Spinal Cord Injury 2 Flashcards

1
Q

Spinal Cord Injury pathology

A
  1. Epidemiology
  • 10% unstable
  • 5% with cord damage
  1. Damage to vertebral column
  • Mechanism of injury
    i. Traction
    ii. Direct injury
    iii. Indirect injury (most common)
  • Type:
    i. stable - normal movements will not cause displacement of vertebral component
    ii. unstable - normal movement has significant risk of causing displacement of vertebral component
  • Stability (Denis classification)
    i. anterior column - anterior longlitudinal lig. to half of vertebral body
    ii. middle column - posterior half of vertebral body to posterior longlitudinal lig
    iii. posterior column - pedicles, facets, post bony arch (lamina, spinous process), interspinous and supraspinous ligament
  • any fracture of middle column + at least one other column is unstable
  • Area of injury
    i. upper cervical spine
  • occipital condyle fracture
  • occipito-cervical dislocation
  • c1 ring fracture
  • c2 pars interarticularis fracture
  • c2 odontoid process fracture
    ii. lower cervical spine
  • posterior lig, injury
  • tear-drop fracture
  • fracture dislocation
  • hyperextension injury
  • whiplash injury
    iii. minor thoracolumbar injury
  • transverse process fracture
  • pars interarticularis fracture
    iv. major thoracolumbar injury
  • flexion-compression fracture
  • axial-compression (burst) fracture
  • flexion-distraction (jack-knife/chance/seat-belt) fracture
  • fracture dislocation
  1. Damage to spinal cord
  • ASIA Impairment Scale (AIS)
    i. Complete: No sacral sparing (sensory or motor fx is absent in sacral segments S4,S5). assess by VAC and DAP
    ii. Incomplete: sacral sparing
  • Frankle Scale
    A: absent sensory and motor (0/5)
    B: sensory present (0/5:
    C: sensory present, motor present but not useful (3/5)
    D: sensory present, motor present and useful (4/5)
    E: normal
  • Examples
    i. Complete neuro deficit: SCI only
    ii. Incomplete neuro deficit:
  • SCI
  • Central cord syndrome
  • Anterior cord syndrome
  • Brown sequard syndrome
  • Posterior cord syndrome
  • Conus medullaris syndrome
  • Cauda equina syndrome
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2
Q

Impairment in Spinal Cord Injury

A
  1. Motor
    - paraplegia
    - tetraplegia
  2. Sensory
  3. Neurogenic bowel
    i. UMNL: intact anal tone, BCR+ve
    - Target: Bristol Stool Type 4 (soft)
    - Suppository Bisacodyl
    - Daily syrup lactulose
    ii. LMNL: lax anal tone, BCR-ve
    - Target: Brostol stool Type 3 (hard)
    - KY Jelly, Lignocaine gel evacuate using finger
    - Syrup lactulose PRN
  4. Neurogenic bladder
    i. UMNL:
    - hyperactive bladder
    - sphincter dysnergia
    ii. LMNL:
    - underactive bladder (overflow incontinence)
    - acontractality
  5. Sexual dysfunction
  6. Autonomic dysreflexia
    - occur in SCI at level T6 and above
    - triggered by symphatetic stimulation from below the level of injury: eg bladder, bowel distention, pressure ulcers, ingrown toe nails
    - peripheral vasoconstriction below level of injury cause hypertension, detected by baroreceptors
    - parasympathetic surge in response unable to be transmitted below level of injury
    - CF:
    i. hypertension
    ii. above lesion: bradycardia, vasodilation, flushing, pupillary constriction, nasal stuffiness
    iii. below lesion: pale and cool skin
  7. Orthostatic hypotension
  8. Neurogenic shock
    - loss of symphatetic pathways when injury above T6
    - CF: bradycardia, warm periphery, hypotension (low diastolic)
  9. Spinal shock
    - physiological temporary shutdown
    - primitive anal wink and bulbocavernous reflex return in 48 hours
    - motor and sensory loss may up to 6 weeks
    - CF: bradycardia, hypotension if symphatetic chain affected
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3
Q

