Spinal Cord Compression Flashcards
The upper motor neurone of the corticospinal tract runs from where?
Motor cortex to anterior grey horn and decussates at medullary level.
What signs are suggestive of an UMN lesion?
- Increased tone.
- Muscle wasting NOT marked.
- No fasciculations.
- Hyper-reflexia.
What is a fasciculation?
Brief spontaneous contraction affecting a small number of muscle fibres, often causing a brief flicker of movement under the skin.
What signs are suggestive of a LMN lesion?
- Decreased tone.
- Muscle wasting.
- Fasciculation.
- Diminished reflexes.
Describe function of spinothalamic tracts.
SENSORY PATHWAY.
- Pain, temperature and crude touch.
- Contralateral.
- Decussates at spinal level.
Describe function of dorsal columns.
SENSORY PATHWAY
- Fine touch, proprioception, vibration.
- Ipsilateral.
- Decussate at medullary level.
Describe broad categories of spinal cord compression.
- Acute or chronic.
- Complete or incomplete.
What may cause acute spinal cord compression?
- Trauma.
- Tumours: haemorrhage or collapse.
- Infection.
- Spontaneous haemorrhage.
What may cause chronic spinal cord compression?
- Degenerative disease e.g. spondylosis.
- Tumours.
- Rheumatoid arthritis.
Describe spinal cord transection?
A complete lesion affecting all motor and sensory modalities.
What is the initial effect of a spinal cord transection?
- Flaccid arreflexic paralysis: “spinal shock”.
UMN signs appear later.
What is Brown-Sequard syndrome?
- Ipsilateral motor level.
- Ipsilateral dorsal column sensory level.
- Contralateral spinothalamic sensory level.
What is central cord syndrome?
Hyperflexion or extension injury to an already stenotic neck.
How does central cord syndrome present?
- Predominantly as distal upper limb weakness.
- “Cape-like” spinothalamic sensory loss.
Lower limb power is preserved.
Dorsal column is preserved.
How does chronic spinal cord compression present?
Predominant UMN signs.