Autres Flashcards

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1
Q

Décrire la matrice d’Hadon

A
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2
Q

Décrire la formule de la force G dans un accident de voiture

A
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3
Q

Décrire les 2 types de fibres qui transmettent la douleur

A

A-δ and C fibers fibers are responsible for the transmission of pain. A-δ fibers transmit sharp, initial pain; C fibers, in contrast, transmit dull, aching, or burning pain

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4
Q

Décrire les réactions à la douleur

A

Increased Sympathetic Tone

Vasoconstriction producing increased peripheral resistance

Increased cardiac output from increased stroke volume and heart rate

Increased blood pressure

Increased metabolic rate and oxygen consumption

Decreased gastric tone and gastric emptying (may progress to ileus)

Decreased urinary tract tone (may lead to urinary retention)

Endocrine Responses

Decreased insulin production

Increased cortisol levels

Increased antidiuretic hormone levels

Increased growth hormone levels

Increased renin, angiotensin II, aldosterone levels

Increased glucagon levels

Increased catecholamine levels

Respiratory Responses

Hyperventilation

Cortical Responses

Anxiety and fear

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5
Q

Nommer 5 récepteurs d’opiacés

A
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6
Q

Nommer les différents sites de traitement de la douleur et des agents associés

A
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7
Q

Nommer des comportements spécifiques et un peu moins spécifiques associés à la dépendance

A
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8
Q

Nommer 6 techniques pour réduire la douleur liée aux anesthésiques locaux

A

Buffering of local anesthetic agents

Counterirritation

Slower rate of injection

Use of topical anesthetics

Warming of solution

Distraction techniques

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9
Q

Décrire les 5 niveaux de sédation

A
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10
Q

Décrire la courbe de dissociation de l’oxygène et ses modificateurs

A
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11
Q

Nomemr 15 causes de méthémobloginémie

A

Hereditary

Hemoglobin M

NADH methemoglobin reductase deficiency (homozygote and heterozygote)

G6PD deficiency

Acquired

Medications

Amyl nitrite

Antineoplastics (eg, cyclophosphamide, ifosfamide, flutamide)

Dapsone

Local anesthetics (eg, benzocaine, lidocaine, prilocaine)

Nitroglycerin

Nitroprusside

Phenacetin

Phenazopyridine (Pyridium)

Quinones (eg, chloroquine, primaquine)

Sulfonamides (eg, sulfanilamide, sulfathiazide, sulfapyridine, sulfamethoxazole)

Chemical Agents

Aniline dye derivatives (eg, shoe dyes, marking inks)

Butyl nitrite

Chlorobenzene

Fire (heat-induced denaturation)

Food adulterated with nitrites

Food high in nitrates

Isobutyl nitrite

Naphthalene (mothballs)

Nitrophenol

Nitrous gases (seen in arc welders)

Paraquat

Silver nitrate

Trinitrotoluene

Well water (nitrates)

Pediatric Cases

Reduced NADH methemoglobin reductase activity in infants (<4 mo)

Seen in association with low birth weight, prematurity, dehydration, acidosis, diarrhea, and hyperchloremia

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12
Q

Nommer les principales causes de cyanose centrale

A

Central cyanosis

A.Decreased arterial oxygen saturation

  1. High altitude (>8000 ft)
  2. Impaired pulmonary function
    a. Hypoventilation
    b. Impaired oxygen diffusion
    c. Ventilation-perfusion mismatching
    (1) Pulmonary embolism
    (2) Acute respiratory distress syndrome
    (3) Pulmonary hypertension
    d. Respiratory compromise
    (1) Upper airway obstruction
    (2) Pneumonia
    (3) Diaphragmatic hernia
    (4) Tension pneumothorax
    (5) Polycythemia

B.Anatomic shunts

1Pulmonary arteriovenous fistulae and intrapulmonary shunts

  1. Cerebral, hepatic, peripheral arteriovenous fistulae
  2. Cyanotic congenital heart disease
    a. Endocardial cushion defects
    b. Ventricular septal defects
    c. Coarctation of aorta
    d. Tetralogy of Fallot
    e. Total anomalous pulmonary venous drainage
    f. Hypoplastic left ventricle
    g. Pulmonary vein stenosis
    h. Tricuspid atresia and anomalies
    i. Premature closure of foramen ovale
    j. Dextrocardia
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13
Q

Indications de traitement de méthémoglobinémie

A

MetHb > 30% ou > 15% avec sx (angine, détresse respi, syncope, arythmies…)

Bleu de méthylène 1-2mg/kg

Si pas amélioration avec bleu de méthylène, considérer sulhemoglobinémie

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14
Q

Nommer 4 grandes classes de complications en greffe

A

Transplant organ complications can generally be classified into one of four categories: anatomy, rejection, infection, and drug toxicity.

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15
Q

nommer les 3 types de rejet en greffe

A

Rejection typically occurs in three phases: hyperacute, acute, and chronic.

Hyperacute rejection occurs with preformed antibodies against major histocompatibility complex or ABO blood type antigens. Typically occurs in the immediate perioperative period.

Acute rejection occurs over days to weeks after transplantation. The patient presents with constitutional symptoms and signs of transplant organ insufficiency.

Chronic rejection, now referred to as chronic graft dysfunction, has a time course of months to years and results in the gradual failure of the transplanted organ over time.

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16
Q

Nommer les infections post greffe selon la durée x greffe

A
17
Q

Nommer 8 médcaments qui interagissent avec la cyclosporine/tacro

A
18
Q

Nommer 2 effets secondaires de chacun des Rx anti rejet

A
19
Q

Nommer 5 étiologies fréquentes de fièvre au retour de voyage

A

Malaria

●Dengue fever

●Mononucleosis (due to Epstein-Barr virus or cytomegalovirus)

●Rickettsial infection

●Typhoid or paratyphoid fever (enteric fever)

20
Q

PEC glaucome aigue angle fermé

A
21
Q

PEC perforation oculaire

A
22
Q

indications de TDM avant PL

A

Altered mentation

●Focal neurologic signs

●Papilledema

●Seizure within the previous week

●Impaired cellular immunity

23
Q
A