Pasteurellosis Flashcards
Asymptomatic carriage of Pasteurella multocida can occur in birds
T
Pasteurella multocida is highly resistant; it can survive in the environment for several months
F
Pasteurella multocida can cause pasteurellosis in small ruminants
T
Pasteurella multocida can infect humans
T
Primary pasteurellosis disease have no predisposing factors
F
They have but they are usually non infectious
Pasteurella multocida can complicate canine distemper
T
Leukotoxin (cytotoxin) production is an important virulence factor of Mannheimia haemolytica
T
The dermonecrotoxin of Pasteurella multocida damages the osteoclast cells
F
The osteoblasts there is decreased production.
Primary pasteurellosis diseases are caused by obligate pathogenic Pasteurella bacteria
F
Dermonecrotoxin can be virulence factor of Pasteurella multocida
T
Dermonecrotoxin of pasteurella multocida causes lesions in the nasal cavity and on the skin of pigs
F
Capsule can be virulence factor of Pasteurella multocida
T
Enterotoxins are virulence factors of Pasteurella multocida
F
Mannheimia haemolytica leukotoxin (cytotoxin) damages the macrophages
T
Transportation is a predisposing factor of respiratory pasteurellosis of calves
T
Primary pasteurellosis diseases have non infectious predisposing factors
T
Emboli are responsible for the necrosis seen in the case of acute systemic pasteurellosis
T
Leukotoxin is a virulence factor of Mannheimia haemolytica
T
Leukotoxin is produced by the majority of Pasteurella multocida
F
Pasteurella multocida can cause disease only in ruminants
F
Mannheimia haemolytica can cause disease mainly in ruminants
T
Bibersteinia trehalosi can infect ruminants
T
Pasteurellaceae are resistant bacteria, they remain viable for a long time in the soil
F
The virulence factors of P. multocida are the capsule and dermonecrotoxin
T
Pasteurella are very resistant in the environment
F
Pasteurella are obligate pathogens
F
We can use inactivated bacteria as a vaccine for Pasteurella
T
Pasteurella bovis can cause pasteurellosis in cattle.
F
Diarrhoea is the main clinical sign of bovine pasteurellosis
F
Overcrowding is a predisposing factor of respiratory pasteurellosis of cattle
T
The lesions of respiratory pasteurellosis of calves are generally in the diaphragmatic lobes
F
Dermonecrotoxin of the agents is responsible for the clinical signs of respiratory pasteurellosis of cattle.
F
Respiratory pasteurellosis occurs only among cattle above 6 months of age
F
Toxoid vaccines can be used for the prevention of respiratory pasteurellosis of cattle
T
Pasteurella multocida can cause pasteurellosis in cattle
T
mannheimia haemolytica can cause respiratory pasteurellosis of calves
T
Mannheimia haemolytica can cause pasteurellosis in cattle
T
Respiratory pasteurellosis can occur after transport
T
Mannheimia haemolytica is an important agent of shipping fever
T 90%
Leukotoxin of M. haemolytica damages the alveolar macrophages
T
Interstitial pneumonia is a frequent post mortem lesion of pulmonary pasteurellosis of cattle
F
Salmonellosis can predispose cattle to pasteurellosis
F
Transportation can predispose cattle to pasteurellosis
T
Bovine pasteurellosis cannot be prevented with vaccination
F
Respiratory pasteurellosis has been already eradicated in Europe
F
Mannheimia haemolytica is a causative agent of respiratory pasteurellosis
T
Mycoplasmas can predispose cattle to respiratory pasteurellosis
T
Respiratory pasteurellosis in cattle is caused by P. multocida A and M. haemolytica A
T
Most importance virulence factor of M. haemolytica is leukotoxin
T
Respiratory pasteurellosis in cattle is a common disease of young animals
T
Macrolides are recommended to use in case of respiratory pasteurellosis in cattle
T
Respiratory pasteurellosis of cattle appears in calves aged 1-3 months old
T
Respiratory pasteurellosis of cattle causes a fibrinous inflammation of serous membranes
T
Leukotoxin of M. haemolytica strains responsible for Pasteurella pneumonia attacks respiratory epithelium
T
Respiratory Pasteurellosis of cattle are mostly seen in the tropics
F??????
