Acute Periodontal Conditions Flashcards

1
Q

What do all acute periodontal conditions have in common?

A

They cause pain and rapid periodontal destruction.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

How are acute periodontal lesions distinct from other forms of periodontitis?

A

They are rapid onset

They cause rapid destruction of periodontal tissues

They cause pain or discomfort which prompts patients to seek urgent care

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is the definition of an abscess?

A

Localized accumulation of pus in the periodontal tissues (gingival wall of a periodontal sulcus/pocket) resulting in a significant tissue breakdown.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How are abscesses of the periodontium classified?

A

Location: Gingival and periodontal abscesses.

Course of the lesion: Acute and chronic

Number of abscesses: Single and multiple

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How are gingival abscesses different from periodontal abscesses?

A

Gingival abscess - don’t have previous attachment loss (healthy or gingivits but not tissue destruction)

Previous periodontitis - then the patient has
periodontal abscess

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What causes periodontal abscesses?

A

Impaction: Dental floss, orthodontic elastic, popcorn hulls.

Harmful habits: Wire or nail biting

Orthodontic factors: Orthodontic forces on a cross-bite.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

How big of an issue is a periodontal abscess?

A

14% of all dental emergencies are periodontal abscesses.

It is the third most prevalent dental infection in the UK

37% of patients are under maintenance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Who are periodontal abscesses seen most often in?

A

Associated with probing depths deeper than 6mm and are more common in molar sites (furcations)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What adverse outcomes can periodontal abscesses lead to?

A

Can lead to tooth loss.

It is the main reason for tooth extraction in maintenance patients for questionable prognoses teeth.

Potential risk for bacteremia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What are potential etiologies of periodontal abscesses?

A

Acute exacerbation of untreated periodontitis

Acute exacerbation of maintenance patients

After periodontal debridement (incomplete or debris pushed)

After systemic antimicrobial intake

After surgical therapy (membrane or suture contamination)

Uncontrolled diabetic patient HbA1c 10+ (if they are periodontitis patients)

Impact of foreign bodies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

How does a periodontal abscess develop?

A

Invasion of bacteria to soft tissue (pocket) -> Development of inflammatory process -> Inflammatory products lead to (PMN influx, production of pus, connective tissue destruction, encapsulation of bacterial infection) -> Rapid rate of destruction pending of bacterial growth, virulence and pH

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Do periodontal abscesses have a distinct pathophysiology when compared to other periodontitis lesions?

A

Periodontal abscesses involve entry of bacterio or foreign body into the soft tissues surrounding the periodontal pocket which leads to PMNs and other immune cells. If the neutrophils fail to control the influx of bacteria or to clear the foreign body, degranulation, necrosis, and further neutrophil influx may occur. This leads to formation of pus and if the pus is not drained it forms into an abscess. This lesion resolves more rapidly because it creates a low pH environment within the abscess which leads to rapid enzymatic disruption of surrounding connective tissues and has a greater potential for resolution if quickly managed.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How does the microbiology of abscesses look?

A

It is polymicrobial Gram negative, rod predominant anaerobic bacteria

EXTRA INFO:

*4 complexes are possible with different bacterial content:

Red complex: Consist mostly of P gingivalis which is the most prevalent and T forshythia as well as treponema species.

Orange complex: Consists of P intermedia and F nucleatum

Yellow complex*
From Clinical and microbiological characterization of periodontal abscesses

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

How can a periodontal abscess be diagnosed?

A

Evaluation of symptoms: Discomfort, pain, tenderness, swelling, tooth mobility, tooth elevation, sensitivity to palpation.

Clinical findings:
Ovoid elevation lateral / labial to a tooth, or diffuse swelling or redness, BOP.
Suppuration, deep periodontal pocket and signs of periodontitis and mobility.

Radiographic examination: Normal appearance, some bone loss

Other: Fever, malaise, lymphadenopathy, elevated leukocyte number.

History of previous dental or antibiotic intake.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are some differential diagnoses of periodontal abscesses?

