8. Regressive changes of teeth; pulpitis, gingivitis and paradentitis Flashcards
regressive changes of teeth
enamel hypoplasia/hypomineralisation
chronic flourine toxicosis
acquired odontoporosis
porodontia
cories
parodontosis
pigmnetation
cementum hyperplasia
cause of enamel hypoplasia
decrease ameloblast cell activity
ameloblast degeneration
enamel hypoplasia
decreased odontoblast activity
local cause of hypoplasia
injury
hypocalcification
why are Ru most at risk of chronic flourine toxicosis
near aluminium smelters & fertilisers
what do aluminium smelters contain
cryolite which causes the degeneration of ameloblasts and odontoblasts
result of chronic flourine toxicosis on teeth
yellow, brown discolouration
erosions in enamel
result of chronic flourine toxicosis in bones
Ca metabolism is disturbed
osteomalacia
osteodystophia fibrosa
exostoses in adults
rickets in young
what is odontoporosis
lacunar resorption of dentin thus decreasing quality & solidity of teeth
what is porodontia
constant wearing of teeth
spontaneous opening of pulp cavity
what is caries
a disease of the calcified tissues of teh teeth due to actions of carbs and microorganisms
predisposition of caries
food prones to fermentation
mistakes in the teeth structure
where does caries starts
at tooth/ oral cavity junction –> chronic progression into deep tooth layers
is there in inflammation in caries
no
what happens to inorganic portion in caries
decalcification
what happens to organic portion in caries
disintegration
aka paradontosis
Rigg disease
what is paradontosis
dystrophy in periodontium w/o inflammation
degeneration/ necrosis of fibres btw alveolus and cement layer
main consequence of paradontosis
loose teeth
what does colour of tooth depend on
tickness and translucency of enamel
extrinsic discolouration of teeth
exogenous pigment accumulated on surface of tooth
intrinsic discolouration of teeth
pathological changes in substance of teeth that make pigmentation
causes of extrinsic
bacterial stains
food
bleeding gums
dental restorative materials
medications
metal
causes of intrinsic
internal (trauma)
systemic infections
medications (ttc)
bilirubin in blood
amelogenesis imperfecta
dentinogenesis imperfecta
local factors causing hyperplasia
trauma
non fuctional tooth
unopposed tooth
periodontitis
periapical granuloma
systemic factors causing hyperplasia
idiopathic
pituitary gigantism
acromegaly
arthritis
calcinossi
rheumatic fever
consequence of hyperplasia
deformed tooth
loss of blood circulation
what is acute pulpitis
inflammation of dental pulp resulting from untreated caries, trauma, multiple restorations
main symptom of acute pulpitis
pain
reversible pulpitis
begins as limited inflammation
tooth savable
irreversible pulpitis
swelling inside the rigid encasement of dentin
compromised circulation
necrotic pulp
chronic pulpitis
granulation tissue instead of pulp - ulceration
gingivitis types
acute/chronic
circumscribed/diffuse
gingivitis ulcerosa
gingivitis necroticans
gingivitis ulceronecrotica
gingivitis marginalis acuta
gingivitis marginalis chronica
gingivitis marginalis acuta
common in
cats
gingivitis marginalis chronica common in
dogs
parodontitis
inflammation of dental supporting tissues
heals with irreversible damage
plaque, tartar
shift to anaerobic oral flora
consequence of parodontitis
local and systemic chagnes
calculus/ tartar
hardened dental plaque
provides an ideal surface for further formation
consequence of tartar
gingival atrophy
gingivitis
peridontisis
loss of teeth
sepsis
malocclusion
