head injuries Flashcards
open head injury
the integrity of the skull is compromised by either a penetrating object or blunt force trauma.
closed head injury
occurs from blunt trauma
head injuries classified as
mild, moderate, or severe, depending upon Glasgow Coma Scale ratings and the length of time the client was unconscious.
concussion
occurs after head trauma that result in a change in the client’s neurological function and usually resolves within 72 hr.
post concussion syndorme
includes persistence of cognitive and physical manifestations for an unknown period of time.
contusion
brain is bruised and the client has a period of unconsciousness associated with stupor and or confusion.
diffuse axonal injury
widespread injury to the brain that results in coma and is seen in severe head trauma.
head injuries clinical findings
- Presence of alcohol or illicit drugs at time of injury.
- Amnesia (loss of memory) before or after the injury.
- Loss of consciousness: Length of time the client is unconscious is significant.
- CSF leakage
cfk leakage from nose
can indicate a basilar skull fracture (“halo” sign: yellow stain surrounded by blood on a paper towel; fluid tests positive for glucose).
intracranial hemorrhage
- can occur in the epidural, subdural, or intracerebral space.
- It is a collection of blood following head trauma.
- There can be a delay of weeks to months in presenting manifestations for a subacute or chronic subdural hematoma.
skull fractures: what should the nurse be alert for
- can occur following forceful head injury.
- The nurse should be alert for drainage from the ears or eyes (cerebral spinal fluid [CSF]).
- A cervical spine injury must be ruled out prior to removing any devices used to stabilize the cervical spine.
- Occur frequently with head trauma.
- Location determines clinical manifestations.
epidural hematoma
- Bleeding between the dura and skull.
- Neurologic Emergency
- Associated with a fracture crossing a major artery.
- Needs Rapid Surgery bc artery bleeding
Subdural Hematoma
- Bleeding between dura and arachnoid layer of meninges.
- Venous bleed, slow to develop. (can take up to 2 days)
basilar skull fractures
- Linear fx at base of skull
- Battles Sign (Raccoon eyes)
- Tear in the dura and CSF leakage from ear and nose.
- Test drainage for glucose
- DO NOT INTRODUCE NGTUBE IF BASILAR FX IS SUSPECTED!!!
- Major complication is infection
- Meningitis
Decerebrate posturing
- Arms are stiffly extended, adducted, and hyperpronated. There is also hyperextension of the legs with plantar flexion of the feet.
Decorticate posturing (flexor)
internal rotation and adduction of the arms with flexion of the elbows, wrists, and fingers.
family care
- Support of the family following head injury is of great importance.
- The Brain Injury Association of America provides families and clients with information needed to cope with this potentially devastating injury.
- The family can face difficult decisions following head injury.
- If brain death has occurred, the family needs support when deciding whether to donate organ
care of a head injury
Respiratory status (the priority assessment):
- The brain is dependent upon oxygen to maintain function and has little reserve available if oxygen is deprived.
- Untreated hypoxia leads to brain injury or death if the brain has been denied adequate oxygenation for 3 to 5 min.
- Changes in level of consciousness, using the Glasgow Coma Scale (GCS), provide the earliest indication of neurological deterioration.
cranial nerve function
Eye blink response, gag reflex, tongue and shoulder movement
assess eyes for
pupils for size, equality, and reaction to light: Pupils that are equal, round, and react to light and accommodation (PERRLA) are a normal finding.
factors that influnce intracranial pressure
- Arterial pressure
- Intraabdominal and intrathoracic pressure (venous congestion)
- Posture
- Temperature
- Blood gases (CO2 levels)
normal icp
5 to 15 mm Hg
Elevated if >20 mm Hg sustained
normal compensation
- Changes in CSF volume
- Changes in intracranial blood volume
- Changes in tissue brain volume
Brain’s ability to compensate is limited If
volume increase continues, ICP rises → decompensation