Gastro - General Surgery Flashcards

1
Q

What does PC stand for? What is it?

A

→ presenting complaint
→ pain assessment using SOCRATES
→ associated symptoms

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2
Q

What does SOCRATES stand for?

A
S = site
O = onset
C = character
R = radiation
A = association
T = time course
E = exacerbating / relieving factors
S = severity
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3
Q

What is the general approach when a patient comes in with acute abdomen issues?

A
→ PC
→ past medical history
→ drug history
→ social history
→ range of investigations
→ manage patient
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4
Q

What are the range of investigations that can be done for a patient?

A

→ Bloods: VBG, FBC, CRP, U&Es (renal profile), LFTs +amylase
→ Urinalysis + Urine MC&S
→ Imaging: Erect CXR, AXR, CTAP, CT angiogram, USS
→ Endoscopy

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5
Q

What is involved in the general approach of management?

A

→ ABCDE management
→ Conservative management
→ Surgical management

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6
Q

What is ABCDE management?

A
A = airways
B = breathing
C = circulation
D = disability
E = exposure
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7
Q

What are some the differentials for pain in the RUQ?

A
→ Bilary Colic
→ Cholecystitis/Cholangitis
→ Duodenal Ulcer
→ Liver abscess
→ Portal vein thrombosis
→ Acute hepatitis
→ Nephrolithiasis
→ RLL pneumonia
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8
Q

What are some of the differentials of pain in the epigastrium?

A
→ Acute gastritis/GORD
→ Gastroparesis
→ Peptic ulcer disease/perforation
→ Acute pancreatitis
→ Mesenteric ischaemia
→ AAA (Abdominal Aortic Aneurysm) 
→ Aortic dissection
→ Myocardial infarction
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9
Q

What are some of the differentials for pain in the LUQ?

A
→ Peptic ulcer
→ Acute pancreatitis
→ Splenic abscess
→ Splenic infarction
→ Nephrolithiasis
→ LLL Pneumonia
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10
Q

What are some of the differentials for pain in the RLQ?

A
→ Acute Appendicitis
→ Colitis
→ IBD
→ Infectious colitis
→ Ureteric stone/Pyelonephritis
→ PID/Ovarian torsion
→ Ectopic pregnancy
→ Malignancy
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11
Q

What are some of the differentials for pain in the suprapubic / central region?

A
→ Early appendicitis
→ Mesenteric ischaemia
→ Bowel obstruction
→ Bowel perforation
→ Constipation
→ Gastroenteritis
→ UTI/Urinary retention
→ PID (Pelvic inflammatory disease) (female reproductory organ infection)
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12
Q

What are some of the differentials for pain in the LLQ?

A
→ Diverticulitis
→ Colitis
→ IBD (Inflammatory Bowel Disease)
→ Infectious colitis
→ Ureteric stone/Pyelonephritis
→ PID/Ovarian torsion
→ Ectopic pregnancy
→ Malignancy
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13
Q

What is bowel ischaemia?

A

→ when the blood flow through the major arteries that supply blood to your intestines slows or stops
→ tissue in intestines begins to die

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14
Q

What is the clinical presentation of bowel ischaemia?

A

→ Sudden onset crampy abdominal pain
→ Severity of pain depends on the length and thickness of colon affected
→ Bloody, loose stool (currant jelly stools)
→ Fever, signs of septic shock

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15
Q

What are the risk factors for bowel ischaemia?

A
→ Age >65 yr
→ Cardiac arrythmias (mainly AF), atherosclerosis
→ Hypercoagulation/thrombophilia
→ Vasculitis
→ Sickle cell disease
→ Profound shock causing hypotension
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16
Q

What are the 2 different types of bowel ischaemia?

A

→ acute mesenteric ischaemia

→ ischaemic colitis

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17
Q

Where does acute mesenteric ischaemia occur?

A

small bowel

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18
Q

What usually causes acute mesenteric ischaemia?

A

usually occlusive, due to thromboemboli

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19
Q

What is the onset of acute mesenteric ischaemia?

A

sudden onset, but presentation + severity can vary

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20
Q

What is the abdominal pain caused by acute mesmeric ischaemia like?

A

abdo pain out of proportion of clinical signs

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21
Q

Where does ischaemic colitis occur?

A

large bowel

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22
Q

What usually causes ischaemic colitis?

A

usually due to non-occlusive low flow states or atherosclerosis

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23
Q

What is the onset of ischaemic colitis?

A

more mild + gradual (80-85% of cases)

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24
Q

What is the abdominal pain caused by ischaemic colitis like?