A

Other abscesses of the mouth such as: Periapical abscess, Lateral periapical cyst, Vertical root fracture,
Post operative infection

Other serious dental conditions such as Osteomyelitis, ONJ, Squamous cell carcinoma, Metastatic carcinoma, Head and neck cancer, Eosinophilic granuloma, Pyogenic Granuloma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How can other periodontal abscesses ruled out?

A

Proper assessment: Caries or deep restorations, periodontal probing, pulp tests, abscess location, radiographic examination, general periodontal condition, and control the HbA1c

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

How can other serious dental conditions be ruled out?

A

Biopsy if not responding

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

How is a periodontal abscess treated?

A

Drainage through pocker or incision compression.

Meticulous periodontal debridement and removal of foreign body if that is the cause.

Extraction: Severe damage and/or hopeless prognosis.

Surgical procedures if needed.

Analgesics

Systemic antimicrobials adjunctive to treatment (especially if there is a fever and swollen lymph nodes)

Sole AB: if there is diffuse infection and inadequate draining.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

How is an acute gingival abscess treated?

A

If it is very localized and there is no previous periodontitis:

Object should be eliminated, drained by incision, and scaling should be done through the sulcus and drained. Followed by warm saline risk and follow up 24 - 48 hours later.

Analgesics can be used but no ABs because it is very localized.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

How should pericoronal abscesses due to impacted third molars be treated?

A

Drainage + irrigation

Antimicrobials

Depending on the prognosis: Removal of the peri-coronal tissue and extraction.

21
Q

How can pericoronal abscesses due to impacted 3rd molars be detected?

A

Causes accumulation of pus around a pericoronal tissue, trismus, and pain

22
Q

What is necrotizing periodontal disease known as?

A

Vincent’s Disease

Trench mouth disease

Necrotising gingivo-stomatitis

Fuso-spirochaetal stomatitis

Ulcerative membranous gingivitis

Acute Ulcerative gingivitis

Necrotising ulcerative gingivitis

Acute necrotising ulcerative gingivitis (ANUG)

23
Q

How can necrotizing periodontal diseases be classified?

A

Location: Necrotizing gingivitis (only gingival tissue), Necrotizing periodontitis (progression to PDL and alveolar bone), and necrotizing stomatitis (deeper beyond the MGL)

24
Q

What is necrotizing gingivitis?

A

An acute inflammatory process of the gingival tissues characterized by presence of necrosis/ulcer of the interdental papillae, gingival bleeding and pain.

25
Q

What is necrotizing periodontitis?

A

An inflammatory condition of the periodontium characterized by presence of necrosis/ulcer of the interdental papillae, gingival bleeding, halitosis, pain, and rapid bone loss. Other signs/symptoms associated with this condition include pseudomembrane formation, lymphadenopathy, and fever.

26
Q

What is necrotizing stomatitis?

A

A severe inflammatory condition of the periodontium and the oral cavity in which soft tissue necrosis extends beyond the gingiva and bone denudation may occur through the alveolar mucosa, with larger areas of osteitis and formation of bone sequestrum.

27
Q

Do necrotizing periodontal diseases have a distinct pathophysiology when compared to periodontitis lesions?

A

Yes, necrotizing gingivitis lesions are characterized by presence of ulcers within the stratified squamous epithelium and the superficial layer of the gingival connective tissue surrounded by non-specific acute inflammatory infiltrate. 4 zones have been described

28
Q

Who are necrotizing periodontal diseases most commonly seen in?

A

Strongly associated with impairment in the host immune system:

1) In chronically, severely immuno compromised patients.
2) In temporarily or moderately compromised patients.

29
Q

Why must necrotizing periodontal diseases be controlled immediately?

A

They are the most severe oral biofilm related condition and must be controlled immediately to avoid progression

30
Q

What are the predisposing factors to necrotizing periodontal diseases?

A

Military personnel

Students

HIV+

Malnutrition

Smoking

31
Q

How common are necrotizing periodontal diseases?

