*Final Exam Flashcards
Prostate
citric acid secretions (potential ATP source), enlarges with age, declotting factors
glomerulus
many folded capillaries
vas deferens
tube transports sperm back into body (outside for lower temp- dartos and cremaster muscles help this)
Plexus of Meissner
neurons for mucus glands (food lubrication) in submucosa layer
erythropoietin
growth factor synthesized by kidneys and functions in the differentiation of RBCs
4 breathing patterns
- eupnea- normal breathing (16-20 per min)
- apnea- temporary cessation of breathing (snoring, overweight, alcohol, preemies)
- dyspnea- painful breathing
- tachypnea- rapid breathing (~40 per min)
testosterone
major male hormone, made by leydig cells, male embryonic development, secondary traits, aggression
passive artificial immunity
antibody shot (rabies shots)
secondary oocyte (what and where)
telophase I, generates first polar body, in graafian follicle, becomes egg
Boyle’s law in relation to ventilation
relaxed diaphragm- lung and outside air pressure are equal (maybe 760mmHg)
diaphragm contracts, increases lung volume, LOWERING pressure
inhale- high pressure outside, low pressure inside pushes air in
air pressure goes to equal or slightly higher (780 mmHg)
relax the diaphragm- volume lowers, pressure inside increases, exhale
raphe
scrotal midline (used for surgery)
thymosin
thymus gland hormone that matures T cells after they leave the red bone marrow
uncontrolled diabetes and metabolic acidosis (why?)
can’t get glucose into cells to make ATP, so cells use fat for energy. when fats are converted to ATP, acidic ketones are produced, causing ketoacidosis (metabolic acidosis) in the blood
Juxtaglomerular cells
make renin, recognize low blood volume, starts renin-angiotensin pathway
salivary amylase
enzyme in saliva that breaks down carbs into sugars
Frank-Starling Law
increased preload causes increased contractility
passive natural immunity
mom and baby, breastmilk, placenta
Inhibin in females
stops FSH so pregnancy can progress, made by granulosa cells
What allows you to hold your breath?
cerebral cortex
larynx
anterior and inferior branch of pharynx, leads to respiratory system
active natural immunity
best, actually get disease (strep)
electrical conduction route through the heart
SA node, atria, AV node, atria contract together, bundle of His, bundle branches, Purkinje fibers, ventricles contract
ANP does what with the urinary system?
suppresses ADH and aldosterone, shuts down renin-angiotension pathway, promotes diuresis (urination)
estrogen
granulosa cells, secondary traits, embryo development, water balance, lowers cholesterol
T cells
combat intracellular antigens, these kill infected cells (Mr. T)
fimbriae
fingers, move egg to fallopian tubes
microvilli
tiny folds in the membrane of the epithelial cells lining the small intestine
Frank-Starling Law
increased preload causes increased contractility
Lack of ADH causes what?
diabetes insipidus
dysuria
painful urination
Where are all blood cells produced?
red bone marrow
progesterone (function and what cells produce)
from thecal cells of corpus luteum, pregnancy hormone, takes uterus from proliferative to secretory state, produce milk
uncontrolled diabetics and polyphagia (why?)
“uncontrolled diabetics have excessive glucose in the blood, but no insulin to open doorways for glucose to get into the cells. The cells are starving and sending the message to eat”
lymph nodes
ENCAPSULATED, found within vessels
zona pellucida
fluid layer initially seen in primary follicles, surrounds egg, immediately closes after fertilization (menstrual phase)
terminal bronchioles
smallest branches
What vessels surround the DCT and PCT?
peritubular capillaries
second line of defense
fever, phagocytic cells (WBCs), inflammatory response
polyuria
overproduction of urine
Pepsinogen/pepsin
HCl activates pepsinogen, converts it to pepsin, pepsin cuts up proteins into small strings called peptides
interleukins
“chemical messengers that coordinate the activities of the immune system”
corpus albicans
dead corpus luteum (no pregnancy) (post ovulatory phase)
uncontrolled diabetics and polyuria (why?)
with excess glucose in the blood, more is being filtered into the filtrate
the cell doors quickly reach Transport Maximum (Tm) and can’t get all the glucose back into the blood
“excess glucose goes down the descending limb, reversing osmosis, and making (LOTS OF) water enter the limb instead of leaving like it should”
kupffer cells
“major phagocytic cells of the liver”, destroy old RBCs, converts heme to bilirubin (enters bile, gives feces its color)
not working = chronic infections and jaundice (bilirubin buildup)
Acini cells
produce pancreatic juiceput HCO3- into pancreatic juice (which becomes sodium bicarbonate)
neutralizing acidic chyme entering small intestines
puts H+ into blood to correct alkaline nature
make the 8 pancreatic enzymes
renal autoregulation
ability of kidneys to maintain a constant glomerular filtration rate despite changes
segmentation
-muscles of the small intestine break chyme into sections and move it along
-breaks big fat globules into smaller globules
MAKES IT EASIER TO ABSORB FATS
antibodies (function and who makes)
made by B cells, destroy antigens
arytenoid cartilage
in back of larynx, controls movement of vocal chords
renal auto regulation
ability of kidneys to maintain a constant glomerular filtration rate despite changes
Juxtaglomerular apparatus (and what happens when you’re angry)
maintains normal glomerular blood pressure
EX- anger (causes BP to rise dramatically), juxtaglomerular apparatus will vasoconstrict afferent arterioles (less blood in) and vasodilate efferent arterioles (more blood out)
(increased) urinary frequency
voiding more than normal
primary oocyte (what and where)
prophase I, initial egg (crossing over & synapsis), in primordial follicle (all eggs are frozen in this state)
secondary bronchi
branches leading to lobes of lungs
What are reticulocytes and what makes them so odd?
immature RBCs that have expunged their nucleus and kept their organelles (mature RBC’s have no organelles)
neutrophils
phagocytic WBC that is first to a wound site
Bohr effect
(how pH affects affinity)
CO2 from tissues goes to blood
this causes acidosis (it’s generating H+ from H2O)
hemoglobin recognizes the pH drop and lets go of its O2
hemoglobing then picks up the extra H+, rasing pH
apneustic center
slows down respiration
Lack of ADH causes what?
diabetes insipidus
seminiferous tubules
produce sperm (in lobules)
leydig cells
make testosterone
interferon
chemicals that warn healthy cells of a virus
intercalated cells
cells involved in pH adjustment (compensatory response cells, move H+ between blood and filtrate)
Trypsinogen/trypsin
enterokinase activates trypsinogen, converts it to trypsin, trypsin then activates the protein enzymes (elastase, chymotrypsin, carboxypeptidase)
asthma
spasm of smooth muscle walls of the terminal bronchioles
nocturia
excessive urination at night (because of posteral change)
is it possible to become pregnant without the big LH surge?
No, without the LH surge the egg can’t rupture the graafian follicle and can’t be released