Q
What are the signs of abomasal ulcers?
A
- Can be non-perforating or perforating
- Type 1 = mucosal
- Type 4 = perforation, hemorrhage
- Anorexia
- +/- Pain - Bruxism
- Decreased ruminal contractions
- Mild to severe colic
- focal pain over abomasum
- Normal hematocrit to severe anemia
- Decreased PCV, with normal to decreased protein
- Normal feces/diarrhea/melena
- Afebrile to febrile (<104)
- +/- peritonitis
- Pallor, decreased milk production, shock, sepsis
14
Q
What signs are associated with perforated abomasal ulcers?
A
- Leukocytosis/leukopenia
- hyperfibrinogenemia
- fever
- scleral injection
15
Q
How are abomasal ulcers diagnosed?
A
- Clinical signs and signalment
- Abdominal ultrasound
- Abdominocentesis