Septicaemia of lambs and kids and acute haemorrhagic septicaemia of swine in tropical areas
Respiratory pasteurellosis of cattle causes purulent pneumonia
F
We can use ELISA to diagnose respiratory pasteurellosis in cattle
T
A predisposing factor of respiratory pasteurellosis in cattle is transport
T
A predisposing factor of respiratory pasteurellosis in cattle is the presence of a viral infection
T
Vaccination is widely used for prevention of bovine respiratory pasteurellosis
T
Haemorrhagic septicaemia of cattle is generally a chronic disease
F
Pasteurella multocida strains are causative agents of haemorrhagic septicaemia of cattle
T
Antibiotic treatment at the time of appearance of the clinical signs of haemorrhagic septicaemia of cattle is generally late
T
Bleeding from the nose is a frequent clinical sign of haemorrhagic septicaemia of cattle
F
Haemorrhagic septicaemia occurs most frequently among 2-3 month old calves in endemic areas
F
The morbidity and mortality of Haemorrhagic septicaemia of cattle is low
F
Oedema formation can be a clinical sign of haemorrhagic septicaemia of cattle
T
Hemorrhagic septicemia mainly occurs in tropical and sub-tropical areas.
T
Pasteurella multocida B:2 and E:2 strains are causative agents of Haemorrhagic septicaemia
T
Monsoon can predispose to haemorrhagic septicaemia
T
Haemorrhagic septicaemia is mainly seen in pigs and horses
F
Haemorrhagic septicaemia is mainly an acute disease
T
After recovering from haemorrhagic septicaemia the animals do not shed the agent
F
Exotoxins are responsible for haemorrhagic septicaemia
F
The agent of haemorrhagic septicaemia can be carried in the tonsils
T
Haemorrhagic septicaemia cannot be prevented with vaccines
F
Haemorrhages on the heart are important post mortem signs of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is caused by Pasteurella multocida
T
Haemorrhagic septicaemia is mainly seen in pigs
F
Oedema can be seen in the case of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is endemic in several European countries
F
Exhausting work can predispose to haemorrhagic septicaemia
T
Re-convalescent animals can carry the agent of haemorrhagic septicaemia
T
The lesions of haemorrhagic septicaemia are caused by the exotoxin of the agent
F
Aerogenic infection is frequent in the case of haemorrhagic septicaemia
T
Haemorrhagic septicaemia is caused by Mannheimia haemolytica
T
The leukotoxin of the agent is responsible for the lesions of haemorrhagic septicaemia
F
Fever is a typical sign of haemorrhagic septicaemia
T
Haemorrhagic septicaemia occurs only in tropical and subtropical countries
F
The morbidity of haemorrhagic septicaemia is high
T
There are no vaccines for the prevention of haemorrhagic septicaemia
F
Haemorrhagic septicaemia is caused by B and E types
T
Haemorrhagic septicaemia of cattle generally occurs in tropical countries
T
Haemorrhagic septicaemia infects cattle mainly from the environment
F
Haemorrhagic septicaemia of cattle’s endotoxin causes clinical symptoms
T
endotoxin effect coagulopathies haemorrhages of p. multocida
Pasteurella multocida is the agent of haemorrhagic septicaemia of cattle
T
The clinical signs of haemorrhagic septicaemia are caused by the dermonecrotoxin of the agent
F
Buffalos and cattle are the most susceptible species to haemorrhagic septicaemia
T
Haemorrhagic septicaemia is mainly seen in 1-2 months old animals
F
Bleeding is the most typical clinical sign of haemorrhagic septicaemia
F
Haemorrhagic septicaemia can be prevented with vaccination
T
Haemorrhagic septicaemia infected animals carry the bacteria for 1 year
F
Hyaluronic acid capsule is important for the bacteria’s ability to replicate unhindered
T
Haemorrhagic septicaemia is a slow and chronic disease
F
Endotoxins are important for the pathogenesis of the haemorrhagic septicaemia disease
T
Antibiotics are highly effective against haemorrhagic septicaemia
T
Attenuated vaccines can be used to prevent haemorrhagic septicaemia
T
After recovery from haemorrhagic septicaemia, cattle remain carriers
T
Haemorrhagic septicaemia of cattle occurs only in tropical countries
F
Secondary infection can occur with haemorrhagic septicaemia of cattle
F
Haemorrhagic septicaemia of cattle can be explained by endotoxin production
T
Haemorrhagic septicaemia is mainly seen in sheep and horses
F
Septicaemic ovine pasteurellosis is mainly seen in pregnant animals
F
Acute systemic pasteurellosis occurs mainly among 3-12 months old lambs
T
Pasteurella ovis can cause pasteurellosis in small ruminants
F
Bibersteinia trehalosi can cause acute systemic pasteurellosis in small ruminants
T
Cough and nasal discharge are clinical signs of ovine pasteurellosis
T
Acute systemic pasteurellosis is caused by Pasteurella multocida.