A

moderate pain + tenderness

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25
Q

What bloods should be done to investigate bowel ischaemia? What are the markers to look for?

A

→ FBC : look for neutrophilic leukocytosis

→ VBG : look for lactic acidosis (form of metabolic acidosis, associated with late stage mesenteric ischaemia)

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26
Q

What imaging should be done to investigate bowel ischaemia?

A

→ CTAP / CT angiogram

→ endoscopy

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27
Q

Why use CTAP / CT angiogram to investigate bowel ischaemia?

A

to look for + detect:
→ vascular stenosis
→ disrupted flow
→ pneumatosis intestinalis (transmural ischaemia / infarction)
→ ischaemic colitis (looks like a thumbprint)

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28
Q

Why do an endoscopy for bowel ischaemia?

A

for mild + moderate cases of ischaemic colitis (oedema, cyanosis, ulceration of mucosa)

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29
Q

What kind of bowel ischaemia can be managed conservatively?

A

mild to moderate cases of ischaemic colitis

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30
Q

How is mild to moderate ischameic colitis managed conservatively?

A

→ IV fluid resuscitation
→ Bowel rest
→ Broad-spectrum ABx - colonic ischaemia can result in bacterial translocation & sepsis
→ NG tube for decompression - in concurrent ileus (no peristalsis)
→ Anticoagulation
→ Treat/manage underlying cause
→ Serial abdominal examination and repeat imaging

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31
Q

What are the indications that bowel ischaemia should be managed surgically?

A
→ Small bowel ischaemia
→ Signs of peritonitis orsepsis
→ Haemodynamic instability
→ Massive bleeding
→ Fulminant colitis with toxic megacolon
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32
Q

What are the two ways in which bowel ischaemia is surgically managed?

A

→ exploratory laparotomy

→ endovascular revascularisation

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33
Q

What is the process of exploratory laparotomy?

A

→ resection of necrotic bowel

→ +/- open surgical embolectomy or mesenteric arterial bypass

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34
Q

What is the process of endovascular revascularisation?

A

→ done before going into theatre
→ more common in choleric ischaemic patients
→ Balloon angioplasty / thrombectomy
→ In patients without signs of ischaemia

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35
Q

What is the clinical presentation of acute appendicitis?

A
→ Initially periumbilical pain that migrates to RLQ (within 24hours)
→ Anorexia
→ nausea +/- vomiting
→ low grade fever
→ change in bowel habit
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36
Q

What are some important clinical signs of acute appendicitis?

A
→ McBurney’s point
→ Blumberg sign
→ Rovsing sign
→ Psoas sign
→ Obturator sign
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37
Q

What is McBurney’s point or sign?

A

→ tenderness in the RLQ (lateral 1/3 of a hypothetical line drawn from the right ASIS to the umbilicus)
→ where the appendix is

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38
Q

What is the Blumberg sign?

A

rebound tenderness especially in the RIF

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39
Q

What is the Rovsing sign?

A

RLQ pain elicited on deep palpation of the LLQ

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40
Q

What is the Psoas sign?

A

RLQ pain elicited on flexion of right hip against resistance

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41
Q

What is the Obturator sign?

A

RLQ pain on passive internal rotation of the hip with hip & knee flexion

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42
Q

What bloods should be done to investigate acute appendicitis?

A

→ FBC : look for neutrophilic leukocytosis and increased CRP
→ Urinalysis : possible mild pyuria/haematuria
→ Electrolyte imbalances in profound vomiting

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43
Q

What imaging should be done for acute appendicitis?

A

→ CT : gold standard in adults esp. if age > 50
→ USS: children /pregnancy /breastfeeding
→ MRI : in pregnancy if USS inconclusive

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44
Q

What can be done for acute appendicitis if imaging is inconclusive but patient still in persistent pain?

A

→ diagnostic laparoscopy

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45
Q

*What is the Alvarado scoring criteria?

A
→ RLQ tenderness
→ fever
→ rebound tenderness
→ pain migration
→ anorexia
→ nausea +/- vomiting
→ WCC > 10
→ Nuetrophilia
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46
Q

What is the conservative management for acute appendicitis?

A

→ IV Fluids
→ Analgesia
→ IV or PO Antibiotics
→ In abscess, phlegmon or sealed perforation, Resuscitation + IV Antibiotics +/- percutaneous drainage

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47
Q

What are the indications for conservative management of acute appendicitis?

