Bovine GI Flashcards

1
Q

Presentation of Pyloric outflow obstruction

A
  • gradual decrease in appetite and milk production
  • distended abdomen, scant, loose feces with undigested material
  • uncomfortable with high HR
  • Urine is alkaloid but has ketones (paradoxic ketonuria)
  • Rumen is acidic with high chloride
  • hypokalemic, hypochloremic metabolic alkalosis
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2
Q

What is vagal indigestion ( or obstructive indigestion syndrome)

A
  • an outflow failure at the reticulo-omasal orifice (type 2)

or at the pyloric sphincter of the abomasum (type 3)

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3
Q

MC Cause of outflow failure at the pylorus

A
  • secondary to abomasal volvulus
  • abomasal volvulus causes damage to the vagal nerve +/- abomasal wall —> this leads to prevention of normal contraction —> leads to abomasal impaction and THUS ….. outflow failure at the pylorus
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4
Q

Presentation of abomasal impaction from poor quality feedstuffs

A
  • almost identical to outflow failure at the pylorus
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5
Q

what is the cause of increased rumen chloride

A

obstruction of the abomasum prevents the passage of ingest and gastric acid to the small intestines.

The small intestine is where the (Cl-) of the gastric acid ( HCl ) is reabsorbed and the (H+) is neutralized by (HCO3) from the pancreas.

—> the HCL eventually refluxes back into the rumen

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6
Q

Cause of Hypokalemia in pyloric obstruction

A
  • when Cl- is low in the blood stream due to sequestration , the kidneys need to resorb a negative ion along with Na+

—> so instead of Cl-, it takes HCO3 —> leading to metabolic alkalosis

Blood is alkalotic …. to balance things out, the body’s cells shifts H+ extracellularly —> leading to intracellular shift of K+ ( which is already sequestered in the abomasum as it is ) –> hypokalemia

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7
Q

Cause of paradoxic aciduria

A

usually K+ is excreted into the urine in exchange for Na+ resorption ….. BUT if the patient is hypokalemia, the kidney needs a replacement .

So instead of K+, the kidney excretes H+ into the urine.

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