F
Arthritis can be a clinical sign of ovine pasteurellosis
T
Acute systemic pasteurellosis does not occur in goats only in sheep
F
Septicemia ovine pasteurellosis is mainly seen in lambs younger than 3 months
T
Mastitis can be a clinical form of ovine pasteurellosis
T
septicemia can be a clinical form of ovine pateurellosis
T
septicemic pasteurellosis doesn’t occur in goats only in sheep
F
respiratory pasteurellosis occurs in goats
T
ewes have to be culled after mastitis caused by mannheimia haemolytica because the udder cannot regenerate
F
inactivated vaccines can be used for the prevention of ovine pasteurellosis
T
respiratory viruses can predispose sheep to pasteurellosis
T
mannheimia haemolytica cause mastitis in goats
T
fibrinous pleuropneumonia is a postmortem lesion of ovine pasteurellosis
T
Respiratory pasteurellosis can occur in goats
T
Septicaemia can be a clinical form of ovine pasteurellosis
T
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of the pigs
T
Mannheimia haemolyitica can cause Pasteurellosis in small ruminants
T
Bibersteinia trehalosi can cause Pasteurellosis in small ruminants
T
Enteritis is a frequent clinical sign of ovine pasteurellosis
F
The agent of Acute Systemic Pasteurellosis is zoonotic
F
Mannheimia haemolytica can cause respiratory pasteurellosis of sheep
T
Fibrinous pneumonia is a common post mortem lesion of respiratory pasteurellosis
T
Mannheimia haemolytica can cause mastitis of sheep
T
Respiratory pasteurellosis of sheep cannot be prevented with vaccines
F
Mannheimia haemolytica is an important agent of ovine respiratory pasteurellosis
T
Leukotoxin of M. haemolytica is responsible for the clinical signs of ovine respiratory pasteurellosis
T
Lesions of ovine respiratory pasteurellosis can be seen in the anterior lobes of the lungs
T
Macrolide antibiotics can be used to the treatment of ovine respiratory pasteurellosis
T
Acute systemic pasteurellosis occurs in lambs that are younger than 3 months
F
Acute systemic pasteurellosis is caused by Mannheimia haemolytica
F
Acute systemic pasteurellosis occurs in sheep but not goats
F
Respiratory form of ovine pasteurellosis can be seen only in suckling lambs
F
Leukotoxin produced Mannheimia haemolytica is responsible for ovine mastitis
T
Acute systemic pasteurellosis is caused by Pasteurella multocida
F
Lesions of ovine respiratory pasteurellosis are typically seen in the diaphragmatic lobe of the lungs
F
Mannheimia haemolytica can cause pneumonia in sheep
T
Mannheimia haemolytica can cause acute systemic pasteurellosis in sheep
F
Mannheimia haemolytica can produce dermonecrotoxin
F
Mannheimia haemolytica can cause septicaemia in suckling lambs
T
Acute systemic pasteurellosis is caused by Bibersteinia trehalosi
T
Acute systemic pasteurellosis occurs in sucking lambs and kids
F
In the case of acute systemic pasteurellosis bacterium, emboli are formed in the blood vessels
T
Nasal discharge and coughing for a week are the main clinical signs of acute systemic pasteurellosis
F
Acute systemic pasteurellosis is caused by type A and D of Pasteurella multocida
F
Necrosis of the mucous membranes and focal necrosis in the parenchymal organs are typical lesions of acute systemic pasteurellosis
T
Antibiotics can be used for the treatment of respiratory pasteurellosis of sheep
T
Respiratory pasteurellosis in sheep cannot be prevented with vaccination
F
Se-deficiency is a predisposing factor in sheep for pasteurellosis
T
Mannheimia haemolytica ́s endotoxin causes respiratory pasteurellosis in sheep
T
In respiratory pasteurellosis in sheep the cranial lung lobes are affected
T
Pasteurellosis does not cause septicaemia
F
Acute systemic pasteurellosis typically occurs in 3-12 months old small ruminants
T
Bacterium emboli are responsible for the clinical signs and lesions of acute systemic pasteurellosis
T