A

→ After negative imaging in selected patients with clinically uncomplicated appendicitis
→ In delayed presentation with abscess/phlegmon formation (use CT-guided drainagefirst before considering interval appendectomy)

48
Q

What is the rate of recurrence for acute appendicitis after conservative management?

A

12-24%

49
Q

What are the 2 ways in which acute appendicitis can be surgically managed?

A

→ laparoscopic appendectomy

→ open appendectomy

50
Q

What are the advantages of laparoscopic over open appendectomy?

A
→ Less pain
→ Lower incidence of surgical site infection
→ ↓ed length of hospital stay
→ Earlier return to work
→ Overall costs
→ Better quality of life scores
51
Q

What is the process of laparoscopic appendectomy?

A

→ Trocar placement (usually 3)
→ Exploration of RIF & identification of appendix
→ Elevation of appendix + division of mesoappendix (containing artery)
→ Based secured with endoloops and appendix is divided
→ Retrieval of appendix with a plastic retrieval bag
→ Careful inspection of the rest of the pelvic organs/intestines
→ Pelvic irrigation (wash out) + Haemostasis
→ Removal of trocars + wound closure

52
Q

What is a bowel obstruction?

A

restriction of normal passage of intestinal contents

53
Q

What are the 2 main groups of bowel obstructions?

A

→ paralytic (adynamic ileus)

→ mechanical

54
Q

How are mechanical obstructions classified?

A

→ Speed of onset: acute, chronic, acute-on-chronic
→ Site: high or low
roughly synonymous with small or large bowel obstruction
→ Nature: simple vs strangulating

55
Q

*What is a simple obstruction vs. a strangulating obstruction?

A

→ Simple: bowel is occluded without damage to blood supply.
→ Strangulating: blood supply of involved segment of intestine is cut off (e.g. in strangulated hernia, volvulus, intussusception (in picture))

56
Q

What are the different groups of causes of mechanical bowel obstruction?

A

→ Causes in the lumen
→ Causes in the wall
→ Causes outside the wall

57
Q

What can cause mechanical bowel obstruction in the lumen?

A

→ faecal impaction

→ gallstone ‘ileus’

58
Q

What can cause mechanical bowel obstruction in the wall?

A

→ Crohn’s disease
→ tumours
→ diverticulitis of colon

59
Q

What can cause mechanical obstruction outside of the wall?

A

→ Strangulated hernia (external or internal)
→ Volvulus
→ Obstruction due to adhesions or bands

60
Q

What are some common causes of small bowel obstruction and their prevalences?

A
→ Adhesions (60%)
Hx of previous abdominal surgery
→ Neoplasia (20%)
Primary, Metastatic, Extraintestinal
→ Incarcerated hernia (10%)
External (abdominal wall), Internal (mesenteric defect)
→ Crohn's Disease (5%)
Acute (oedema), Chronic (strictures)
→ Other (5%)
Intussusception, intraluminal (foreign body, bezoar)
61
Q

What are some common large bowel obstructions?

A
→ Colorectal carcinoma
→ Volvulus
Sigmoid, Caecal
→ Diverticulitis
Inflammation, strictures
→ Faecal impaction
→ Hirschsprung disease
commonly found in infants/children
62
Q

What is the clinical presentation of vomiting in SB obstruction?

A

early onset
large amounts
bilious

63
Q

What is the clinical presentation of constipation in SB obstruction?

A

late sign

64
Q

What is the clinical presentation of distention in small bowel obstruction?

A

less significant

65
Q

What kind of pain does LB obstruction present with?

A

colicky or constant

66
Q

What is the clinical presentation of vomiting in LB obstruction?

A

late onset
initially billious
progresses to faecal vomiting

67
Q

What is the clinical presentation of constipation in LB obstruction?

A

early sign

68
Q

What is the clinical presentation of distention in LB obstruction?

A

early sign + significant

69
Q

What are some other signs of bowel obstruction?

A

→ Dehydration
→ Increased high pitched tinkling bowel sounds (early sign), or absent bowel sounds (late sign)
→ Diffuse abdominal tenderness

70
Q

What is important when diagnosing bowel obstruction?

A

→ diagnosed by presence of symptoms
→ examination should always include search for hernias + abdominal scars, including laparoscopic portholes
→ assess whether any hernias are simple or strangulating

71
Q

What is a simple bowel obstruction compared to a strangulating one?

A

→ simple = bowel is still viable

→ strangulating = bowel is no longer viable

72
Q

What features suggest a strangulating hernia?

