Chapter 2 Flashcards

1
Q

A deranged cellular growth observable in uterine crevical epithelium is

a. atrophy
b. hyperplasia
c. hypertrophy
d. dysplasia
e. metaplasia

A

dysplasia

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2
Q

What is the consequence when a cell is forced into glycolysis

a. insufficient glucose production
b. excessive pyretic acid retention
c. increased lactic acid production
d. excessive CO2 production

A

Increased lactic acid production

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3
Q

what is the probable cause of cellular swelling during the early stages of cell injury?

a. fat inclusion
b. loss of genetic integrity
c. hydrolytic enzyme activation
d. NA+ K+ pumps fail to remove intracellular NA+

A

NA+ K+ pump fails to remove intracellular NA+

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4
Q

Calcification

a. alters membrane permeability
b. the result of low calcium levels in the blood
c. caused by UV light
d. Caused by hypoparathyroidism

A

Alters membrane permeability

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5
Q

Cellular swelling?

a. reversible
b. evident early in all types of cellular injury
c. associated with hyperkalemia
d. extracellular movement of fluid

A

Reversible

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6
Q

Which of the following is reversible?

a. karyolysis
b. fatty infiltration
c. hydropic degeneration
d. glycogen formation

A

karyolysis

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7
Q

Aging:

a. likely involves autoantibodies
b. does not have a genetic relationship
c. results from damage accumulation
d. decreases hormonal degredation

A

likely involves autoantibodies

results from damage accumulation

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8
Q

In the theories of aging, cross-linking implies that:

a. the lifespan and number of times the cell can replicate are programmed
b. the number of cells doubling is limited
c. there is oxygen toxicity
d. cell permeability decreases

A

cell permeability decreases

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9
Q

Caused by TB infection

a. liquefactive nécrosais
b. rigor mortis
c. caseous necrosis

A

caseous necrosis

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10
Q

Rigidity of muscles after somatic death

a. hyperplasia
b. rigor mortis
c. mechanoporation

A

Rigor Mortis

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11
Q

increased tissue mass because of increased cell numbers

a. hyperplasia
b. metaplasia
c. hypertrophy

A

Hyperplasia

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12
Q

results from lysosomal release of hydrolytic enzymes

a. liquefactive nécrosais
b. gangrene
c. casseous necrosis

A

liquefactive necrosis

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13
Q

replacement of one cell type with another more suitable type

a. metaplasia
b. fatty necrosis
c. lead

A

metaplasia

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14
Q

Disruption of cell membranes

a. Hypertrophy
b. Dysplasia
c. Mechanoporation

A

mechanoporation

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15
Q

pancreatic necrosis

a. Fatty Necrosis
b. caseous Necrosis
c. Gangrene

A

fatty Necrosis

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16
Q

Coagulative and liquefactive necrosis

a. Apoptosis
b. Gangrene
c. Caseous Necrosis

A

caseous necrosis

17
Q

tissue death

a. gangrene
b. rigor mortis

A

gangrene

18
Q

normal pathologic cellular self destruction

a. apoptosis
b. gangrene
c. caseous necrosis

A

apoptosis

19
Q

lipid peroxidation

a. oxygen deprived free radicals
b. lead
c. carbon monoxide

A

oxygen-deprived free radicals

20
Q

neurotransmitter interference

a. lead
b. mercury
c. carbon monoxide

A

lead

21
Q

asphyxiation

a. lead
b. carbon monoxide
c. free radicals

A

carbon monoxide

22
Q

depressed fatty acid oxidation

a. tobbaco
b. ethanol
c. detached ribosomes

A

ethanol

23
Q

depressed protein synthesis

a. xenobiotics
b. detached ribosomes
c. creatine kinase

A

detached ribosomes

24
Q

during reperfusion with oxygenation _________ is produced, which creates superoxides, hydrogen peroxide, and free radicals

a. carbon dioxide
b. pyruvic acid
c. xanthine oxidase

A

xanthine oxidase

25
Q

specific enzymes, such as ________. are released into ECF, during muscular injury

a. creatine kinase
b. lactic acid
c. myoglobin

A

creatine kinase (CK)