GI- pediatrics Flashcards

1
Q

Characteristics of GI Malformation: pyloric stenosis

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-3 months
  • Emesis: nonbilious, projectile.
  • Findings: Male» female. Olive shaped mass in the epigastrium, Low CL/ Low K metabolic alkalosis
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2
Q

Characteristics of GI Malformation: Intestinal atresia

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-1 week
  • Emesis: Bilious
  • Findings: “Double-bubble” sign, Down syndrome
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3
Q

Characteristics of GI Malformation: TE fistula

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-2 week
  • Emesis: food regurgitation
  • Findings: respiratory compromise with feeding, aspiration pneumonia, inability to pass NGT into stomach, gastric distention
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4
Q

Most common variant of TE fistula

A

esophageal atresia with a fistula from the bronchus to the distal esophagus. Gastric distention as each breach transmits air to the GI tract.

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5
Q

Characteristics of GI Malformation: Hirschsprung Disease

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-1yr
  • Emesis: feculent
  • Findings: abdominal distentions, obstipation, no nerve ganglia seen on rectal biopsy, males»females
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6
Q

Characteristics of GI Malformation: anal atresia

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-1 wk
  • Emesis: Late, feculent
  • Findings: Detected on initial exa, in nursery, Males> females
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7
Q

Characteristics of GI Malformation: Choanal atresia

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-1wk
  • Emesis: none
  • Findings: cyanosis with feeding that improves with crying. inability to pass a NGT through nose.
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8
Q

Characteristics of Peds GI condition: Intussusception

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 3mo-2yr
  • Emesis: Bilious
  • Findings: Currant-jelly stools (blood &mucus), palpable sausage-shaped mass,
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9
Q

Tx Intussesception

A

pneumatic or hydrostatic enema guided by fluoro or U/S (diagnostic and therapeutic)

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10
Q

Characteristics of Peds GI condition: Necrotizing enterocolitis

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-2 months
  • Emesis: Bilious
  • Findings: premature baby, fever, rectal bleeding, air in bowel wall.
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11
Q

Tx Nectrotizing enterocolitis

A

NPO, orogastric tube, IV fluids, abx

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12
Q

Characteristics of Peds GI condition: Meconium ileus

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-1wk
  • Emesis: feculent, late
  • Findings: cystic fibrosis manifestation (as is rectal prolapse)
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13
Q

Characteristics of Peds GI condition: Midgut Volvulus

  • Presenting age
  • vomit description
  • other findings
A
  • Age: 0-2yr
  • Emesis: Bilious
  • Findings: sudden onset pain, distention, rectal bleeding, peritonitis, “bird beak” abd X-ray
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14
Q

S&S of Meckel Diverticulum

A
-Age: 0-2yr
Rule of 2s, 
Painless GI bleed
Anemia 
Remnant of Vitelline (omphalomesenteric) duct
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15
Q

Characteristics of Peds GI condition: Strangulated hernia

  • Presenting age
  • vomit description
  • other findings
A
  • Age: any age
  • Emesis: bilious
  • Findings: bowel loops in inguinal canal
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16
Q

What is the Rule of 2s for Meckle Diverticulum

A
2% of the population
2 inches long
2 feet from the Ileocolic junction
presents at 2yo
2x more likely in boys
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17
Q

Complications of Meckles

A

Present with abd pain:

Intussusception
Obstruction
Volvulus
Diverticulitis (mimics appendicitis)

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18
Q

What GI malformation primarily causes respiratory problems?

Signs and symptoms

A

Diaphargmatic Hernia

Males
Left sided (common) herniation through a diaphragmatic defect 
Pulmonary Hypoplasia
Respiratory distress
Bowel sounds in chest
Bowel loops in thorax on Xray
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19
Q

Differentiate between omphalocele and gastroschisis

A

Omphalocele: midline, hernia sac with multiple organs, absent umbilical ring, other anomalies present

Gastroschisis: right of midline, small bowel exposed, no true hernia sac, umbilical ring present, other anomalies rare.

20
Q

What is Henoch-Schonlein purpura?

A

Vasculitis that may present with GI bleeding and abdominal pain.

Hx of URI, rash on legs and buttocks, swelling hands and feet, hematuria/ proteinuria.

21
Q

T or F: children may develop inflammatory bowel disease and irritable bowel sydnrome

A

TRUE
Diarrhea, fever, bloody stool, poor growth are concerning for OBD.

GI complaints may be psychosomatic; watch for separation anxiety, depression or child abuse.

