Yersiniosis + Necrobacillosis Flashcards
Yersinia enterocolitica does not infect humans
F
Lesions caused by Yersinia pseudotuberculosis are always limited to the gut
F
Yersinia pseudotuberculosis can cause a generalised disease in hares
T
Rodents and hares are susceptible to Yersinia pseudotuberculosis
T
Yersinia pseudotuberculosis can cause abortion in cattle
T
Focal inflammation and necrosis are postmortem lesions of the disease caused by Yersinia pseudotuberculosis
T
Only rodents are susceptible to Yersinia pseudotuberculosis
F
Some Yersinia enterocolitica strains have cross reaction with brucellae
T
Yersinia pestis is endemic in certain countries of Europe
F
Yersinia enterocolitica can infect pigs
T
Starvation and long, cold winter can predispose wild living animals to disease caused by Yersinia pseudotuberculosis
T
Yersinia pseudotuberculosis mainly causes respiratory clinical signs in cattle
F
yersinia pestis has been eradicated from earth
F
yersinia enterocolitica generally infects hosts per os
T
fruits and vegetables can be the source of human infection by yersinia
T
the agent of the plague is transmitted by fleas
T
yersinia pseudotuberculosis is a zoonotic agent
T
diseases caused by yersinia pseudotuberculosis occur only in tropical areas
F
Diarrhoea is a common clinical sign of yersiniosis in pig
T
Yersinia pseudotuberculosis can cause diarrhoea
T
Carrier animals shed the agent of yersiniosis in the faeces
T
Plague is caused by Yersinia pestis
T
Only rats are susceptible to plague
F
Fleas can transmit plague
T
Yersinia pseudotuberculosis can infect brown hares
T
Yersinia pseudotuberculosis can cause septicaemia is rodents
T
Farm animals are regularly vaccinated to prevent infection caused by Yersinia pseudotuberculosis
F
Yersinia pseudotuberculosis can infect humans
T
Plague is caused by Yersinia pseudotuberculosis
F
Arthropods are involved in spreading of plague
T
Yersinia pseudotuberculosis has a wide host range
T
Yersinia pseudotuberculosis is not a zoonotic agent
F
Yersinia enterocolitica can cause diarrhoea only in rodents
F
Yersinia enterocolitica can cause septicaemia
T
Yersinia enterocolitica can cause lesions in the mesenteric lymph nodes
T
Yersinia enterocolitica can infect humans.
T
Plague occurs in the United States
T
Only humans are susceptible to the agent of plague
F
Yersinia pseudotuberculosis causes frequently clinical signs in rodents
T
Yersinia pseudotuberculosis can cause enteritis
T
Yersinia enterocolitica can infect only swine
F
Yersinia enterocolitica can infect animals per os.
T
Yersinia pseudotuberculosis can infect rodents
T
Yersinia pseudotuberculosis can colonize the gut
T
Yersinia pseudotuberculosis cause tuberculosis in wild living animals
F
Yersinia pseudotuberculosis can infect only rodents
F
Animals can be infected with Yersinia pseudotuberculosis mainly per os
T
Starvation of wild living animals is a predisposing factor of yersiniosis
T
Infection with Yersinia pseudotuberculosis is limited to the gut
F
Yersinia pseudotuberculosis can cause septicaemia in rodents and hares
T
Yersinia pseudotuberculosis can cause focal inflammation and necrosis in the parenchymal organs
T
Yersinia pseudotuberculosis can survive in the environment
T
Yersinia pseudotuberculosis can cause disease only in rodents
F
Yersinia pseudotuberculosis usually appears in tropical regions
F
Yersinia pseudotuberculosis generalizes in brown hares
T
The agent of yersiniosis cannot be cultured, PCR is the only way of its detection
F
Yersiniosis of rodents can be caused by Yersinia pseudotuberculosis
T
Carrier animals shed the agent of yersiniosis in the faces
T
Yersinia pseudotuberculosis can cause severe pleuropneumonia in rodents
F
Yersinia pseudotuberculosis can cause disease in hares
T
Infection caused by Yersinia pseudotuberculosis is always limited to the gut
F
Primary replication sites of Y. enterocolitica are the tonsils and lymphoid tissue of gut
T
Y. enterocolitica has a narrow host range.
F
Y. enterocolitica can be treated with tetracyclines
T
Swine is an important host of Y. enterocolitica
T
Chronic lesions caused by Y. enterocolitica resemble the lesions of tuberculosis
F
Hare and chinchilla are most susceptible to Y. enterocolitica
F–swine
Rodentiosis is caused by Y. pseudotuberculosis
T
Y. pseudotuberculosis is relatively resistant and replicates in the environment
T
Arthritis can occur in the chronic form of the Y. pseudotuberculosis
T
Lesions are most often seen in the small intestines in case of Y. pseudotuberculosis
F
Y. pseudotuberculosis is normally present in rodents
F
Y. pseudotuberculosis sporadically causes abortion in cattle
T
Y. pseudotuberculosis can cause inflammatory necrosis lesions in rodent
T
Disease caused by Y. pseudotuberculosis is common in rodents and hares
T
Foot rot is more severe in cattle than in sheep
F
The hoof can be detached in the case of foot rot
T
Virulent foot rot can be treated with antibiotics
T
Foot rot occurs only in sheep
F
The agent of foot rot can produce proteases; they are virulence factors
T
the causative agent of foot rot is dichelobacter nodosus
T
the main virulence factors of dichelobacter nodosus are toxins
F—proteases
the warm and wet environment can predispose to foot rot
T
Keratinsase production is a virulence factor of the agent of Foot Rot
T
Foot Rot cannot be prevented by vaccination
F
Production of keratinase is a virulence factor of Dichelobacter nodosus
T
Morbidity of foot rot is very low, 1-5%.
