Chapter 2 Flashcards
A deranged cellular growth observable in uterine crevical epithelium is
a. atrophy
b. hyperplasia
c. hypertrophy
d. dysplasia
e. metaplasia
dysplasia
What is the consequence when a cell is forced into glycolysis
a. insufficient glucose production
b. excessive pyretic acid retention
c. increased lactic acid production
d. excessive CO2 production
Increased lactic acid production
what is the probable cause of cellular swelling during the early stages of cell injury?
a. fat inclusion
b. loss of genetic integrity
c. hydrolytic enzyme activation
d. NA+ K+ pumps fail to remove intracellular NA+
NA+ K+ pump fails to remove intracellular NA+
Calcification
a. alters membrane permeability
b. the result of low calcium levels in the blood
c. caused by UV light
d. Caused by hypoparathyroidism
Alters membrane permeability
Cellular swelling?
a. reversible
b. evident early in all types of cellular injury
c. associated with hyperkalemia
d. extracellular movement of fluid
Reversible
Which of the following is reversible?
a. karyolysis
b. fatty infiltration
c. hydropic degeneration
d. glycogen formation
karyolysis
Aging:
a. likely involves autoantibodies
b. does not have a genetic relationship
c. results from damage accumulation
d. decreases hormonal degredation
likely involves autoantibodies
results from damage accumulation
In the theories of aging, cross-linking implies that:
a. the lifespan and number of times the cell can replicate are programmed
b. the number of cells doubling is limited
c. there is oxygen toxicity
d. cell permeability decreases
cell permeability decreases
Caused by TB infection
a. liquefactive nécrosais
b. rigor mortis
c. caseous necrosis
caseous necrosis
Rigidity of muscles after somatic death
a. hyperplasia
b. rigor mortis
c. mechanoporation
Rigor Mortis
increased tissue mass because of increased cell numbers
a. hyperplasia
b. metaplasia
c. hypertrophy
Hyperplasia
results from lysosomal release of hydrolytic enzymes
a. liquefactive nécrosais
b. gangrene
c. casseous necrosis
liquefactive necrosis
replacement of one cell type with another more suitable type
a. metaplasia
b. fatty necrosis
c. lead
metaplasia
Disruption of cell membranes
a. Hypertrophy
b. Dysplasia
c. Mechanoporation
mechanoporation
pancreatic necrosis
a. Fatty Necrosis
b. caseous Necrosis
c. Gangrene
fatty Necrosis
Coagulative and liquefactive necrosis
a. Apoptosis
b. Gangrene
c. Caseous Necrosis
caseous necrosis
tissue death
a. gangrene
b. rigor mortis
gangrene
normal pathologic cellular self destruction
a. apoptosis
b. gangrene
c. caseous necrosis
apoptosis
lipid peroxidation
a. oxygen deprived free radicals
b. lead
c. carbon monoxide
oxygen-deprived free radicals
neurotransmitter interference
a. lead
b. mercury
c. carbon monoxide
lead
asphyxiation
a. lead
b. carbon monoxide
c. free radicals
carbon monoxide
depressed fatty acid oxidation
a. tobbaco
b. ethanol
c. detached ribosomes
ethanol
depressed protein synthesis
a. xenobiotics
b. detached ribosomes
c. creatine kinase
detached ribosomes
during reperfusion with oxygenation _________ is produced, which creates superoxides, hydrogen peroxide, and free radicals
a. carbon dioxide
b. pyruvic acid
c. xanthine oxidase
xanthine oxidase
specific enzymes, such as ________. are released into ECF, during muscular injury
a. creatine kinase
b. lactic acid
c. myoglobin
creatine kinase (CK)