Fibrinous pneumonia is the main post mortem lesion of acute systemic pasteurellosis
F
Bibersteinia trehalosi is involved in development of acute systemic pasteurellosis
T
Serology is widely used to diagnose Pasteurella in sheep
F
Penicillin can be used to treat ovine pasteurellosis
T
The septicaemic form of pasteurellosis is most common in sheep above 1 year
F
You can see croupus pneumonia in case of ovine systemic pasteurellosis
F
Systemic pasturellosis is seen in 2-4-week-old lambs
F
Pasteurella causes septicaemia in lambs
T
In suckling lambs, acute systemic pasteurellosis may occur
F
Pasteurellosis in the sheep may cause interstitial pneumonia
F
Pasteurellosis in the sheep can occur in the form of mastitis
T
Pasteurellosis in sheep may occur as a septicaemia
T
Emboli is the cause of sudden death in acute systemic pasturellosis
T
Vaccines can be used to prevent pasteurellosis in sheep
T
In the respiratory form of ovine pasteurellosis, haemorrhagic pneumonia is seen
F
Mycoplasmas predispose pigs to pulmonary pasteurellosis
T
Pulmonary pasteurellosis is more frequent in suckling piglets than in adults
F
Bordetella bronchiseptica predisposes pigs to pulmonary pasteurellosis
F
Pasteurella multocida can cause haemorrhagic septicaemia in pigs
T
Pulmonary pasteurellosis of pigs is mainly caused by Mannheimia hemolytica
F
respiratory pasteurellosis of pigs is generally an acute, generalized disease
F
Vaccines can prevent pneumonia of swine caused by pasteurella
F
P. multocida A is causative agent of pneumonia caused by pasteurellosis in swine
T
Rabbit pasteurellosis is a generalised disease
T
Pasteurella multocida A and D strains can cause pasteurellosis in rabbits
T
Clinical signs of pasteurellosis in rabbits are most severe in new-born animals
F
The agent of rabbit pasteurellosis can cause septicaemia
T
Nasal discharge is a typical clinical sign of rabbit pasteurellosis
T
Subcutaneous abscesses can be seen in the case of rabbit pasteurellosis
T
Otitis media can be a clinical sign of rabbit pasteurellosis
T
Rabbit pasteurellosis causes only respiratory clinical signs
F
rabbit pasteurellosis is more frequent in large scale farms than in small ones
T
Mannheimia haemolytica causes rabbit pasteurellosis
F
Pasteurellosis is limited to the respiratory tract in rabbits
F
Bibersteinia trehalosi is the causative agent pasteurellosis of rabbits
F
Serous pneumonia is typical in the case of pasteurellosis of rabbits
F
Pasteurellosis is limited to the respiratory track in rabbits
F
Neurologic signs can be seen in the case of pasteurellosis of rabbits
T
Pasteurellosis is common in suckling rabbits
F
The poor quality of the air can predispose to rabbit pasteurellosis
T
Purulent bronchopneumonia is a frequent post mortem lesion of rabbit pasteurellosis
T
Fibrinous pneumonia can be seen during necropsy in case of pasteurellosis in rabbit
T
Pasteurellosis in rabbit causes high amount of nasal discharge
T
Pasteurellosis in rabbit is caused by P. multocida A
T
Europe is free from atrophic rhinitis
F
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of pig
T
Dermonecrotoxin producing Pasteurella multocida can cause irreversible lesions in the nose of pigs
T
Toxoid vaccines are used for the prevention of atrophic rhinitis
T
The block of the lachrymal channel is a clinical sign of atrophic rhinitis
T
The turbinate bones can absorbed in the case of atrophic rhinitis
T
Overcrowding can predispose pigs to atrophic rhinitis
T
The maxilla can be shortened in the case of atrophic rhinitis
T
The most severe clinical signs of atrophic rhinitis can be seen in suckling piglets
F
Atrophic rhinitis can be prevented by vaccinating the pregnant sows
T
The lesions of atrophic rhinitis are examined after sawing the nose behind the first premolar teeth
T
Clinical signs of a atrophic rhinitis appear if piglets are infected in the first few weeks of their life:
T
