*Test Four Flashcards
What happens to blood CO2 levels during respiratory acidosis?
goes up
peripheral chemoreceptors
monitor pCO2 and H+ concentrations in body
Two types of alveolar cells
- type I alveolar cells- “gas exchange between air and blood happens across these cells”
- type II alveolar cells- make surfactant
3 factors that enhance glomerular filtration (and BHP)
- long glomerular capillaries increase resistance and pressure
- walls of glomerulus and capsule are thin and porus
- arterioles- afferent are larger than efferent, creating back pressure (which pushes everything into capsular space)
Juxtaglomerular cells
make renin, recognize low blood volume, starts renin-angiotensin pathway
apneustic center
slows down respiration
normal baby breathing
- baby inhales water with its first breath
- when baby exhales the alveoli collapse
- this collapseallows water molecules in the alveoli to draw near each other
- this nearness creates hydrogen bonds and surface tension
- type II alveolar cells make surfactant, disrupting hydrogen bonds/surface tension, allowing alveoli to reinflate
Hyaline Membrane Disease (why and how do you treat)
- no surfactant before 40 weeks, so preemies can’t break the hydrogen bonds (water surface tension) to reinflate the alveoli
- solution- shoot mom with a steroid, give baby surfactant
uncontrolled diabetics and polyphagia (why?)
“uncontrolled diabetics have excessive glucose in the blood, but no insulin to open doorways for glucose to get into the cells. The cells are starving and sending the message to eat”
epiglottis
flap of tissue guarding glottis, directs food and drink to esophagus
polyuria
overproduction of urine
Boyle’s law in relation to ventilation
- relaxed diaphragm- lung and outside air pressure are equal (maybe 760mmHg)
- diaphragm contracts, increases lung volume, LOWERING pressure
- inhale- high pressure outside, low pressure inside pushes air in
- air pressure goes to equal or slightly higher (780 mmHg)
- relax the diaphragm- volume lowers, pressure inside increases, exhale
urinary retention
failure to release urine
renal auto regulation
ability of kidneys to maintain a constant glomerular filtration rate despite changes
intercalated cells
cells involved in pH adjustment (compensatory response cells, move H+ between blood and filtrate)
(increased) urinary frequency
voiding more than normal
Internal respiration
exchange of gasses between blood and tissues
Bohr effect
(how pH affects affinity)
- CO2 from tissues goes to blood
- this causes acidosis (it’s generating H+ from H2O)
- hemoglobin recognizes the pH drop and lets go of its O2
- hemoglobing then picks up the extra H+, rasing pH
Where are the pneumotaxic and apneustic centers?
the pons
compensatory response
when kidneys correct acidosis caused by respiratory system (body isn’t going to just give up)
renal hilus
area of kidney where blood vessels, nerves, and ureter enter/exit
uncontrolled diabetics and metabolic acidosis (why?)
can’t get glucose into cells to make ATP, so cells use fat for energy. when fats are converted to ATP, acidic ketones are produced, causing ketoacidosis (metabolic acidosis) in the blood