Pneumo Flashcards

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1
Q

Nommer 10 causes d’hémoptysie

A

Airway Disease

Bronchitis (acute or chronic)

Bronchiectasis

Neoplasm (primary and metastatic)

Trauma

Foreign body

Parenchymal Disease

Tuberculosis (TB)

Pneumonia, lung abscess

Fungal infection

Neoplasm

Vascular Disease

Pulmonary embolism

Arteriovenous malformation

Aortic aneurysm

Pulmonary hypertension

Vasculitis (Wegener’s granulomatosis, systemic lupus erythematosus [SLE], Goodpasture’s syndrome)

Hematologic Disease

Coagulopathy (cirrhosis or warfarin therapy)

Disseminated intravascular coagulation (DIC)

Platelet dysfunction

Thrombocytopenia

Cardiac Disease

Congenital heart disease (especially in children)

Valvular heart disease

Endocarditis

Miscellaneous

Cocaine

Postprocedural injury

Tracheal-arterial fistula

SLE

Dx graves:

Critical Diagnoses

Disseminated intravascular coagulopathy (DIC)

Tracheo-innominate artery fistula (TIF)

Aortobronchial fistula

Iatrogenic (postprocedural) hemoptysis

Pulmonary embolism

Emergent Diagnoses

Trauma

Bronchiectasis

Pneumonia

Abscess/fungal infection

Oral anticoagulant overdose

Endocarditis

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2
Q

Nommer 10 FR de décès en asthme

A

Asthma History

Previous severe exacerbation (intubation or ICU admission for asthma)

Two or more hospitalizations for asthma in the past year

Three or more ED visits for asthma in the past year

Hospitalization or an ED visit for asthma in the past month

Use of more than two MDI short-acting beta-2 agonist canisters per month

Requiring three or more classes of asthmatic medication

Current use of or recent withdrawal from systemic corticosteroids

Difficulty perceiving asthma symptoms or severity of exacerbations

Social History

Low socioeconomic status or inner-city residence

Serious psychosocial problems

Alcohol or illicit drug use, especially inhaled cocaine and heroin

Comorbidities

Cardiovascular disease

Other chronic lung disease

Chronic psychiatric disease

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3
Q
A
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4
Q

Nommer 10 dxd de l’asthme

A

Cardiac conditions

Valvular heart disease

Congestive heart failure

COPD exacerbation

Pulmonary infection

Pneumonia

Allergic bronchopulmonary aspergillosis

Löffler’s syndrome

Chronic eosinophilic pneumonia

Upper airway obstruction

Laryngeal edema

Laryngeal neoplasm

Foreign body

Vocal cord dysfunction

Endobronchial disease

Neoplasm

Foreign body

Bronchial stenosis

Pulmonary embolus

Cystic fibrosis

Carcinoid tumor

Allergic/anaphylactic reaction

Adverse drug reaction (ACE inhibitors)

Miscellaneous conditions

Churg-Strauss syndrome

GERD

Hyperventilation with panic attack

Noncardiogenic pulmonary edema

Addison’s disease

Invasive worm infection

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5
Q

Indices objectifs d’asthme sévère

A
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6
Q

Nommer 4 mécanismes d’action du Mg en bronchospasme

E2nd

A
  • blocage canaux Ca
  • libération NO
  • effet anti cholinergique
  • stabilisation mastocytes et lymphocytes T

Side effects of magnesium infusion are dose related and include warmth, flushing, sweating, nausea and emesis, muscle weakness and loss of deep tendon reflexes, hypotension, and respiratory depression

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7
Q

Nommer les 4 entités reliés à la sensibilité à l’aspirine

A

Asthme

Polypes nasaux

sinusite éosinophiles

Sensibilité aspirine

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8
Q

Asthme sévère et non contrôlé - définition

A
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9
Q

Asthme périmenstruel

A

Estradiol permet d’inhiber la dégranulation des éosino et effet antileucotriène. La baisse d’estradio dans la phase lutéal est suspectée dans la pathophysio

Progestérone: bronchodilatateur et effet anti-infllammatoire. Diminution progestérone pré-menstruation

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10
Q

Différence a/n physiopatho entre asthme et MPOC

A

In asthma, the cellular response is largely eosinophil-mediated, whereas in COPD, neutrophils, CD8+ lymphocytes, and macrophages predominate in bronchial washings

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11
Q

Nommer 15 causes de détérioration d’un patient MPOC

A

Acute Exacerbations

Infectious

Viral

Rhinovirus, respiratory syncytial virus, coronavirus, influenza virus

Bacterial

Haemophilus influenzae, Streptococcus pneumoniae, Moraxella (Branhamella) catarrhalis, Pseudomonas aeruginosa

