Primary Headaches (IHH, Migraine, Medication overuse, Cluster)☺️ Flashcards

1
Q

IHH Presentation

A

Persistant frontal, retroorbital
Bilateral, dull

Worsened by coughing, physical activity, pressing

Papilloedema => Ongoing progressive visual loss - different to migraine
Enlarged blind spot
If CNVI involved => diplopia

N+photophobia

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2
Q

IHH Pathophysiology

Risk factors

A

High ICP

Most common - obese females in 20-30s
Pregnancy
Drugs
-COCP, CS, tetracycline, VitA, Li

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3
Q

IHH Investigations

Diagnosis

A

Find any underlying causes
CT, MRI
ICP monitoring

IIH diagnosis of exclusion

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4
Q

IHH Management

A

Lifestyle - weight loss

Medication - diuretics, antiepileptic (topiramate)

Surgical -

  • repeated lumbar puncture
  • optic nerve sheath decompression and fenestration
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5
Q

Migraine prevalence

Pathophysiology

A

Young females

Result of abnormal brain activity affecting nerve signals, chemicals, blood vessels => pain

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6
Q

Migraine diagnosis

A

Min 5 attacks lasting 4-72hrs

Min 2 of

  • unilateral
  • pulsation
  • moderate/severe
  • worse with activity

Min 1 of

  • N+V
  • photophobia/phonophobia
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7
Q

Clinical course of migraine

A

Prodrome - 48hrs due to hypothalamic involvement

  • fatigue
  • cravings

Aura - 20min per symptom, last for 1hr - hypothalamic activity spreads to other brain areas

  • marching progression through visual => sensory => motor, aphasia
  • LOSS OF FUNCTION

Headache - 72hrs

  • photophobia, phonophobia
  • N+V

Resolution
-fatigue

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8
Q

Migraine management

  • acute
  • preventative
A

Acute treatment
1st line - paracetamol, ibuprofen at first signs of headache
2nd line - triptan (before its at its worse+ antiemetics (metoclopramide or domperidone)

Preventative

  • topiramate OD (antiepileptic)
  • others - propanolol/amitriptyline

Identify and avoid triggers - migraine diary

  • date, time, duration
  • warning signs
  • symptoms
  • medication
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9
Q

Common migraine triggers

A
Tired, stress
Alcohol
COCP, periods
Lack of food, fluids
Bright light

Cheese, chocolate, red wine, citrus

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10
Q

Medication overuse headache prevalence

Pathophysiology

A

More common in women

Pathophysiology unclear

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11
Q

Medication overuse diagnostic criteria

A

Have preexisting headache disorder

15 days+/month with headache

Regular overuse for 3months+ of acute/symptomatic headache treatment

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12
Q

Medication overuse management

A

Definitive - withdrawal of overused drug

  • simple analgesis, triptans can be stopped abruptly
  • warn that symptoms may initially worsen but should improve over weeks

Keep headache diary

Reassess underlying cause

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13
Q

Cluster headache presentation

Diagnostic criteria

A

Min 5 attacks with the same presentation

15mins-3hrs
Severe unilateral eye pain, same side everytime

Restlessness/agitation
Ipsilateral to pain
-Tears, runny nose. sweating
-Eyelid edema
-miosis, ptosis

Frequency ranging from 1 every other day - 8 a day
-attacks will cluster

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14
Q

Management of cluster headache

  • acute
  • preventative
A

Acute - high flow O2
-2nd line - triptan (SC, IN) - can only use it 2x a day due to increased risk of side effects with prolonged use

Confirmation needed with neuroimaging

Preventative - verapamil whilst they have episodic clusters

  • taper off when clusters end
  • alts - topiramate, lithium
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15
Q

Primary vs secondary headache

A

Primary more likely if

  • headache type known for years
  • gradual onset
  • no neuro deficit

Secondary more likely if

  • new unknown headache
  • sudden onset (as if something fell on your head = ASSUME VASCULAR UNLESS PROVEN OTHERWISE WITH CT, LP
  • electric shock-like - trigeminal?
  • neuro deficit, altered consciousness
  • 50+
  • positional changes, precipitated by something
  • systemically unwell
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16
Q

Thunderclap headache

A

SAH
ICH
Cerebeal venous thrombosis
Arterial dissection - intracranial/extracranial

17
Q

Headache due to CSF pressure change

A

Raised pressure - SOL, bleed, abscess, IIH

  • worse in morning
  • better upright
  • worse with Valsalva

Low pressure - dural tear (idiopathic/trauma)

  • worse as day progresses
  • better recumbent
18
Q

Key questions to differentiate between primary headaches

A

Duration

  • constant - tension?
  • seconds - trigeminal?
  • mins to hours - cluster?

Localisation

  • same unilateral attacks - trigeminal?, cluster?
  • bilateral - tension?
  • unilateral - migraine

Accompanying symptoms
-nausea, photophobia, lacrimation, aura?

Intensity, changes with physical activity?

  • worsens on mv - migraine?
  • improves on mv - tension? cluster?
19
Q

Trigeminoautonomic cephalalgia

A

All types cause autonomic symptoms
-lacrimation
But can differentiate between the 4 by attack duration

Paroxysmal hemicrania, continuous hemicrania - BOTH ARE INDOMETHACIN RESPONSIVE
-if you want to differentiate between migraine and these trigeminal headaches, use indomethacin