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Medicine II - OSCE > Peripheral Neuropathy > Flashcards

Peripheral Neuropathy Flashcards

1
Q
A
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2
Q

Classification of Peripheral Neuropathy

A
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3
Q

Causes of Mononeurpathy & Mononeuropathy Multiplex

A
  • Traumatic
  • Infective
  • Vascular
  • Metabolic
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4
Q

Causes of Mononeurpathy & Mononeuropathy Multiplex

  • Traumatic
A
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5
Q

Causes of Mononeurpathy & Mononeuropathy Multiplex

  • Infective
A
  1. Leprosy
  2. Herpes Zoster
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6
Q

Causes of Mononeurpathy & Mononeuropathy Multiplex

  • Vascular
A

Polyarteritis Nodosa

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7
Q

Causes of Mononeurpathy & Mononeuropathy Multiplex

  • Metabolic
A

DM

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8
Q

Causes of Polyneuropathy

A
  • Heredofamilial
  • Acquired
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9
Q

Causes of Polyneuropathy

  • Heredo-Familial
A
  • Peroneal muscle atrophy.
  • Hypertrophic interstitial polyneuropathy.
  • Refsum disease.
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10
Q

Causes of Polyneuropathy

  • Acquired
A
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11
Q

Acquired Causes of Polyneuropathy

  • Infective
A
  • Viral: mumps, measles.
  • Bacterial: Typhus, Typhoid, Tetanus.
  • Mycobacterial: Leprosy.
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12
Q

Acquired Causes of Polyneuropathy

  • Toxic
A
  • Inorganic: lead, copper, arsenic, antimony, gold. (Heavy metals).
  • Organic: alcohol, insecticides.
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13
Q

Acquired Causes of Polyneuropathy

  • Metabolic & Endocrine
A

Diabetes mellitus, uremia, amyloidosis, acromegaly, myxedema.

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14
Q

Acquired Causes of Polyneuropathy

  • Nutritional
A

Pellagra, Beri-Beri, subacute combined degeneration.

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15
Q

Acquired Causes of Polyneuropathy

  • Iatrogenic
A

Isoniazid, sulphonamides, phenytoin, vincristine.

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16
Q

Acquired Causes of Polyneuropathy

  • Autoimmune
A

Guillain Barre Syndrome, collagen vascular disorders.

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17
Q

Acquired Causes of Polyneuropathy

  • Paraneoplastic
A

Bronchogenic carcinoma, lymphoma.

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18
Q

Pathology of Axonal Neuropathy

A
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19
Q

Pathology of Demyelinating Neuropathy

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20
Q

CP of Polyneuropathy

  • General
A
  • Bilateral
  • Symmetrical
  • Distal more than Proximal
  • Lower Limbs earlier than Upper Limbs.
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21
Q

CP of Polyneuropathy

  • Sensory
22
Q

CP of Polyneuropathy

  • Motor
23
Q

CP of Polyneuropathy

  • Autonomic
24
Q

Investigations for Polyneuropathy

25
Q

Investigations for Polyneuropathy

  • NCS
A
  • Reduced nerve conduction velocity
26
Q

Investigations for Polyneuropathy

  • Nerve Bx
A
  • usually the Sural Nerve
27
Q

Investigations for Polyneuropathy

  • Detect the cause
A
  • Blood sugar
  • Tests for collagen vascular disorders
  • S. creatinine
28
Q

Types of Diabetic Neuropathy

A
  • Diabetic Sensorimotor Polyneurtopathy
  • Diabetic Autonomic Neuropathy
  • Diabetic Proximal Neuropathy (Diabetic Amyotrophy)
  • Diabetic Mononeuropathy
  • Diabetic Truncal Neuropathy
29
Q

Diabetic Sensorimotor Polyneuropathy is common in

30
Q

CP of Diabetic Sensorimotor Polyneuropathy

31
Q

Pathogenesis of Diabetic Sensorimotor Polyneuropathy

A
  • Hyperglycemia will cause sorbitol accumulation → decreased myoinositol uptake by nerve Fibers —-> impaired impulse conduction.
32
Q

Another Name of Diabetic Proximal Polyneuropathy

A

Diabetic Amyotrophy

33
Q

Diabetic Proximal Polyneuropathy is common in ……

A

Type II DM

34
Q

CP of Diabetic Proximal Polyneuropathy

35
Q

CP of Diabetic Mononeuropathy

36
Q

CP of Diabetic Truncal Neuropathy

A
  • Recurrent attacks of truncal pain with sensory deficit in the distribution of a single thoracic root.
  • Spontaneous recovery occurs in few months..
37
Q

TTT of Diabetic Neuropathy

38
Q

Another Name of Guillian-Barre Syndrome

A

Acute Post Infective Polyradiculoneuropathy

39
Q

Etiology of Guillian-Barre Syndrome

40
Q

CP of Guillian-Barre Syndrome

41
Q

CP of Guillian-Barre Syndrome

  • Motor
42
Q

CP of Guillian-Barre Syndrome

  • Sensory
A

Glove and stocking hypoesthesia

43
Q

Prognosis of Guillian-Barre Syndrome

A
  • Recovery in 3-6 months.
  • May be with mild residue in 40% of cases.
  • Survival for 8 weeks carries a good prognosis.
44
Q

Investigations for GBS

45
Q

TTT of GBS

46
Q

TTT of GBS

  • NGT
A

feeding for bulbar palsy.

47
Q

TTT of GBS

  • Assissted Ventilation
48
Q

TTT of GBS

  • SC Heparin
A

to guard against deep venous thrombosis (DVT).

49
Q

TTT of GBS

  • Plasmapheresis
A

must be done in the first 2 weeks

50
Q

TTT of GBS

  • IVIG
A

0.4 g/Kg / day for 5 successive days.

Medicine II - OSCE (32 decks)

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