22
Q

What is the first step in evaluation neonatal jaundice?

A

to determine whether jaundice is physiologic or pathologic.

Measure total, direct, and indirect bilirubin.

23
Q

Why is jaundice of concern in a neonate?

A

Main concern is Bilirubin-Induced Neurologic Dysfunction (BIND).

Due to unconjugated bilirubin deposit in basal ganglia.

24
Q

Symptoms of BIND and sequelae

A
Poor feeding
Seizures
Flaccidity
Opisthotonos
Apnea

Kernicterus is chronic and permanent sequelae of BIND.

25
What are the causes of physiologic jaundice?
Caused by normal neonatal changes in bilirubin metabolism: Increased bilirubin production, decreased clearance and increased enterohepatic circulation. Mostly low risk unconjugated (indirect) bilirubin because of incomplete liver function. More prevalent in premature infants.
26
How is severe hyperbilirubinemia recognized?
- Jaundice that is recognized in the first 24hrs of life. - T Bili higher than the hour specific 95% - Rate of rising T Bili >0.2mg/dl per hour - Direct bili more then 20% of T bili
27
What are causes of neonatal Jaundice?
``` Breast-feeding jaundice Breast Milk jaundice Illness Hemolysis Metabolic disorders Biliary Atresia Medications ```
28
What is Breast-feeding jaundice?
An exaggerates physiologic jaundice to insufficient milk intake with leads to fluid and weightless and an inadequate number of bowel movement to remove bilirubin from body. 1st week of life
29
What is great milk jaundice?
3-5 days of life Results from direct effect of breast mild as human milk promotes an increase in intestinal absorption of bilirubin. Can continue breastfeeding as long as Bili levels stay safe
30
What is the cause of hemolysis jaundice in a new born?
Rh incompatibility or congenital red cell disease. look for anemia, peripheral smear abnormalities, higher unconjugated bilirubin
31
What metabolic disorders can cause neonatal jaundice?
Crigler-Najjar syndrome Roter syndome Dublin-Johnson syndrome
32
Presentation and treatment of biliary atresia
Full term infant clay or gray collide stool high conjugated bilirubin Tx: surgery
33
What medication can cause neonatal jaundice?
Sulfa drugs. avoid in neonates, they displace bilirubin from albumin and may precipitate kernicterus
34
How is sever hyperbilirubinemia treated?
Phototherapy. Converts unconjugated bilicurib to a water soluble form that can be excreted.
35
What should you do is an infant is born to a mother with active hepatitis B?
They should receive the first immunization and hep B globulin at birth
36
What should you watch for in children after diarrhea?
Hemolytic uremic syndrome (HUS). Thrombocytopenia Hemolytic anemia (schistocytes and fragment cells) acute renal failure.
37
S&S infant physiologic gastroesophageal reflux (GER)
``` In the first 6 months of life (peak 4mo) Spit-up/ vomiting partially digested milk Feed eagerly Normal weight gain Normal examination ↑ risk with tobacco smoke exposure ```
38
Differentiate between pathological GER and physiological
Pathologic GER: irritable, poor feeding, weight loss Physiologic: "happy spitters"
39
Management of Pathologic GER
Thicken formula feeds Switch to extensively hydrolyzed formula Remove cow's milk and soy protein from mothers diet. Resistant: acid-suppression therapy
40
Management of physiologic GER
``` Preventative measures (upright positioning) Reassurance ```
41
Nutritional supplement for breastfeeding
Breast milk has inadequate vit D with poor bioavailability. Cholecalciferol supplementation for ALL breastfed infants
42
Management of children with dehydration
Determin severity. Oral or IV resuscitation to prevent endometrial organ damage. Isotonic crystalloid bolus.
43
Moderate dehydration
``` ↓ skin turgor Dry MM Tachycardia Irritability Delayed Capillary refill ↓ Urine output ```
44
Severe dehydration
``` Cool, clammy skin Delayed cap refill (>3sec) Cracked lips Dry MM Sunken eyes/ Sunken fontanelle Minimal/ no urine output ```
45
Diagnosis of MEckel diverticulum
Technetium-99m pertechneate scan. AKA: Nuclear Scintigraphy Increased uptake by gastric mucosa, shows atopic tissue.
46
Features that suggest a pathologic lead point for intussusception
Recurrent episodes Atypical locations (SB into SB) Atypical age Persistent rectal bleedings despite reduction MCC: Meckels --> anemia/ occult blood