The mortality and the economic impact of atrophic rhinitis are high
F
low mortality high eco impact
Atrophic rhinitis in fattening pigs is caused by Bordetella bronchiseptica
T
In 4-6 months old pigs B. bronchiseptica strains cause severe pneumonia
F
Dermonectotoxin is an important virulence factor of B. bronchiseptica
T
Atrophic rhinitis cannot be prevented with vaccination
F
Block of the lacrimal channel is a typical sign of atrophic rhinitis
T
The dermonecrotoxin producing Pasteurella multocida strains are responsible for the severe lesions of atrophic rhinitis
T
The dermonecrotoxin of P. multocida inhibits the activity of the osteoclast cells
F
Atrophic rhinitis can be prevented by vaccinating the day-old piglets
T
Atrophy of the turbinate bones is a typical lesion of atrophic rhinitis
T
Isolation of the causative agent from the nose confirm the diagnosis of atrophic rhinitis
F
The effects of the dermonecrotoxin produced by Pasteurella multocida are reversible
F
Bordetella bronchiseptica and Pasteurella multocida cause atrophic rhinitis
T
Bordetella bronchiseptica causes reversible lesions in the nasal cavity of pigs
T
Infection of pigs with Bordetella bronchiseptica and Pasteurella multocida at any age can result in atrophic rhinitis
F
The mortality of atrophic rhinitis can be 50-60%.
F
Clinical signs of atrophic rhinitis can be seen if piglets infected first week of life
T
The lesions of atrophic rhinitis are reversible
T
The lesions of atrophic rhinitis are caused by an endotoxin
F
Hungary is free from atrophic rhinitis of swine
F
The clinical form of atrophic rhinitis can be seen if the piglets were few weeks old when infected
F
Bordetella bronchiseptica causes reversible changes in swine
T
At atrophic rhinitis the conchae absorb
T
Isolating Pasteurella multocida from pigs’ noses proves atrophic rhinitis
F
The endotoxin of the agent is responsible for the clinical signs and lesions of atrophic rhinitis
F
Atrophic rhinitis can be prevented with toxoid vaccine given at weaning
F
PM lesions of atrophic rhinitis can be examined after transverse cut of the nose
T
Atrophic rhinitis is examined PM by a longitudinal section of the nose
F
Animals showing signs of atrophic rhinitis remain carriers
T
Atrophic rhinitis can be prevented by vaccinating the sow
T
Atrophic rhinitis cause disease in swine and calves
F
Dermonectotocin in case of atrophic rhinitis acts on the osteoblast cells
T
Atrophic rhinitis is caused by a synergistic interaction between B. bronchiseptica and P. multocida D
T
B. bronchiseptica can cause immunosuppression
T
Toxoid vaccines can be used for prevention of atrophic rhinitis
T
B. bronchiseptica strains producing toxins causing serious lesions
F
The typical PM lesions of atrophic rhinitis are caused by B. bronchiseptica strain
F
Atrophic rhinitis is a common disease that causes severe losses
F
Atrophic rhinitis only occurs pigs that were infected as suckling piglets
F
Atrophic rhinitis is proven by isolating P. multocida
F
Tetracyclines can successfully be used to treat atrophic rhinitis
T
Wrinkles and torsion of nose is the most prominent clinical sign of atrophic rhinitis
T
Fowl cholera is caused by Pasteurella multocida A, D and F strains
T
Fowl cholera can occur is ducks and geese
T
The agent of fowl cholera can survive in water for a few days
T
Fowl cholera is caused by Pasteurella gallinarum
F
The mortality of fowl cholera is low
F
Arthritis is a clinical sign of fowl cholera
T
The resistance of the agent of fowl cholera is low
T
Fowl cholera occurs mainly in the winter in Europe
F
Pasteurella multocida strains with lower virulence can cause chronic fowl cholera
T
The most severe form of fowl cholera occurs in day-old birds
F
Waterfowl is more susceptible to fowl cholera than hens
T
Focal inflammation and necrosis in the liver is a common postmortem lesion on of fowl cholera
T
Wild birds can introduce the agent of fowl cholera into a herd