F
Foot bath in zinc sulphate can be used for the treatment of foot rot
T
Dichelobacter nodosus causes foot rot in sheep
T
Dichelobacter nodosus is helped by other bacteria in the pathogenesis of foot rot
T
Dichelobacter nodosus can produce keratinase
T
Foot rot has more severe clinical signs in goats than sheep
F
Exotoxins of the causative agent are responsible for the lesions of foot rot
F–ec enzymes
Extracellular enzymes of the causative agent are responsible for the lesions of foot rot
T
Footbath with formalin can be used for prevention or for treatment of milder cases of foot rot
T
Toxins are the main virulence factors of Dichelobacter nodosus
F
Warm and wet climate predispose animals to foot rot
T
Animals with softened hoof wall are more susceptible for panaritium (foot rot).
T
Contaminated, dirty bedding is a predisposing factor for foot rot
T
In the development of foot rot, fimbriae of the pathogen have an important role
T
Treatment of foot rot is using foot bath containing formalin and sterogenol
T
For treatment of panaritium, formalin foot baths are recommended
T
Foot rot is a very rare disease in Europe
F
Fimbria and extracellular enzymes are the virulence factors of the agent of foot rot
T
Dichelobacter causes Necrobacillosis
F
The causative agent of foot root can produce exotoxins
F
The causative agent of foot root can produce enzymes
T
Foot rot can be prevented by foot bath
T
Animals with soft horn are more predisposed to panaritium
T
Strongly contaminated bedding is a predisposing factor to panaritium
T
The fimbriae of the pathogens play an important role in the development of panaritium
T
To treat severe foot rot we use parenteral antibiotics
T
Fusobacterium necrophorum subsp. necrophorum causes necrobacillosis
T
Fusobacterium necrophorum subsp. necrophorum can help the agent of foot rot in causing disease
T
Aerosol infection is a common form of transmission of the agent of necrobacillosis
F
Necrobacillosis is mainly seen in young animals
T
Rumen parakeratotosis can predispose to necrobacillosis
T
Insufficient disinfection of the navel can predispose to necrobacillosis
T
Abortion is a frequent clinical sign of necrobacillosis
F
Laryngeal erosion serves as place of entry for the agent of calf diphtheria
T
calf diphtheria is caused by Dichelobacter nodosus
F
Calf diphtheria is caused by Fusobacterium necrophorum subsp. necrophorum
T
Respiratory diseases can predispose to calf diphtheria
T
Fusobacterium necrophorum subsp. funduliforme is the agent Necrobacillosis in lambs
F
Necrobacillosis can occur as a consequence of navel infection in lambs
T
Pulmonary necrosis is a frequent lesion of necrobacillosis of lambs
F
Liver necrosis is a frequent lesion of necrobacillosis of lambs
T
Fusobacterium funduliforme is the causative agent of Necrobacillosis
F
Necrosis of the mucous membranes in the oral cavity is a clinical sign of Necrobacillosis
T
Focal necrosis in the kidney is a frequent post mortem lesion of Necrobacillosis
F
Necrobacillosis is prevented with wide vaccination
F
Wounds can predispose to Necrobacillosis
T
Navel infection can occur in the case of Necrobacillosis
T
Lesions of Necrobacillosis can be seen frequently in the oral cavity
T
Rough, sticky feed can predispose to Necrobacillosis
T
Necrosis in the liver can be a post mortem lesion of Necrobacillosis
T
Necrobacillosis can only be seen in lambs
F
In the case of Necrobacillosis infection can happen through wounds
T
In the case of Necrobacillosis, aerogenic infection is common
F
Lesions of Necrobacillosis are frequently found in or around the oral cavity
T
Fusobacteria are highly resistant, they remain viable in the environment for several weeks
F
Wounds and abrasions can predispose to Necrobacillosis
T
Incorrect disinfection of the naval area can lead to Necrobacillosis
T
Wounds on the oral mucosa can predispose animals to Necrobacillosis
T
The clinical signs of Necrobacillosis can frequently be seen in young animals
T
Necrobacillosis is a generalized disease with high fever and depression
F
Vaccines containing attenuated agents are widely used to prevent Necrobacillosis
F
Necrobacillosis is caused by Dichelobacter nodosus
F
Foot rot is the main sign of Necrobacillosis
F
The lesions in Necrobacillosis are restricted to the mouth
F
Necrobacillosis occurs mainly in young animals
T
There is no treatment for Necrobacillosis
F
Umbilical disinfection has an important role in the prevention of Necrobacillosis
T
Necrobacillosis in lambs can be seen as a result of an umbilical infection
T
Fusobacterium necrophorum subsp. necrophorum strains can cause Necrobacillosis of calves
T
You can find necrosis in the mouth in case of Necrobacillosis in lambs
T
You can find liver abscesses in case of Necrobacillosis
F—liver focal necrosis
