Acute MI Flashcards
Acute myocardial infarction also called as
Heart attack
Occurs when there is a diminished blood supply to the heart which leads to myocardial cell damage and ischemia.
MI
stops in the necrotic areas of
the heart.
Contractile fx
usually occurs due to blockage of the
coronary vessels.
Ischemia
is often the result of thrombus that is superimposed on an ulcerated or unstable atherosclerotic plaque formation in the coronary artery.
Ischemia
- MI’s are described by the area of occurrence.
Anterior, inferior, lateral, posterior
MI classification
Anatomic
Diagnostic
Anatomic
Transmural
Subendocardial
Diagnostic
ST Elevation (STEMI)
Non ST Elevation (Non STEMI)
Non modifiable risk factors
Age
Gender
Fam hx
Modifiable
Smoking
Htn
Dm
Obesity
S/sx
Chest pain
Nausea and vomiting
Cardiovascular changes
The most common initial manifestation
Chest pain
PQRST assessment
Precipitating events
Quality of pain
Radiation of pain
Severity of pain
Timing
results as a reflex from severe pain.
Vomitng
reflexes initiated from area of ischemia.
Vasovagal reflexes
Cardiovascular changes
Initial, increase BP and pulse
Later, decrease bp
Decrease Urine output
Crackles
Distended jugular vein
Obvious pulse
First 10 minutes
Check vs and o2
Establish Iv access
Obtain and review 12 lead ECG
Take brief hx and perform PE
Obtain bld samples: initial cardiac markers, electrolytes, coagulation
Dx
ECG confirm MI
12 lead ECG distinguish STEMI and NONSTEMI
produce certain proteins and enzymes associated with cellular functions.
Myocardial cells
- When cell death occurs, these cellular enzymes are released into the blood stream.
CPK and TROPONIN
CPK
Creatine Phosphokinase
Cpk begins
rise 3 to 12 hours after acute MI.
CPK peak
In 24 hrs