Occipital condyle fracture

A
  1. Mechanism of injury
    i. axial loading - Type I
    ii. shear force to basal skull - Type II
    iii. contralateral bending or rotation - Type III
  2. Anderson-Montesan Classification
    I - impacted fracture, comminuted
    II - basal skull fracture extending to occipital condyle
    III - avulsion fracture by alar ligament
    ++risk of occipito-cervical dissociation
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4
Q

Occipito-cervical dislocation

A
  1. Mechanism of injury
    Type I : Anterior displacement of occiput
    Type II - Longlitudinal distraction of occiput from atlas
    Type III - Posterior displacement of occiput
  2. X-ray diagnosis
    - Ratio between distance of BP/AO normally is equal to 1
    B: Basion (mid anterior margin of foramen magnum)
    P: Posterior C1
    A: Anterior C1
    O: Opisthion (mid posterior margin of foramen magnum)

BP/AO > 1 : Type I injury
BP/AO < 1 : Type III injury
Widening space: Type II

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5
Q

C1 Ring Fracture (Jefferson)

A
  1. Mechanism of injury: axial loading
  2. Classification
    Type I : bony injury with intact transverse ligament
    Type II : bony injury with transverse lig. tear
  3. Diagnosis
    i. Hx: Axial loading
    ii. X-Ray:
    Type I:
    - atlantodental interval (ADI) less than 3mm
    - lateral mass displacement less than 7mm (between lateral border of C1, C2)
    Type II:
    - ADI more than 3mm
    - lateral displacement more than 7mm
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6
Q

C2 Pars Interarticularis Fracture (Hangman’s)

A
  1. Mechanism of injury:
    - hyperextension
  2. Levine and Edwards Classification:
    Type I : Less than 3 mm displacement, stable, no angulation
    Type II : More than 3mm displacement, unstable with angulation
    Type III : C2, C3 facet dislocation
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7
Q

C2 Odontoid Process Fracture

A
  1. Mechanism of injury:
    - hyperflexion (II, III)
    - lateral bending, rotation (I)
  2. Anderson and D’Alonzo Classification
    Type I - Avulsion fracture tip of odontoid (alar ligament)
    Type II - Base of odontoid
    Type III - Body of C2
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8
Q

Posterior ligament complex injury of lower cervical

A
  1. Mechanism of injury: hyperflexion
  2. X-Ray
    - posterior translation and angulation
    - loss of 50% anterior column height
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9
Q

Tear Drop Fracture

A
  1. Definition: Fracture of anterior inferior aspect of vertebral body (aka wedge compression fracture)
  2. Mechanism of injury: flexion-compression
  3. Associated injury:
    - posterior spinous ligaments injury
    - vertebral body forward displacement cause spinal cord injury
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10
Q

Burst fracture of lower cervical

A
  1. Definition: Compression >40% of vertebral body height affecting anterior and middle column
  2. Mechanism of injury: axial loading
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11
Q

Facet Dislocation

A
  1. Mechanism of injury: flexion rotation
  2. Type:
    i. Unilateral:
    - less than 50% translation (25% of its width)
    - neurological involvement rare

ii. Bilateral:
- more than 50% translation
- neurological involvement common

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12
Q

Whiplash injury

A
  1. Mechanism of injury:
    - neck hyperextension followed by hyperflexion (accident)
  2. Common associated injury:
    - intervertebral disc tear
    - nerve root impingement
    - muscle tear
    - posterior ligament complex injury
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13
Q

Cervical Injury Imaging

A
  1. AP
    - lateral outline intact
    - spinous process
    - tracheal shadow midline
  2. Open mouth view
    - C1, C2
  3. Lateral view:
    - must include all 7 cervical vertebrae and upper half of T1 (if fail, do swimmer’s view)
    - lordotic curve
    - four parrallel lines: anterior vertebral body, posterior vertebral body, transverse process, spinous process
    - displacement
    - loss of vertebral height
    - soft tissue shadow
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14
Q

Flexion-compression fracture (Compression)

