FON CH 11and CH 21 Flashcards
**Types of pain
MILD/SEVERE
CHRONIC/ACUTE. Chronic- usually the result of prior tissue damage. Pain lasting more than 6 months. Can be constant or intermittent increase/decrease
Acute-intense, usually of short duration therefore usually tx w/opioids and other analgesics
REFERRED PAIN-can radiate, felt at a site other than the injured or diseased part of the body
Acute pain response
Creates a response that starts in the sympathetic nervous system (SNS) flooding the body with epinephrine/fight or flight response. Associated w/ anxiety can be a warning of actual or potent tissue damage
**Chronic pain
Does not serve as warning of tissue damage in process. May be linked to arthritis, back injuries. Accidents, or neurological condition, cause may be unidentifiable **6months
**High risk of self injury, cause of depression, suicide
Chronic pain
**Gate control theory
Pain impulses can be regulated or even blocked by gating mechanisms along the central nervous center (CNS).
Location of the gate is in the dorsal horn of spinal cord. Pain and other sensations of the skin and muscles travel this route
**Endorphins
Morphine like substance produced by body(naturally occurring)
Activated by stress and pain
Attach to opioid receptors sites in brain
Prevent release of neurotransmitters
Inhibit transmission of pain impulses
Analgesia
People who feel less pain than others have a higher endorphin level
TENS , acupuncture and placebos may cause the relief of endorphins
Subjective data
Characteristics and descriptions of the pain where?, anywhere else?
Pain is what pt. says it is
**Objective data
Is measurable
Tachycardia
Increased rate and depth of respirations
Diaphorisi
Increased systolic / diastolic BP
Pallor
Facial expressions
Restlessness
Muscle tension
Pain assessment
Subjective and objective data (crucial)
Subjective:pain is what the pt. says it is
Objective data: is measurable
H I L D A
H- how does your pain feel?
Intensity
Location
Durations
Aggravating and alleviating factors
P Q R S T Pain assessment used by ATI
The Joint Commision Requiements for controlling pain
The management of pain is appropriate for all patients, not just dying patients
Routine and PRN analgesics are to be administered as ordered
Education of practitioners: assessment and management
Fifth vital sign
Pain is monitored on regular basis
Appropriate pain management brings about
Quicker recovery‘s
Shorter hospital stays
Fewer readmissions
Improved quality of life
Unrelieved pain
Harmful physical and psychological effects
**Noninvasive pain relief techniques
Removal of pain source
Distraction
Relaxation
Guided imagery
Hipnosis
Biofeedback
**Invasive approaches to pain
Higher risk than non-invasive strategies
Nerve blocks
Epidural analgesics
Acupuncture
Injections
Oral medications
Medication for pain management
ANALGESIC=Acting to relieve pain
Non opioids
Opioids
Compound analgesics
Non-opioid pain medication
NSAID= non steroidal anti inflammatory drugs used to relieve pain reduce inflammation
Antiprostaglandins= agains inflammation (?)
Ibuprofen= advil, Motrin, nupren (faster)
Naproxen= used for arthritis, swelling inflammation, stiffness joint pain
Aleve is Naproxen (longer lasting)
Rx versions= Celebrex, toradol, daypro
(?3200 mg -4equal doses daily?)
Tylenol= APAP. (metabolized in liver) is NOT a NSAID. is anti-pyrectic. It is acetaminophen treats pain and fever headaches
No more than 4000mg per day
Aspirin=ASA
Risk of bleeding
Do not give to children
Mechanism of action (meds)
Different analgesics relieve pain in different ways
NON OPIOIDS
TYLENOL= seems to inhibit prostaglandins (inflammation)
that may serve as mediators of pain and fever primarily in the central nervous system but they may also block pain impulses peripherally
ASPIRIN= blocks pain impulses in the central nervous system and reduces inflammation in
NSAIDS= TRAMADOL, IBUPROFEN=MOTRIN,ALEVE, TORADOL work in the CNS but their better characterized actions are peripheral ( at site of injury)
Mechanism of action
Opioids
Hopefully to leave pain mainly by action in the CNS Binding to the opioid receptor sites in the brain and spinal cords. When a drug attaches to these sites pain relief occurs
Narccan ( opioid antagonist)
Blocks/reverses the action of all opioids
Adjuvant analgesic=Diverse mechanisms of actions
Drugs meant to relieve one condition but also work on other conditions
Some antidepressants appear to relieve pain by blocking the way up take a serotonin some local anesthetics such as Mexitil in certain anticonvulsants such as Tegretol a sodium channel blocking agents and this part of the mechanism contributes to their ability to relieve certain pain. Neuropathic pain is difficult to treat and some antidepressants and anti-convulsant such as duloxetine which is Cymbalta are used
Antidepressants - Cymbalta
Anticonvulsants- tegretol, Neurontin, Lyrica
Opioids they REMOVE pain sensation (it does not exist)
Open voice being binds to receptors sites in the brain and spinal cord they decrease perception of pain they do not fix the cause of the pain
NAMES of some opioid medications
Morphine Dilaudid, Fentanyl, Percocet= Oxycodone, codeine,