T
Fowl cholera has an exponential mortality rate
T
Fowl cholera is a generalized disease it causes septicaemia
T
Animals recovered after fowl cholera remain bacterium carriers
T
The dermonecrotoxin of the agent is responsible for the clinical sign of fowl cholera
T
Vaccine for the prevention of fowl cholera provide type specific protection
T
The agent of fowl cholera is an obligate pathogenic bacterium
F
Antibiotics cannot be used for the treatment of fowl cholera
F
human can introduce the agent of fowl cholera into a herd
T
per os antibiotic treatment must be used in the case of fowl cholera
T
nasal discharge and conjunctivitis are clinical signs of fowl cholera
T
the morbidity of fowl cholera is high
T
the exotoxin of the agent is responsible for the clinical signs of fowl cholera
F
inflammation of the wattle is a clinical sign of acute fowl cholera
F
the endotoxin of the agent is responsible for the clinical signs of fowl cholera
T
Europe is free from fowl cholera
F
Haemorrhagic diarrhoea is a clinical sign of fowl cholera
T
Fowl Cholera can occur in ducks and geese
T
Fowl cholera is always an acute disease
F
Feather pecking or force feeding can predispose animals to fowl cholera
T
Parent birds have to be vaccinated just before laying in order to prevent fowl cholera
F
Germinative infection is a frequent way of transmission of the agent of fowl cholera
F
Geese is resistant to fowl cholera
F
Fowl cholera cannot be prevented by vaccination
F
Focal inflammation in the liver is a typical post mortem lesion of fowl cholera
T
Hemorrhages generally cannot be seen as post mortem signs of fowl cholera
F
After recovering from fowl cholera the animals do not carry the agent any more
F
There are vaccines on the market to prevent fowl cholera
T
Fowl cholera occurs only in hens
F
Fowl cholera is caused by certain serotypes of Pasteurella multocida
T
The agent of fowl cholera is an obligate pathogen
F
Germinative infection is common in the case of fowl cholera
F
Turkey are highly susceptible to fowl cholera
T
Fowl cholera is caused by Pasteurella multocida strains
T
Fowl cholera occurs only in chicken
F
Fowl cholera is typically seen in day-old birds
F
Fowl cholera is a septicemic disease
T
The agent of fowl cholera is a facultative pathogenic bacterium
T
The endotoxin of the agent is responsible for the lesions of fowl cholera
T
Fowl cholera is caused by Riemerella anatipestifer
F
Endotoxins of Pasteurella multocida cause fowl cholera
T
Fowl cholera has always an acute course
F
Fowl cholera could cause focal inflammation-necrosis in the liver
T
Fowl cholera is caused by introducing highly virulent Pasteurella multocida strains
T
Plucking and fattening of geese is a predisposing factor for fowl cholera
T
Fowl cholera causes acute septicaemia
T
The chronic form of fowl cholera is caused by less virulent strains
T
Day-old chickens are resistant to fowl cholera
T
Vaccines against fowl cholera are not available
F
Fowl cholera may cause high mortality in water birds
T
Turkeys are not sensitive to fowl cholera
F
Fowl cholera occurs mainly at the end of the winter
F
Fowl cholera occurs mostly during the summer and autumn
T
Germinative infection is the primary way of spreading fowl cholera
F
Mortality of fowl cholera can reach 100% in a susceptible flock
T
In development of fowl cholera, plucking the animal has an important role
T
Fowl cholera can cause high losses among day-old chicken
F
Fowl cholera is caused by leukotoxin-producing Pasteurella (Mannheimia) haemolytica
F
Fowl cholera has higher mortality in young broiler stocks
F
Fowl cholera has high mortality in water poultry
F
T??
Fowl cholera is caused by Pasteurella haemolytica
F
Fowl choler occurs in the tropics and subtropics mostly
T
Wild birds are very susceptible to fowl cholera
F
Susceptibility in hens decrease with age in case of fowl cholera
F