A
  1. Epidemiology
    - most common vertebral fracture
  2. Mechanism of injury
    - high energy: spinal flexion
    - low energy: osteoporosis
  3. Prognosis
    - stable fracture
    - neurological impairment is rare
  4. Diagnosis
    i. Hx
    - hyperflexion thoracolumbar
    - fracture sx
    ii. Pe
    - same as fracture
    iii. X-ray
    - Lateral: loss of anterior vertebral height
  5. Treatment
    i. minimal wedging (<20% loss of vertebral height): bed rest until pain subsides. no support needed
    ii. moderate (<40% loss of vertebral height): bed rest until pain subsides, later support with body cast or thoracolumbar brace. repeat x-ray at 3 months
    iii. severe (>40% loss of vertebral height): posterior ligament disruption expected. surgical intervention
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15
Q

Axial-compression fracture (Burst)

A
  1. Mechanism of injury
    - axial loading
  2. Prognosis
    - usually unstable
  3. Diagnosis
    i. Hx
    - axial loading
    - fracture sx
    ii. Pe
    - same as fracture
    iii. X-ray
    - AP: increase interpedicular distance
    - Lateral: fracture involving anterior, middle column. retropulsion of posterior fragment into spinal canal
    iv. CT
    - If retropulsion is suspected
  4. Treatment
    i. Conservative:
    - if no neurological sx
    - cast or brace for 12 weeks
    ii. Surgery
    - anterior decompression and stabilization
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16
Q

Flexion-distraction fracture (Jack-knife/Chance/Seat-belt)

A
  1. Mechanism of injury
    - flexion against restraining strap causing posterior distraction
  2. Prognosis
    - unstable in flexion
    - neurological damage uncommon
  3. Diagnosis
    i. Hx
    - flexion and distraction component
    - fracture sx
    ii. Pe
    - same as fracture
    iii. X-ray
    - AP: increase vertebral height
    - Lateral: disc space opened up posteriorly. horizontal fracture in pedicles, transverse process, posterior vertebral body (posterior and middle column)
  4. Treatment
    i. Conservative:
    - if no neurological sx
    - cast or brace for 12 weeks
    ii. Surgery
    - posterior spinal fusion
17
Q

Fracture-dislocation

A
  1. Mechanism of injury
    - high energy flexion distraction, flexion-rotation
  2. Prognosis
    - grossly unstable
    - neurological damage common
  3. Diagnosis
    i. Hx
    - flexion and distraction component
    - fracture sx
    - neurological deficits
    ii. Pe
    - same as fracture
    - neurological deficits
    iii. X-ray
    - AP: increase vertebral height
    - Lateral: fracture involving all three column, facet dislocation
19
Q

Imaging Thoracolumbar Injury

A

X-ray

  1. AP view:
    - loss of vertebral height
    - widening of interpedicular distance
  2. Lateral:
    - alignment (4 lines)
    - bone outline
    - disc space defects
    - soft tissue shadow
20
Q

Principles of management

A

Early Management

  1. Spinal immobilization
    i. cervical: in-line immobilization, cervical collar
    ii. thoracolumbar: scoop stretcher, spinal board + padding
  2. Circulation
    i. Hypovolaemic shock:
    - tachycardia
    - cold extremity
    - hypotension
    ii. Neurogenic shock (loss of symphatetic pathways)
    - if injury above T6
    - bradycardia
    - warm periphery
    - hypotension (low diastolic)
    iii. Spinal shock (physiological temporary shutdown)
    - primitive anal wink and bulbocavernous reflex return in 48 hours
    - motor and sensory loss may up to 6 weeks
    - bradycardia, hypotension if symphatetic chain affected
  3. Neurological examination
    - conscious: as usual
    - unconscious patient: spinal injury must be assumed until proven otherwise. suggested by:
    i. hx: fall, mva
    ii. pe: hypotensive, bradycardia, no pain response below clavicle, head injury, flaccid anal sphincter
    iii. x-ray mandatory
  4. Investigation
    i. x-ray: with minimum manipulation
    ii. ct: structural damage of individual vertebrae and displacement of fragment
    iii. mri: indicated for those with neurological deficits. disc, lig. flavum, nerve

Definitive Management

  1. Preserve neurological fx, minimize threat to neurological compression
  2. Stabilize the spine
    - cervical: collars, tongs, halo rings, surgery
    - thoracolumbar: mattress, brace, surgery
  3. Rehabilitation