eyes Flashcards

1
Q

What are common causes of eye trauma?

A

Everyday activities (cooking, cleaning, gardening), power tool use, home repair work, sports, work-related injuries, trauma

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2
Q

What are the common signs and symptoms of eye trauma?

A

Pain, abnormal or decreased visual acuity, redness, swelling, drainage, visual foreign body, photophobia

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3
Q

What diagnostic tests are used for eye trauma?

A

Snellen chart, visual acuity tests, ophthalmoscope, CT/MRI

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4
Q

What is a Morgan lens used for?

A

To provide continuous irrigation of an injured eye, especially for chemical or thermal burns

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5
Q

What are some medical treatments for severe eye trauma?

A

Surgical repair for penetrating injury, globe rupture, or globe avulsion

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6
Q

What should be assessed in eye trauma cases?

A

Mechanism of injury, presence of other injuries, and visual acuity

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7
Q

Why is it important to avoid putting pressure on the eye after trauma?

A

To prevent further damage and avoid increasing intraocular pressure (IOP)

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8
Q

What actions should be avoided to prevent increased IOP?

A

Avoid blowing the nose or stooping

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9
Q

What immediate action should be taken for chemical exposure to the eye?

A

Begin eye irrigation immediately

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10
Q

What patient education is essential for eye trauma prevention?

A

Educate about wearing proper eye protection during activities that pose a risk

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11
Q

How should the head of the bed (HOB) be positioned in cases of eye trauma?

A

Elevate the HOB to 45 degrees

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12
Q

What is the pathophysiology of cataracts?

A

Opacity within the lens, possibly in one or both eyes. Most are age-related.

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13
Q

What are the common causes of cataracts?

A

Age-related changes, trauma (blunt or penetrating), smoking, alcohol use, radiation or UV light exposure, steroid use, ocular inflammation, diabetes.

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14
Q

What is a senile cataract?

A

A cataract formed due to water accumulation and changes in lens fiber structure with age.

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15
Q

What are common symptoms of cataracts?

A

Abnormal color perception and glare caused by light scatter from lens opacities, worse at night.

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16
Q

What diagnostic tests are used for cataracts?

A

Snellen chart, decreased visual acuity assessment, ophthalmoscope.

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17
Q

What preoperative medications are commonly used for cataract surgery?

A

Mydriatic drops, cycloplegics, and NSAIDs.

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18
Q

What is the surgical treatment for cataracts?

A

Phacoemulsification: An ultrasound probe dissolves the clouded lens, which is suctioned out and replaced with an artificial lens.

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19
Q

What postoperative medications are used for cataract surgery?

A

Antibiotic and corticosteroid eye drops.

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20
Q

What activities should be avoided postoperatively to prevent increased IOP?

A

Avoid bending, coughing, stooping, and lifting.

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21
Q

What nonsurgical interventions can help manage cataracts?

A

Changing glasses prescription, strong reading glasses or magnifiers, increased lighting, lifestyle adjustments.

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22
Q

What lifestyle adjustments may help with cataracts?

A

Using stronger lighting and magnifiers, and adjusting activities to accommodate vision changes.

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23
Q

What is retinal detachment?

A

A separation of the sensory retina and the underlying pigment epithelium with fluid accumulation between the two layers. It is an emergency.

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24
Q

What are the most common causes of retinal detachment?

A

Retinal breaks, including retinal holes (spontaneous small breaks) and retinal tears (due to aging and vitreous humor shrinkage).

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25
Q

How does rhegmatogenous retinal detachment occur?

A

Fluid enters the subretinal space as the vitreous humor pulls on the retina, lifting it away from the underlying tissue.

26
Q

What are nonrhegmatogenous detachments?

A

Tractional: caused by scar tissue pulling the retina (e.g., from diabetes). Exudative: caused by AMD, tumors, blunt injuries, or inflammation.

27
Q

What are risk factors for retinal detachment?

A

Age, AMD, diabetic retinopathy, eye surgery/trauma, family history, severe myopia, thinning of the peripheral retina.

28
Q

What are the common symptoms of retinal detachment?

A

Dark curtain in the field of vision, flashes of light, a ring in the field of vision, and gradual loss of peripheral or central vision.

29
Q

What diagnostic tests are used for retinal detachment?

A

History/physical exam, visual acuity measurement, ophthalmoscopy, slit lamp microscopy, and ultrasound for hazy or opaque cornea/lens/vitreous.

30
Q

What is laser photocoagulation?

A

A laser treatment that creates small burns around a retinal tear to seal it.

31
Q

What is cryopexy?

A

A procedure using a freezing probe to apply intense cold around the retinal tear.

32
Q

What is scleral buckling?

A

A surgical procedure where a rubber band is placed around the eye to push the retina back against the wall.

33
Q

What is pneumatic retinopexy?

