Chronic Heart Failure (CHF) Flashcards

1
Q

Define heart failure

A

a condition where the heart is unable to adequately pump blood throughout the body

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2
Q

Define CHF

A

a condition that occurs subsequent to heart failure leading to increased pressure in the venous and capillary system

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3
Q

What is the result of CHF?

A

congestion of lungs or liver (varies with which side of the heart is affected) with blood or fluid (edema)

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4
Q

What is the result of heart failure?

A

decrease in the delivery of O2 to the body’s tissues

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5
Q

What happens with left-sided failure?

A

congestion of lungs (pulmonary edema, pleural effusion)

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6
Q

What happens with right-sided failure?

A

congestion of liver

fluid leaks into abdomen (ascities)

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7
Q

Is left or right sided failure more common?

A

left

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8
Q

What commonly causes right-sided failure in SAM?

A

HW Dz

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9
Q

Lab work and rads with left sided failure

A
  • labs usually normal
  • change in heart size/shape
  • pulmonary edema (usually ,but not always, concentrated around the heart) and/or pleural effusion
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10
Q

Lab work and rads with right sided failure

A
  • CBC mild to mod anemia if HW Dz

- Chem may show increased Alk Phos (cholestatic hepatic enzyme) and increased ALT (enzyme associated with liver damage)

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11
Q

What is the initiating factor for CHF

A

any cardiac dz that results in decrease of cardiac output or volume of blood ejected by the ventricle

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12
Q

How does the nervous system respond to decreased cardiac output/decreased volume of ejected blood?

A
  1. Increases HR to attempt to increase cardiac output

2. Vasoconstriction attempts to raise systemic blood pressure

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13
Q

What is the event cascade that results from the NS response?

A
  • cardiac output and BP return to normal
  • but heart has to work harder to maintain this compensation
  • heart gets tired
  • cardiac output and systemic BP decrease again
  • body reimplements increasing HR and initiation of vasoconstriction
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14
Q

What happens if the heart can’t compensate anymore?

A

pressure begins to build in the circulatory system prior to the failing portion of the heart

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15
Q

Who is at risk for developing CHF?

A

any patient affected by primary cardiac Dz

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16
Q

Examples of congenital cardiac dz

A
Patent Ductus Arteriosus (PDA)
Subaortic Stenosis (SAS)
Pulmonic Stenosis (PS)
17
Q

Examples of acquired cardiac dz

A

mitral valve endocardosis
mitral valve regurgitation
various cardiomyopathies
HW Dz

18
Q

3 main goals of medical management for CHF

A
  1. relieve edema/pleural effusion by removing/controlling water retention and salt intake
  2. improve ability of heart muscle to pump adequately
  3. reduce worklaod of the heart
19
Q

Meds to relieve edema

A

diuretics

  • Furosemide
  • Thiazide
  • Potassium-sparing diuretics
20
Q

Emergency drugs to improve ability of heart muscle to pump

A

Positive inotropes

  • dobutamine
  • dopamine
21
Q

Meds to reduce the workload of the heart

A

Angiotensin Converting Enzyme (ACE) inhibitors

  • Enalapril
  • Benazepril
22
Q

What is a potential side effect of ACE inhibitors that you need to be careful about when using them in a heart patient?

A

can cause hypotension and worsen the situation if severe heart failure is present

23
Q

Strategy for addressing pleural effusion in cardiac patients

A

diuretic won’t work well for this if at all; will need to do Thoracocentesis to remove fluid from the pleural space

24
Q

Things owners should know about CHF

A
  • CHF is not the primary Dz present but is instead the result of an underlying cardiac Dz
  • current status of their pet
  • outline ultimate goals of therapy (short term = eliminate CS causing decreased QoL; long term = established if P responds well to Tx)
  • understand meds being Rxed and why they’re needed
  • therapy is life-long
  • recheck appointments 3-4x a year
  • many emotional ups and downs