Parasites of Domestic Production - Cattle (11) Flashcards

1
Q

What are trichostrongylid nematode parasites of cattle, sheep, goats and camelids?

A

small, gut-dwelling nematodes
no extensive tissue migration

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2
Q

What is the life cycle of trichostrongylids of cattle, sheep, goats and camelids?

A

simple, direct life cycle

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3
Q

What is arrested development, aka hypobiosis?

A

an important adaptation to survival and maintenance of parasite populations

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4
Q

What is the most economically significant parasite for cattle?

A

ostertagia spp. (type I and II)

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5
Q

What is the most economically significant parasite for sheep, goats, and camelids?

A

haemonchus contortus - fatal anemia

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6
Q

What is arrested development?

A

recently ingested pasture larvae enter the gut tissue, stop developing, and remain in juvenile state for several months
resume development when environmental conditions are favorable for parasite reproduction
adaptive strategy for survival of parasite populations

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7
Q

What is the life cycle biology of trichostrongylid parasites?

A

adult worms live in the gut and reproduce
eggs passed in feces
free, living larvae develop to infective stage on pasture

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8
Q

For trichostrongylids, animals become infected while _____

A

grazing

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9
Q

The pre-patent period for trichostrongylids is ____ days

A

about 21 days

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10
Q

How do you diagnose trichostrongylids nematodes?

A

periodic assessment of herd health (as a production unit)

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11
Q

Eggs of trichostrongylids for all species are morphologically [indistinguishable/distinguishable]

A

indistinguishable

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12
Q

What are the diagnostic methods for trichostrongylids?

A

fecal flotation
McMasters Quantitative (Fecal Egg Count Test)

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13
Q

What is the appearance of trichostrongylids like on a fecal examination?

A

thin-shelled, modulated eggs

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14
Q

What is the brown stomach worm?

A

ostertagia spp.

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15
Q

For ostertagia spp., adult worms are parasitic in the ______

A

abomasum

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16
Q

What are characteristics of ostertagia spp.? (Sex differentiation, size)

A

size: small
females larger
males with copulatory bursa - morphology of spicules are diagnostic

17
Q

Ostertagia spp. has a [direct/indirect] life cycle with _____ in the gastric pits

A

direct
arrested development

18
Q

Type [1/2] ostertagia spp. has tissue damage to abomasum

A

2

19
Q

What is Type I ostertagia spp. disease?

A

many worms acquired over short period of time
favorable environmental conditions
usually young animas (1st season on pasture)

20
Q

What is Type II ostertagia spp. disease?

A

occurs months after initial infection
older animals (2nd season on pasture)

21
Q

In Type 1 disease ostertagia spp., young cattle acquire substantial worm burdens grazing pasture between _____ and ______

A

October
March

22
Q

For Type I ostertagia spp., ingested infective larvae begin to arrest in tissues ______-______

A

April - September

23
Q

For type 1 ostertagia spp., larvae mature in the _____ of the stomach. Damage occurs when larvae leave here to become adult worms and reproduce. The process is continuous and _______

A

gastric glands
cumulative

24
Q

Type I ostertagiosis is characterized by what?

A

profuse watery diarrhea (often bright green)
bottle jaw from tissue leaking and impaired digestive function
loss of appetite, failure to Gian weight, loss of body condition
young animals most severely infected

25
Q

Type II ostertagia spp. infected larvae are acquired during ______

A

late spring months

26
Q

In Type 2 ostertagia spp., larvae enter the ______ and are [active/dormant]

A

gastric glands
remain dormant

27
Q

How does damage occur in Type 2 ostertagia spp.?

A

when larvae leave the glands “en masse” during late summer-fall months

28
Q

What is Type 2 ostertagia spp. clinical disease?

A

accumulation of large numbers of parasites over longer period of time when environment is hostile to pasture larvae survival
clinical signs similar to type 1 disease - this most severe
fecal exams likely negative

29
Q

What is Type 2 ostertagiosis characterized by?

A

brown, watery diarrhea
loss of appetite and loss of body conditoin
rumen pH approaches neutral
negative fecal exams
older animals

30
Q

Type 2 ostertagiosis has a [good/poor] response to treatment.

A

poor

31
Q

T/F: Type 2 ostertagiosis diagnosis is based on clinical signs and seasonal onset of disease from emergence of larvae

A

TRUE

32
Q

What are the other trichostrongylid parasites of cattle?

A

trichostrongylus sp.
cooperia - calves
nematodirus spp. - calves in late spring

33
Q

What is strategic de-worming?

A

removal of parasites from the host by exploiting their seasonal biology
- targeting inhibited larval stages - differential selectivity against inhibited larvae

34
Q

What is selective de-worming?

A

removal of parasites from “selected hosts” within a managed population
allowing a portion of the total parasite population to survive and reproduce in the absence of drug selection
host selection based on risk-mapping

35
Q

T/F: Strategic and selective de-worming is mutually exclusive

A

FALSE - is not mutually exclusive

36
Q

What is the prevention and control of ostertagiosis?

A

goal is to reduce pasture contamination and hazardous re-infection
map disease risk in herd