Atypical bacteria

Chlamydia pneumoniae, Legionella

Air Pollution

Nitrogen dioxide

Ozone

Particulates, dust

Other Critical Events

Pneumothorax

Pulmonary embolism (PE)

Lobar atelectasis

Congestive heart failure (CHF)

Pneumonia

Pulmonary compression (eg, obesity, ascites, gastric distention, pleural effusion)

Trauma (eg, rib fractures, pulmonary contusion)

Neuromuscular and metabolic disorders

Unrelated treatable chronic pulmonary disease (bronchiectasis, tuberculosis, sarcoidosis)

Noncompliance with prescribed treatment regimens

Iatrogenic

Inadequate therapy

Inappropriate therapy (eg, deleterious drugs)

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12
Q

Nommer indications et CI de la VNI

Indications de VM

A

Respiratory arrest

Worsening level of consciousness despite maximal therapy*

Cardiovascular instability (shock, heart failure)*

NIPPV failure or exclusion criteria (see Table 64.3)

Severe dyspnea with use of accessory muscles and paradoxical abdominal motion*

Severe tachypnea*

Life-threatening hypoxia

Severe acidosis and hypercapnia*

Other complications (metabolic abnormalities, sepsis, pneumonia, pulmonary embolism [PE], barotraumas, massive pleural effusion

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13
Q
A
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14
Q

DxD infectieux de pharyngite

A
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15
Q

Nommer 10 causes de PTX secondaires

A

Airway Disease

Chronic obstructive pulmonary disease

Asthma

Cystic fibrosis

Infections

Necrotizing bacterial pneumonia, lung abscess

Pneumocystis jiroveci pneumonia

Tuberculosis

Interstitial Lung Disease

Sarcoidosis

Idiopathic pulmonary fibrosis

Lymphangiomyomatosis

Tuberous sclerosis

Pneumoconioses

Neoplasms

Primary lung cancers

Pulmonary or pleural metastases

Miscellaneous

Connective tissue diseases

Pulmonary infarction

Endometriosis, catamenial pneumothorax

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16
Q

Comment distinguer un PTX petit, moyen et grand?

A

However, precise quantification is often inaccurate and, in general, it is more reasonable simply to characterize the pneumothorax as small, moderate, large, or total. British Thoracic Society guidelines define size based on measurement of the interpleural distance at the level of the hilum: small, less than 1 cm; moderate, 1 to 2 cm; and large, more than 2 cm. The American College of Chest Physicians measures from the apex to the cupola—small is less than 3 cm, and large is more than 3 cm

17
Q

Nommer les 4 causes les plus fréquentes d’épanchement pleural en ordre

A

The most common cause of pleural effusions in the West countries is congestive heart failure, followed by malignancy, bacterial pneumonia, and PE

18
Q

Nommer 20 causes d’épanchement pleural

A

Transudates

Congestive heart failure

Cirrhosis with ascites

Nephrotic syndrome

Hypoalbuminemia

Myxedema

Peritoneal dialysis

Glomerulonephritis

Superior vena cava obstruction

Pulmonary embolism

Exudates

Infections

Bacterial pneumonia

Bronchiectasis

Lung abscess

Tuberculosis

Viral illness

Neoplasms

Primary lung cancer

Mesothelioma

Pulmonary or pleural metastases

Lymphoma

Connective Tissue Disease

Rheumatoid arthritis

Systemic lupus erythematosus

Abdominal or Gastrointestinal Disorders

Pancreatitis

Subphrenic abscess

Esophageal rupture

Abdominal surgery

Miscellaneous Conditions

Pulmonary infarction

Uremia

Drug reactions

Postpartum

Chylothorax

19
Q

Nommer critères de Light et pH habituels…

A

A pleural fluid pH less than 7.3 is associated with parapneumonic effusions, malignancies, rheumatoid effusions, tuberculosis, and systemic acidosis. A pH less than 7.0 strongly suggests empyema or esophageal rupture and is generally taken to be an indication for tube thoracostomy.

Pleural fluid is considered an exudate if one or more of the following conditions are met:

1.

Pleural fluid protein level/serum protein level exceeds 0.5.

2.

Pleural fluid lactate dehydrogenase (LDH) level/serum LDH level exceeds 0.6.

3.

Pleural fluid LDH level exceeds two-thirds of the upper limit of normal for the serum LDH level.

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23
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