A

14% prevalence in WWII

Developed countries less than 0.5%

Developing countries 1.7 to 1.5%

Chile 6 - 7%

NP less frequent 0 - 11% HIV+

32
Q

What are the likely outcomes of necrotizing periodontal diseases?

A

Susceptible to recurrence

Can become chronic

Can lead to cancrum oris or noma (Systemic diseases-malnutrition)

Can be treated

33
Q

How does NPD occur? (Aetiology)

A

Spirochetes and fusiform bacteria

Invasion of epithelium and connective tissues

Necrotic tissue perfect microbiological niche

Active periodontal destruction by activation host response

34
Q

What bacteria are seen in necrotizing periodontal diseases?

A

Spirochetes such as:

Treponema species

Selenomona species

Fusobacterium species

P intermedia

Possible role of viruses

35
Q

What does the histology of NPD look like?

A

epithelial and connective tissue ulcers surrounded by non-specific acute inflammatory infiltrate.

4 zones:

Zone 1: Superficial bacterial zone

Zone 2: Neutrophil rich zone

Zone 3: Necrotic zone

Zone 4: Spirochete infiltration zone

36
Q

What are the predisposing factors for NPD?

A

Poor oral hygieve + Immune suppression.

37
Q

How is Necrotizing periodontal gingivitis diagnosed?

A

Based on clinical findings:

Necrosis and ulcers on free gingiva,

SPECIALLY tip of interdental papilla

Spontaneous or easy gingival bleeding

Rapid onset pain

Pouch out appearance

Marginal erythema

Pseudomembrane Whitish / yellow

Halitosis

Lymphadenopathies, fever and discomfort

38
Q

How is Necrotizing periodontitis diagnosed?

A

Necrosis and Ulcers as NG

Extent to PDL and alveolar bone

Attachment loss

Pocket pending on extension of necrosis

Open interdental papilla
Necrosis in the middle (crater)

Interdental bone exposed and denuded

Crater favors plaque accumulation

Bone sequestrum

39
Q

How is Necrotizing stomatitis diagnosed?

A

Denudation extending throuhg alveolar mucosa

Bone sequestrum

Loose bone fragments

Interdental or labial palatal

40
Q

How is acute condition controlled of NPD?

A

Immediate treatment needed to arrest disease process and destruction:

Control patient discomfort and pain.

Periodontal debridement under LA

Use of power instrumentation devices

Restricted oral hygiene (chlorhexidine mouth-rinses)

Antibiotics adjunctive to treatment (metronidazole 400 mg)

Recall and reassessment 24 to 48 hours

41
Q

How is pre-existing condition treated for NPD?

A

Control gingivitis or periodontitis

OHI

Control local predisposing factors

Control existing systemic predisposing factors

42
Q

What corrective treatment is used for NPD?

A

Correct altered gingival topography

Gingivectomy

Gingivoplasty

Regenerative / resective surgery

43
Q

What happens during maintenance phase of NPD?

A

Control predisposing factors

Ensure patient is complying with treatment

44
Q

How should HIV patients be treated differently?

A

Screening for HIV

Irrigation of disease sites with iodine-povidone

Metronidazole is still a choice

Opportunistic candida overgrowth so check for that

45
Q

What causes primary herpetic infection and where does it present?

A

HSV-1

May present labially or intraorally

46
Q

How does primary herpetic gingivo-stomatitis present?

A

Dysphagia, fever, malaise, submandibular adenopathy.

Children 2 - 5 years

Oral vesicles (erosion - ulcers)

47
Q

How is primary herpetic gingivo stomatitis treated?

A

Treat if symptomatic

Use antiviral agents, nutritional support

48
Q

What are the signs of recurrent herpetic infection?

A

Intraoral or labial

Discomfor itching, stinging, vesicle, erosion, ulceration

No treatment or topical antiviral agents

49
Q

What do allergic reactions in the mouth look like?

A

In the mouth urticarial, angioedema. Erythema
multiforme

Contact allergy (Medications, Metals, materials)

Allergies associated to toothpaste, mouth rinse, chewing gum

Burning Itching stinging