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CPR Exam 1 > 12. CPR Automonics > Flashcards

12. CPR Automonics Flashcards

1
Q

SA node parasym innervation:

Via:

Receptor:

Action:

Effect:

A

Via: Vagus N

Receptor: Muscarinic

Action: decrease rate of depol

Effect: slow HR

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2
Q

SA node sym innervation

Via:
Receptor:
Action:
Effect:

A

Via: fibers from T1-T5
Receptor: Beta-1
Action: increase rate of depol
Effect: increase HR

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3
Q

AV node parasym innervation

Via:
Receptor:
Action:
Effect:

A

Via: Vagus N
Receptor: Muscarinic
Action: decrease slope of pacemaker potential & depol rate during phase 0
Effect: decrease conduction velocity thru AV node/ventricles

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4
Q

AV node sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: T1-T5
Receptor: Beta-1
Action: increase rate of depol during phase 0 & pacemaker potential
Effect: incresae conduction velocity thru AV node

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5
Q

Atrial myocyte parasym innervation

Source:
Receptor:
Action:
Effect:

A

Source: vagus N
Receptor: muscarinic
Action: decrease Ca into myocyte
Effect: decrease atrial contractilty & maybe ventricular contractility too

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6
Q

Atrial & ventricular myocyte sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: T1-T5
Receptor: Beta 1
Action: increase Ca into myocytes
Effect: increase myocyte contractility (cardiac M pumps harder)

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7
Q

Cerebral vasculature sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: local
Receptor: Alpha-1
Action: contraction of vascular smooth M
Effect: vasoconstriction

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8
Q

when is cerebral vasculature stimulated by the sym system

A

systemic BP = high enough to damage cerebral capillaries

-constrictionof the small As decrease blood flow thru caps to protect them

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9
Q

Coronary A parasym innervation

Source:
Receptor:
Action:
Effect:

A

Source: vagus N
Receptor: muscarinic
Action: relax smooth M
Effect: vasodilate and increase coronary blood flow

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10
Q

Coronary A symp innervation

Source:
Receptor:
Action:
Effect:

A

Source: T1-T5
Receptor: alpha-1
Action: contract smooth M
Effect: vasoconstrict to decrease blood flow (short lived)

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11
Q

why is vasoconstriction of coronary A short lived

A

bc this will increase metabolic need

and lead to vasodilation that overcomes the sym effect

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12
Q

cutaneous vasculature sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: multiple
Receptor: alpha 1
Action: contract vascular smooth M
Effect: vasoconstrict

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13
Q

how are sweat glands innervated

A

sympathetic muscarinic

relax vascular smooth m

–> vasodilate to increase primary sweat production

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14
Q

skeletal M vasculature sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: multiple
Receptor: alpha 1 & beta 2
Action: alpha 1- contract vascular smooth M & beta-2 relax it
Effect: alpha 1: vasoconstrict

beta 2: vasodilate

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15
Q

splanchnic vessels sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: T5-L2
Receptor: alpha 1
Action: contract vascular smooth m
Effect: vasoconstriction of splanchnic BV

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16
Q

which ganglion are involved in the splanchic vessel sym innervation

A

celiac ganglion

superior mesenteric ganglion

inferior mesenteric ganglion

17
Q

when do splanchnic vessels vasodilate

A

when sym activity is decreased

18
Q

sym innervation of Vs

Source:
Receptor:
Action:
Effect:

A

Receptor: alpha 1, 2 & beta 2
Action:

  1. alpha 1: contract venous smooth m
  2. alpha 2 & beta 2: relax it

Effect:

  1. alpha 1: contract V –> increase blood return to heart
  2. alpha 2/beta: dilate –> slow return to heart
19
Q

pulmonary vasculature parasym innervation

Source:
Receptor:
Action:
Effect:

A

Source: vagus
Receptor: muscarinic
Action: direct = produce NO and release from endothelial cells & indirect = use NO & relax vascular smooth M
Effect: vasodilation

20
Q

where is pul vasculature innervated more

A

most: extrapul As (pul A and pul trunk)

decrease as you move closer to alveoli

21
Q

pul vasculature sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: first 5 thoracic ganglia
Receptor: primarily alpha 1 but also have beta 1 & beta 2
Action: alpha 1: contract vascular smooth M & beta 1/2: relax it
Effect: alpha 1- vasocontrict

beta 1/2 vasodilate

22
Q

how else can ANS activity be activated for pul vasculature

A

mechanoreceptors - detect distention

& exposure to cold air or hypoxia

23
Q

nasal mucosa parasym innervation

Source:
Receptor:
Action:
Effect:

A

Source: trigemial N and sphenopalatine ganglion
Receptor: muscarinic
Action: vasodilate
Effect: increase mucus secretion

24
Q

nasal mucosa sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: superior cervial ganglia via CN VII
Receptor: alpha 1
Action: indirectly contract smooth M
Effect: reduce mucus production

25
Q

lower airway, extrinisic parasym innervation

Source:
Receptor:
Action:
Effect:

A

Source: vagus N
Receptor: muscarinic
Action/Effect: bronchoconstriction & increased mucus synthesis

26
Q

lower airways (bronchial), extrinsic sym innervation

Source:
Receptor:
Action:
Effect:

A

Source: T1-T5
Receptor: beta 2
Action: relax bronchial smooth M
Effect: bronchodilation & slightly increase secretions

27
Q

what are the vascular components of the bowman’s capsule

A

afferent arterioles: blood flow to glomerular capillaries

glomerular capillaries: deliver blood/plasma so plasma can be filtered

efferent arterioles: blood flow out of glomerular capillaries

28
Q

what is the vascular component of the proximal convoluted tubule

A

peritubular capillaries: secrete substance into proximal convoluted tubules to be excreted in urine & reabsorb things that were filtered out but are actually needed

( = efferent arterioles are broken down into these)

29
Q

what is the vasa recta

A

blood supply of the loop of henle

= secrete Na, Cl & K & reabsorb h2o to keep interstitium concentrated

30
Q

what is the blood supply os the distal convoluted tubule/collecting duct

A

perituular capillaries

= reabsorb Na & h2o

hormonally controlled by ADH & Aldosterone

31
Q

kidney vasculature sym innervation
Receptor:
Action:
Effect:

A

Receptor: alpha adrenergics
Action: contract vascular smooth M
Effect: vasoconstrict - renal A, afferent & efferent arterioles

==> decrease blood flow to kidney–> decrease urine production

32
Q

justaglomerular apparatus sym innervation

Receptor:
Action:
Effect:

A

=granule cells of afferent arterioles

Receptor: Beta-1
Action: increase release of renin
Effect:

  1. increase angiotensin & angiotensin II production
  2. angiotensin cuases increased releast of aldosterone
  3. aldosterone increases Na reabsorption & release ADH
  4. ADH increase h2o resorptio ==> INCREASE BLOOD VOLUME

CPR Exam 1 (27 decks)

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