Approach to patient with motor weakness Flashcards

1
Q

2 cerebral hemispheres
• They are connected to each other

A

Corpus callosum

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2
Q

outer gray matter (cerebral cortex):

A

is composed of
nerve cells & contains area that control specific function

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3
Q

Inner white matter (depth of cerebral hemisphere): it
is composed

A

nerve fibers that conduct impulses to
and from cerebral cortex

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4
Q

Basal ganglia
Site
Structure
Function

A

At the base of each hemisphere deep in white
matter.
• Caudate, Putamen, Globus pallidus.
• Control extrapyramidal system

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5
Q

Brain Stem (from above downwards)

A

Midbrain: contains the motor nuclei of cranial
nerves 3, 4.
2- Pons: contains the motor nuclei of cranial
nerves 5, 6, 7.
3- Medulla: contains the motor nuclei of cranial
nerves 9, 10, 11, 12.

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6
Q

have no motor nuclei.
They are sensory nerves concerned with special sensations.

A

The 1st, 2nd, and 8th cranial nerv

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7
Q

Cerebellum site

A

It lies at the back and bottom of the cranium
behind the brain stem in the posterior cranial
fossa.

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8
Q

Spinal cord (Transverse section)

A

It contains gray matter (Cells) surrounded by
white matter (Fibers)
• The gray matter: H shaped
2 anterior horns: motor function
2 posterior horns: sensory function
• The white matter: nerve fibers arranged into
ascending and descending tracts

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9
Q

Spinal cord ascending tracts

A

Lateral & ventral spinothalamic: for superficial sensations
Posterior column: for deep sensations
Spinocerebellar: for cerebellar information

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10
Q

Spinal cord The important descending tracts

A

The pyramidal tract (corticospinal)
The extrapyramidal tracts
The cerebello-spinal tracts

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11
Q

50 years old
gentleman
presented with low
back pain.
He has history of
lifting heavy objects.

A

Intervertebral disc prolapse

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12
Q

The pyramidal system
Oridin
Terminal
Control
Function

A

Origin: in the cerebral cortex (motor area 4,
premotor area 6).
• Termination: at the AHCs of the different levels of
the spinal cord.
• Control: it controls the opposite side of the body.
• Function:
Initiation of the voluntary motor activity
Inhibition of the deep reflexes
Inhibition of the muscle tone

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13
Q

The extrapyramidal system
Origin ‘termination
Conytal
Function

A

Origin: from the basal ganglion
• Termination: at the AHCs of the different
levels of the spinal cord.
• Control: it controls the opposite side of the
body
• Functions:
Regulation of the voluntary motor activity
Regulation of the emotional & associated movements
Inhibition of the muscle tone

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14
Q

The cerebellar system
Origin
Terminal
Control
Functions

A

Origin: from the cerebellum.
• Termination: at the AHCs of the different
levels of the spinal cord.
• Control: it controls the same side of the body.
• Functions:
Coordination of the voluntary motor activity initiated by
pyramidal system
Maintenance of equilibrium

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15
Q

The voluntary motor impulses originates mainly
in the large pyramidal cells

A

Betz cells

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16
Q

In the brain stem, some of the descending fibers
separate to supply the motor nuclei of the
cranial nerves of BOTH sides except

A

lower ½
of the facial nucleus and all of the hypoglossal
nucleus which are supplied only from the
opposite pyramidal tract.

17
Q

Does all cortico spinal tract decessuate at lower medilla

A

85% of fibers cross (decussate) to descend in the
white matter of the opposite side of the spinal cord.
- 15% of the fibers descend directly in the white
matter of the same side of the spinal cord.

18
Q

The surest sign of pyramidal lesion.

A

Clonus

19
Q

Signficance of fassiculation

A

Lmnl at ahc

20
Q

Difference in wasting between umnl
Lmnl

A

Late dissue
Early marked

21
Q

Difference between babinksin in umnl lmnl

A

Plantar extension
Plantar fekxion or absent

22
Q

Site of lesion of hemiplegia

A

pyramidal tract lesion at any point
from its origin in the cerebral cortex down to the
5th cervical segment of the spinal cord.

23
Q

Paraplegia

A

UMNL at any
level of pyramidal tract below level of C5 and
above level of L2) or flaccid paraplegia (LMNL at
any level from AHCs till muscles)

24
Q

Bilat umnl

A

Paraplegia
Quadriplegia

25
Q

Relapsing intermiitent hemuplegia

A

Ms

26
Q

Gradual progressive hemiplegia

A

Neoplastic

27
Q

Acute onset hemiplegia

A

Stroke

28
Q

Cause of paraplegia without sensory level loss

A

Bilateral
symmetrical
(motor neuron
disease),
disseminated
asymmetrical
(multiple
sclerosis)

29
Q

Paraplegia with sensory level

A

Compression (disc
prolapse, Pott’s
disease), vascular
(anterior spinal
artery occlusion),
inflammatory
(transverse myelitis)

30
Q

Lmnl withabnormal sensation

A

Nerve roots cauda equina

31
Q

Lmnl with nrromal sensation
Fatigue.
Ptosis
Descending paralysis

A

Myasthenia gravis
Lamert Eaton
syndrome

32
Q

Lmnl with normal sensation
Proximal muscles weakness

A

Muslce
Duvhene
Polio

33
Q

Lnml with normal sensation with fassicukation

A

Ahc
Polio
Prog muscle atrophy

34
Q

Ascending paralysis
Prox muscles
A reflexia
Normal sensation
Lmnl

A

,otor n
Gbs