A

→ Change in character of pain from colicky to continuous
→ Tachycardia
→ Pyrexia (fever)
→ Peritonism (localised inflammation of bowel)
→ Bowel sounds absent or reduced
→ Leucocytosis
→ increased CRP

73
Q

What is the mortality rates of strangulating obstruction with peritonitis?

A

up to 15%

74
Q

*What are some common hernial sites?

A
→ epigastric
→ umbilical
→ incisional
→ inguinal
→ femoral
75
Q

*Why is the neck of a hernia sac important?

A

→ very narrow hernia sac increases chances of it being a strangulated hernia
→ larger neck = higher chances of bowel being able to slip in and out more easily

76
Q

*What is a strangulated hernia?

A

→ bowel can’t escape the hernia
→ blood supply to + from bowel is constricted
→ bowel becomes ischaemic + is no longer viable

77
Q

*What is a Richter’s hernia?

A

hernia that doesn’t cause bowel obstruction

78
Q

What are the bloods that can be done to investigate bowel obstruction?

A

→ WCC/CRP usually normal (if raised suspicion of strangulation/perforation)
→ U&E: electrolyte imbalance
→ VBG if vomiting: HypoCl-,HypoK+ metabolic alkalosis
→ VBG if strangulation: Metabolic Acidosis (lactate)

79
Q

What imaging can be done to investigate bowel obstruction?

A

→ erect CXR
→ erect + supine Abdo XR
→ CT abdo/pelvis

80
Q

What does an SBO CXR look like?

A

Dilated small bowel loops >3cm proximal to the obstruction (central)

81
Q

What does an LBO CXR look like?

A

Dilated large bowel >6cm (if caecum >9cm) predominantly peripheral

82
Q

What makes a SBO from an LBO on X-rays?

A

→ Ladder pattern of dilated loops & their central position

→ Striations that pass completely across the width of the distended loop produced by the circular mucosal folds.

83
Q

*What makes an LBO AXR distinct from a SBO AXR?

A

→ Distended large bowel tends to lie peripherally

→ Show haustrations of taenia coli - do not extend across whole width of the bowel.

84
Q

What can CT scan detect in case of bowel obstruction?

A

→ Can localise site of obstruction
→ Detect obstructing lesions & colonic tumours
→ May diagnose unusual hernias (e.g. obturator hernias).

85
Q
  • What would a CT scan show in SBO?
A

Collapsed & dilated loops of small bowel due to transition point in the pelvis

86
Q
  • What would a CT scan show in LBO?
A

Sigmoid stricture with

proximal dilation

87
Q

Can bowel obstruction patients be conservatively managed?

A

Yes, in patients with no signs of ischaemia/no signs of clinical deterioration

88
Q

What is some supportive management for bowel obstruction?

A

→ NBM
→ IV peripheral access with large bore cannula with IV Fluid resuscitation
→ IV analgesia, IV antiemetics, correction of electrolyte imbalances
→ NG tube for decompression, urinary catheter for monitoring output
→ Introduce gradual food intake if abdominal pain and distention improve

89
Q

What is some conservative management for bowel obstruction?

A

→ Faecal impaction: stool evacuation (manual, enemas, endoscopic)
→ Sigmoid volvulus: rigid sigmoidoscopic decompression (tube that straightens out colon)
→ SBO: oral gastrograffin (highly osmolar iodinated contrast agent)can be used to resolve adhesionalsmall bowel obstruction

90
Q

What are the indications for surgical management in bowel obstruction?

A

→ Haemodynamic instability or signs of sepsis
→ Complete bowel obstruction with signs of ischaemia
→ Closed loop obstruction
→ Persistent bowel obstruction >2 days despite conservative management

91
Q

What surgeries can be done for bowel obstruction?

A

→ Exploratory Laparotomy / Laparoscopy
→ Restoration of intestinal transit (depending on intra-operational findings)
→ Bowel resection with primary anastomosis or temporary/permanent stoma formation

92
Q

What symptoms does GI perforation clinically present with?

A

→ Sudden onset severe abdominal pain associated with distention
→ Diffuse abdominal guarding, rigidity, rebound tenderness
→ Pain aggravated by movement
→ Nausea, vomiting, absolute constipation
→ Fever, Tachycardia, Tachypnoea, Hypotension
→ Decreased or absent bowel sounds

93
Q

What are 4 common + distinct GI perforations?

A

→ perforated peptic ulcer
→ perforated diverticulum
→ perforated appendix
→ perforated malignancy

94
Q

What is distinct in clinical perforation of a perforated peptic ulcer?

