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Blood Banking > Other Blood Grou > Flashcards

Other Blood Grou Flashcards

1
Q

The popular ag in kell grouo are

A

k ag and K ag

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2
Q

kel 1

A

K Kell

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3
Q

Kell 3

A

Kpa penny

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4
Q

Kell 6

A

Jsa sutter

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5
Q

Kell 4

A

Kpb rautenberg

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6
Q

Kell 2

A

k cellano

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7
Q

Kell 7

A

Jsb matthews

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8
Q

This is immunogenic and exposure to K +?red cell resull the prod of anti K ab

A

K ag

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9
Q

k aag has frquency of 99.9% thats why hat is rarely encountered

A

anti k

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10
Q

Precursor substance of the Kell Ag

A

Kx

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11
Q

Present on wbc and rbc most individual

A

Kx

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12
Q

If Kx is lacking from red cell the cells have an abnormal shape which is

A

Acanthocytes

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13
Q

Acanthocytes in shape and reduced in vibo survival what ohenotype

A

Mcleod

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14
Q

The absence of Kx from leukocytes has been described with dse

A

CGD

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15
Q

Most common gene complex

A

Kell blood group

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16
Q

Reduced expression of kell ag

A

Mcleod syndrome

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17
Q

Mcleod syndrome ass with

A

HA and CGD

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18
Q

This is usually igg and require AHG

A

Kell ab

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19
Q

Rare reaction in saline

A

Kell ab

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20
Q

Implicated in Htr and Hdn

A

Kell ab

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21
Q

Is a very common ab

A

Anti K

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22
Q

Many ag in this system and some are alleles

A

MNSs

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23
Q

MNSs ass with

A

Gpa and Gpb

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24
Q

Four gene complexes

A

MS Ms NS Ns

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25
Q

Other alleles of MNSs

A

Mg, Mc, Mk, Mr, Mz, Mv, Na, T1m, Sj, S2

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26
Q

M and N ag amino acid composition

A

Glycophorin A

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27
Q

M ag amino acid

A

Serotonin
Sertonin
theronin
theronin + glycine

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28
Q

N ag amino acid

A

Leucine
Serotonin
threonine
Threonine + glycose

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29
Q

S, s ag amino acid

A

Glycophorin B

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30
Q

S contains what AA

A

Methionine

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31
Q

s contain what AA

A

Threonine

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32
Q

Acts as anti zN

A

Vicea gaminea

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33
Q

Ab of MNS are

A

Polar opposites

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34
Q

MNSs demonstrate

A

Dosage effect

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35
Q

Codominant allele in paternity

A

M and N

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36
Q

MNSs are discovered by

A

Levine and Lansteiner

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37
Q

MNSs differe from ABO ag in 3 ways

A

Ag found in red cells and tissue and not in secretion
Anti M and N resukt of transfusion and pregnancy
Anti M and N rarely occur naturally

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38
Q

Anti M and anti N ab are

A

Natural and Igm

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39
Q

Anti S, s, and U

A

Exposure igg

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40
Q

Transfusion reaction due to

A

anti S and anti s

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41
Q

Hdn due to

A

Anti s and anti U

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42
Q

Medico legal cases

A

Paternity

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43
Q

Paternity testing in Father NN ANTI M AND ANTI N

A

Negative and 3+

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44
Q

Paternity testing in Father MM ANTI M AND ANTI N

A

4+ and negative

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45
Q

Paternity testing in baby MN ANTI M AND ANTI N

A

3+ 3+

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46
Q

Usually Igm and room temp and some at AHG

A

Anti S ab

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47
Q

Destroyed by enzyme

A

Anti S ab

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48
Q

Rare Htr and Hdn

A

Anti S ab

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49
Q

Usually Igg and Ahg in MNSs

A

Anti s and anti U

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50
Q

Not detroyed by enzyme

A

Anti s and anti U

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51
Q

Htr and Hdn

A

Anti s and Anti U

52
Q

Found as aarm autoab and dies not react well with rh null cells

A

Anti U

53
Q

Common Ag that detected in MNSs

A

Anti Mg

54
Q

This is more commonly found in oriental particularly filipino

A

JK (a-b)

55
Q

Variable expression with time

A

Kidd

56
Q

Exposure requiring warm igg

A

Kidd

57
Q

Usually require iGg and Ahg

A

Angi Jka and Jkb ab

58
Q

Bind complement

A

Anti jka and jkb

59
Q

Enhanced by enzymes

A

Anti jka and angi jkb

60
Q

Seldom potent and deteriorate rapidly

A

Anti jka and anti jkb

61
Q

Classic delayed htr

A

Anti jka and anti jkb

62
Q

Found in some Jka-b- individual

A

Anti Jk3

63
Q

Reacts with Jka and Jkb

A

Anti Jk3

64
Q

Importance blood group that demostrate kultiple method for inheritance of null cell type

