Urinary: Infection Flashcards

1
Q

Classic appearance of pyelonephritis on CT

A
  • Striated nephrogram (best seen on excretory phase)
  • Vague, focally reduced areas of enhancement with associated perinephric stranding
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2
Q

How can you differentiate between pyelonephritis and renal infarction on imaging?

A

Cortical rim sign:

Pyelonephritis classically involves a complete wedge of kidney extending through the cortex

Infarct classically involved a central wedge where there is a cortical rim of normally enhancing cortex peripherally

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3
Q

Pyelonephrosis

A

Pyelonephritis with obstruction

Note: A fluid-fluid level is often seen on ultrasound.

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4
Q

Differential for striated nephrogram

A

Things that cause hypoperfusion or edema:

  • Acute ureteral obstruction
  • Acute pyelonephritis
  • Acute renal vein thrombosis
  • Radiation nephritis
  • Acutely following renal contusion
  • Hypotension (would be bilateral)
  • Infantile polycystic kidney (would be bilateral)
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5
Q

Treatment for pyelonephrosis

A

Urgent decompression with percutaneous nephrostomy

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6
Q

Treatment for renal abscess

A

IR-guided drainage (if >3 cm)

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7
Q

Spectrum of pyelonephritis

A
  • Pyelonephritis (striated nephrogram)
  • Lobar nephronia (phlegmon: ill-defined focal low density but not yet cystic, more common in pediatrics)
  • Renal abscess (well-defined cystic core, usually with a thick enhancing rim)
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8
Q

CT appearance of chronic pyelonephritis

A

Renal scarring with atrophy/cortical thinning and hypertrophied residual tissue

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9
Q
A

Emphysematous pyelonephritis

Note: Perinephric echogenic line with dirty shadowing.

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10
Q

Emphysematous pyelonephritis is most common in what pt population?

A

Diabetics

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11
Q

Emphysematous pyelitis

A

When gas is limited to the collecting system (not as bad as emphysematous pyelonephritis)

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12
Q
A

Emphysematous pyelitis

Note: Gas limited to collecting system.

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13
Q

Common causes of papillary necrosis

A
  • Diabetes (most common)
  • Pyelonephritis
  • Sickle cell disease
  • Tuberculosis
  • Analgesic use
  • Cirrhosis
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14
Q
A

Renal papillary necrosis

Note: “Lobster claw sign” of the renal calyx.

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15
Q
A

Renal papillary necrosis

Note: Linear streaks/filling defects in renal calyces.

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16
Q

What percentage of pts with sickle cell disease develop renal papillary necrosis?

17
Q
A

Xanthogranulomatous pyelonephritis (in the setting of stag horn calculus)

Note: Characteristic “bear paw” appearance. This kidney is not functional.

18
Q

Xanthogranulomatous pyelonephritis

A

A chronic destructive granulomatous process involving the kidney (virtually always seen in the setting of a stag horn calculus), giving a characteristic “bear paw” appearance on CT. The affected kidney is not function and sometimes treated with nephrectomy.

19
Q

Pts with xanthogranulomatous pyelonephritis are at high risk for…

A

Psoas abscess

20
Q

AIDS pt with nephrotic syndrome (massive proteinuria)…

A

HIV nephropathy

Note: Bilateral hyperdense kidneys (more dense than liver) in an AIDS pt with nephropathy.

21
Q

Diagnosis of HIV nephropathy

22
Q

Most common cause of renal impairment in AIDS pts

A

HIV nephropathy

23
Q

HIV pt

A

Disseminated PCP infection

Note: Punctate cortical calcifications, appears the same as cortical nephrocalcinosis.

24
Q

What is the most common extra-pulmonary site of tuberculosis?

A

Urinary tract

25
What is the earliest finding of renal tuberculosis?
Calyceal blunting ("most eaten calyces") Note: This is nonspecific and can be seen with ischemia, diabetes, etc.
26
Renal tuberculosis Note: Dilated calyces with "cavity" formation and calcifications.
27
Renal tuberculosis Note: Distorted calyces with dilatation and papillary necrosis and classic "Kerr Kink" at the renal pelviureteral junction.
28
Renal tuberculosis Note: Complete replacement of the kidney with calcifications ("putty kidney").
29
What peritoneal sign might help you identify renal abnormalities as renal tuberculosis?
The presence of multiple calcified mesenteric lymph nodes
30
Risk factors for contrast-induced nephropathy
- Renal insufficiency - Diabetes - Heart failure - Dehydration - Myeloma
31
What can be done to minimize contrast induced nephropathy in high risk pts?
IV 0.9% normal saline 6-12 hours before the study and continuing 4-12 hours after Note: Oral hydration does not work as well.