A

→ Sudden epigastric or diffuse pain
→ Referred shoulder pain
→ Hx of NSAIDs, steroids, recurrent epigastric pain

95
Q

What is distinct in clinical perforation of a perforated diverticulum?

A

→ LLQ pain

→ Constipation

96
Q

What is distinct in clinical perforation of a perforated appendix?

A

→ Migratory pain
→ Anorexia
→ Gradual worsening RLQ pain

97
Q

What is distinct in clinical perforation of a perforated malignancy?

A

→ Change in bowel habit
→ Weight loss
→ Anorexia
→ PR Bleeding

98
Q

What bloods can be done to investigate GI perforation? What is being looked for?

A

→ FBC: neutrophilic leukocytosis
→ Possible elevation of Urea, Creatinine
→ VBG: Lactic acidosis

99
Q

*What imaging can be done to investigate GI perforations?

A

→ Erect CXR: subdiaphragmatic free air (pneumoperitoneum)
→ CT abdo/pelvis: Pneumo-peritoneum, free GI content,localised mesenteric fat stranding, can exclude common differential diagnoses such as pancreatitis

100
Q

What are the differential diagnosis for GI perforation?

A

→ Acute cholecystitis
→ Appendicitis
→ Myocardial infarction
→ Acute pancreatitis

101
Q

What is the supportive management given to patient on presentation with GI perforation?

A
→ NBM & NG tube 
→ IV peripheral access with large bore cannula -IV Fluid resuscitation
→ Broad spectrum Abx
→ IV PPI
→ Parenteral analgesia & antiemetics
→ Urinary catheter
102
Q

Can GI perforations be conservatively managed?

A

only in localised peritonitis without signs of sepsis - very rare

103
Q

What is conservative management for GI perforations?

A

→ IR - guided drainage of intra-abdominal collection

→ Serial abdominal examination & abdominal imaging for assessment

104
Q

What is the surgical management for generalised peritonitis +/- signs of sepsis?

A

→ Exploratory laparotomy/laparascopy
→ Primary closure of perforation with or withoutomental patch (most common in perforated pepticulcer)
→ Resection of theperforated segment of the bowelwith primary anastomosis or temporary stoma
→ Obtainintra-abdominal fluid for MC&S, peritoneal lavage
→ If perforated appendix: Lap or open appendicectomy
→ If malignancy: intraoperative biopsies if possible

105
Q

What are the symptoms of biliary colic?

A

→ PostprandialRUQ pain with radiation to the shoulder.

→ Nausea

106
Q

What are the investigations for biliary colic, and what results indicate biliary colic?

A

→ Normal blood results

→ USS: cholelithiasis (presence of gall stones)

107
Q

What is the management for biliary colic?

A

→ Analgesia
→ Antiemetics (drugs to help with nausea + vomiting)
→ Spasmolytics (drugs to relieve spasming of the smooth muscle)
→ Follow up for elective cholecystectomy

108
Q

What are the symptoms of acute cholecystitis?

A

→ Acute, severe RUQ pain
→ Fever
→ Murphy’s sign

109
Q

What are the investigations for acute cholecystitis, and what results indicate acute cholecystitis?

A

→ Elevated WCC/CRP

→ USS: thickened gallbladder wall

110
Q

What is the management for acute cholecystitis?

A

→ Fluids, ABx, Analgesia, Blood cultures

→ Early (<72 hours) or elective cholecystectomy (4-6 weeks)

111
Q

What are the symptoms of acute cholangitis?

A

Charcot’s triad:
→ jaundice,
→ RUQ pain,
→ fever

112
Q

What are the investigations for acute cholangitis, and what results indicate acute cholangitis?

A
  • Elevated LFTs, WCC, CRP, Blood MCS (blood culture) (+ve)

* USS: bilary dilatation

113
Q

What is the management for acute cholangitis?

A
  • Fluids, IV Abx, Analgesia

* ERCP (endoscopic retrograde cholangio pancreatography) (within 72hrs) for clearance of bile duct or stenting

114
Q

What are the symptoms of acute pancreatitis?

A

→ Severe epigastric pain radiating to the back
→ Nausea +/- vomiting
→ Hx of gallstones or alcohol use

115
Q

What are the investigations for acute pancreatitis, and what results indicate acute pancreatitis?

A

→ Raised amylase/lipase
→ High WCC/Low Ca2+
→ CT and US to assess for complications/cause

116
Q

What is the management for acute pancreatitis?

A

→ Admission score (Glasgow-Imrie)
→ Aggressive fluid resuscitation, O2
→ Analgesia, Antiemetics
→ ICU/HDU involvement