A

Lutheran

65
Q

Lub how many percent

A

99

66
Q

Lua how many percent

A

8

67
Q

Lutheran dominate inhibitor gene

A

InLu

68
Q

Recessive lack of Lu gene

A

Lulu

69
Q

Sex inhibitor gene

A

Lu a-b- inheritance

70
Q

Not common,reacts in saline but can be igg and require Ahg give agglutination, unclear about htr and hdn

A

Anti Lua

71
Q

Reacts with all but lua-b- of the recessive type

A

Anti Luab or anti Lu3

72
Q

Rare most igg and require ahg probably htr and hdn

A

Anti Lub

73
Q

Other ab reacts with rare lulu phenotypes found in

A

Lua-b-

74
Q

Found in chipoawah native americans and japanese and chknese

A

Dia

75
Q

Uncommon ab ahg reaction and important in htr and hdn

A

Diego Dia

76
Q

Marker in mongolian ancestry

A

Diego Dia

77
Q

Common autoab very high frequency ab

A

Anti Wrb

78
Q

Nine ag all normal individuals are either Rg + ir Ch+

A

Chido rogers

79
Q

Use plasma inhibition

A

Htla

80
Q

Deterrminants on c4 molecules and linked to HLA

A

Chido rodgers

81
Q

Sex linked inheritance

A

Xg

82
Q

Located in short arm of x chromosome

A

Xg

83
Q

Destroyed by enzyme treatment

A

Xg

84
Q

Found in glycophorin c and d which ass witg rbc membrane band 4.1

A

Gerbich

85
Q

Ab usually igg which require ahg and clinically significant

A

Gerbich

86
Q

Ab are rare

A

Schianna

87
Q

Schianna under 0.3 percent

A

Schianna 2

88
Q

Sch under 100%

A

Schianna 1 and 3

89
Q

Found in aquaporin -1

A

Colton

90
Q

Null phenotype has been found and ass with gen abnormality and anemia

A

Colton

91
Q

Coa -99.7

A

Colton1

92
Q

Coa 100

A

Coa3

93
Q

Coa 10.7

A

Coa 2

94
Q

Found in decay acceperating factor daf or cd55

A

Cromer

95
Q

Total absence of cromer ag is observed in

A

Pnh 3 rbc

96
Q

For,erly cartwright

A

Yt

97
Q

Yt 0.2%

A

Ytb

98
Q

Yt 99.8

A

Yta

99
Q

Add ag added holly gregory and joseph

A

Dombrock

100
Q

Uncommon ab htr and hdn

A

Dombrock

101
Q

Found in complement receptor 1 cr1 or cd35

A

Knops

102
Q

Decreased expression in cases of sle and cold agglutinin dse

A

Knops

103
Q

Found on semaphorin cdw108

A

John milton hagen blood group system

104
Q

Ansene of JMH found in

A

Pnh 3

105
Q

Important in HLa typing

A

Bennett goodspeed ag

106
Q

Bga

A

Hla b7

107
Q

Bgb

A

Hla b17

108
Q

Bgc

A

Hla 28

109
Q

Destroyed by treating rbc ag with chloroquine or edta glycine hcl

A

Bg ab

110
Q

Unaffected ezyme class

A

Colton
Diego
Kell

111
Q

Decreased enzyme class

A

MNS system
Duffy
Lutheran

112
Q

Enhanced

A 
Abo h system
Lewis
I system
P
Rh
Kidd
113
Q

Naturally occuring

A

Abo, lewis, P1, Mn, Lua

114
Q

Clin significant

A

Abo rh kell duffy kidd

115
Q

Warm ab

A

Rh kell duffy, kidd

116
Q

Cold ab

A

M, N, P1

117
Q

Usually react in AHg

A

Kell duffy kidd

118
Q

Can react in any ohase of testing

A

Lewis

119
Q

Detection enhanced by enzyme treatment

A

Rh lewis kidd p1

120
Q

Nkt detected with enzyme treatment of test cell

A

M, N, Duffy

121
Q

Enhanced by acidification

A

M

122
Q

Show dosage

A

Rh, MNS, Duddy, Kidd

123
Q

Bind complement

A

I, kidd, lewis,

124
Q

Cause in vitro hemolysis

A

Abo, lewis, kidd some P1

125
Q

Labile in vivo and invitro

A

Kidd

126
Q

Common cause of anamenstic response

A

Kidd

127
Q

Produced from the precursor substance Kx that is coded by gene XK xon the X chromosome

A

Kell blood grouo

Blood Banking (16 decks)

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