A

An intraocular procedure where a gas bubble is inserted into the vitreous to press the retina into place.

34
Q

What is vitrectomy?

A

The surgical removal of the vitreous humor to treat retinal detachment.

35
Q

What are key nursing interventions for retinal detachment?

A

Bedrest and positioning, eyedrops, pain management, avoiding activities that increase IOP, providing emotional support, fall precautions, and applying an eye patch.

36
Q

What medications are used post-treatment for retinal detachment?

A

Topical antibiotics, corticosteroids, and mydriatics.

37
Q

What is glaucoma?

A

A group of disorders characterized by increased intraocular pressure (IOP) causing optic nerve atrophy and peripheral vision loss.

38
Q

What are the two main types of glaucoma?

A

Primary open-angle glaucoma (POAG) and angle-closure glaucoma (ACG), including acute angle-closure glaucoma (AACG).

39
Q

What causes increased IOP in glaucoma?

A

An imbalance between aqueous humor inflow and outflow, with decreased outflow causing a buildup of pressure.

40
Q

How does primary open-angle glaucoma (POAG) occur?

A

Outflow is decreased in the trabecular meshwork due to clogged drainage channels, leading to optic nerve damage.

41
Q

What causes angle-closure glaucoma (ACG)?

A

Reduction in outflow caused by angle closure, often due to lens bulging forward with aging or pupil dilation in narrow angles.

42
Q

What are symptoms of acute angle-closure glaucoma (AACG)?

A

Sudden, severe eye pain, nausea/vomiting, colored halos around lights, blurred vision, and eye redness.

43
Q

What are the symptoms of chronic POAG?

A

Gradual visual field loss, often unnoticed until peripheral vision is severely compromised, leading to “tunnel vision” if untreated.

44
Q

What diagnostic tests are used for glaucoma?

A

Visual field confrontation, tonometry, ophthalmoscopy, slit lamp microscopy, gonioscopy, visual field perimetry.

45
Q

What is the goal of treatment for chronic glaucoma?

A

To keep IOP low to prevent optic nerve damage and vision loss.

46
Q

What medications are used for chronic glaucoma?

A

Prostaglandin agonists, alpha-adrenergic agonists, beta-adrenergic blockers.

47
Q

What is Argon Laser Trabeculoplasty (ALT)?

A

A procedure that stimulates scarring and contraction of the trabecular meshwork to open outflow channels.

48
Q

How is acute angle-closure glaucoma (AACG) treated?

A

It is an emergency treated by quickly lowering IOP with medications such as carbonic anhydrase inhibitors and IV hyperosmotic agents, followed by surgical intervention.

49
Q

What should patients be taught about glaucoma?

A

Glaucoma risk increases with age; early detection and treatment are critical to prevent visual impairment. Regular eye exams are essential, especially for high-risk individuals.

50
Q

What is age-related macular degeneration (AMD)?

A

A condition that affects the macula, causing central vision loss. It has two types: dry (non-exudative) and wet (exudative).

51
Q

What is dry AMD?

A

A more common and less severe form where macular cells atrophy, leading to slow, painless vision loss.

52
Q

What is wet AMD?

A

A more severe form with rapid onset caused by abnormal blood vessel growth that leaks fluid or bleeds, forming scar tissue.

53
Q

What are the main risk factors for AMD?

A

Retinal aging, family history, obesity, hypertension, being white, smoking.

54
Q

How does dry AMD develop?

A

It begins with the accumulation of yellowish deposits called drusen in the retinal pigment epithelium, leading to macular cell degeneration.

55
Q

How does wet AMD develop?

A

Abnormal blood vessels form in the retinal epithelium due to vascular endothelial growth factor. These vessels leak fluid or bleed, forming scar tissue.

56
Q

What are common symptoms of AMD?

A

Blurred vision, darkened vision, scotomas (blind spots), and metamorphopsia (wavy vision distortion).

57
Q

What diagnostic tests are used for AMD?

A

Visual acuity measurement, ophthalmoscopy (to detect drusen and fundus changes), Amsler grid test, fundus fluorescein angiography, or indocyanine green dye tests.

58
Q

How is wet AMD treated?

A

Medications are injected into the vitreous cavity in 4-6 week intervals. Photodynamic therapy (PDT) with verteporfin IV uses laser light to block abnormal blood vessels.

59
Q

What precautions should be taken after PDT?

A

Avoid direct sun exposure and intense light for 5 days post-treatment.

60
Q

What lifestyle changes can help slow AMD progression?

A

Smoking cessation, use of antioxidant vitamin supplements, and making home adjustments for safety and low vision.

61
Q

What assistive measures may help AMD patients?

A

Low-vision devices, magnifiers, rearranging the home for accessibility, and driving restrictions such as limiting to daytime and lower speeds.